Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
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Novel p47(phox)-related organizers regulate localized NADPH oxidase 1 (Nox1) activityIdentification and characterization of VPO1, a new animal heme-containing peroxidaseDirect interaction between Tks proteins and the N-terminal proline-rich region (PRR) of NoxA1 mediates Nox1-dependent ROS generationEffects of tempol and redox-cycling nitroxides in models of oxidative stress.Autophagy, mitochondria and oxidative stress: cross-talk and redox signallingThreats to the mind: aging, amyloid, and hypertensionRedox mechanisms in hepatic chronic wound healing and fibrogenesisChemistry and antihypertensive effects of tempol and other nitroxidesRedox mechanisms in age-related lung fibrosisNADPH oxidase inhibitors: a decade of discovery from Nox2ds to HTSNADPH oxidase in stroke and cerebrovascular diseaseAmyloid-β and proinflammatory cytokines utilize a prion protein-dependent pathway to activate NADPH oxidase and induce cofilin-actin rods in hippocampal neuronsRegulation of A20 and other OTU deubiquitinases by reversible oxidationConverging roles of ion channels, calcium, metabolic stress, and activity pattern of Substantia nigra dopaminergic neurons in health and Parkinson's diseaseModified Lipids and Lipoproteins in Chronic Kidney Disease: A New Class of Uremic ToxinsResveratrol ameliorates ionizing irradiation-induced long-term hematopoietic stem cell injury in miceEnhanced ROS production and redox signaling with combined arsenite and UVA exposure: contribution of NADPH oxidaseBridged tetrahydroisoquinolines as selective NADPH oxidase 2 (Nox2) inhibitorsNovel role of NOX in supporting aerobic glycolysis in cancer cells with mitochondrial dysfunction and as a potential target for cancer therapyAlteration in the expression of inflammatory parameters as a result of oxidative stress produced by moderate zinc deficiency in rat lungPoldip2, a novel regulator of Nox4 and cytoskeletal integrity in vascular smooth muscle cellsCadmium induction of reactive oxygen species activates the mTOR pathway, leading to neuronal cell deathAntioxidant Capacity and Proanthocyanidin Composition of the Bark of Metasequoia glyptostroboidesVoltage-gated proton channels: molecular biology, physiology, and pathophysiology of the H(V) familyMolecular insights of p47phox phosphorylation dynamics in the regulation of NADPH oxidase activation and superoxide production.Tissue elasticity and the ageing elastic fibre.Reactive oxygen species regulate F-actin dynamics in neuronal growth cones and neurite outgrowth.Molecular evolution of Phox-related regulatory subunits for NADPH oxidase enzymes.Cell surface changes in the egg at fertilizationDecreased superoxide production in macrophages of long-lived p66Shc knock-out mice.Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) miceReactive oxygen species mediate mitogenic growth factor signaling pathways in human leiomyoma smooth muscle cellsTotal body irradiation causes residual bone marrow injury by induction of persistent oxidative stress in murine hematopoietic stem cells.Nox5 and the regulation of cellular function.NOX enzymes and pulmonary disease.Commensal and probiotic bacteria may prevent NEC by maturing intestinal host defenses.NOX activity is increased in mild cognitive impairmentNOX activity in brain aging: exacerbation by high fat diet.Administration of the resveratrol analogues isorhapontigenin and heyneanol-A protects mice hematopoietic cells against irradiation injuries.Calcium/calmodulin-dependent kinase II mediates the phosphorylation and activation of NADPH oxidase 5.
P2860
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P2860
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
@ast
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
@en
type
label
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
@ast
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
@en
prefLabel
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
@ast
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
@en
P2860
P1476
Nox enzymes, ROS, and chronic disease: an example of antagonistic pleiotropy.
@en
P2093
J David Lambeth
P2860
P304
P356
10.1016/J.FREERADBIOMED.2007.03.027
P577
2007-03-31T00:00:00Z