Chronic Contractile Dysfunction without Hypertrophy Does Not Provoke a Compensatory Transcriptional Response in Mouse Hearts.

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Chronic Contractile Dysfunction without Hypertrophy Does Not Provoke a Compensatory Transcriptional Response in Mouse Hearts.

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2016 nî lūn-bûn

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2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

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name

Chronic Contractile Dysfunctio ...... onal Response in Mouse Hearts.

@ast

Chronic Contractile Dysfunctio ...... onal Response in Mouse Hearts.

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type

Item

label

Chronic Contractile Dysfunctio ...... onal Response in Mouse Hearts.

@ast

Chronic Contractile Dysfunctio ...... onal Response in Mouse Hearts.

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prefLabel

Chronic Contractile Dysfunctio ...... onal Response in Mouse Hearts.

@ast

Chronic Contractile Dysfunctio ...... onal Response in Mouse Hearts.

@en

P1476

Chronic Contractile Dysfunctio ...... onal Response in Mouse Hearts.

@en

P2093

David R Grubb

http://www.w3.org/2001/XMLSchema#string

Elizabeth A Woodcock

http://www.w3.org/2001/XMLSchema#string

Julie R McMullen

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P2860

Modulation of cardiac contractility by the phospholamban/SERCA2a regulatome

Differential gene and transcript expression analysis of RNA-seq experiments with TopHat and Cufflinks

Transient cardiac expression of constitutively active Galphaq leads to hypertrophy and dilated cardiomyopathy by calcineurin-dependent and independent pathways

Signaling effectors underlying pathologic growth and remodeling of the heart

Abnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balance

Regulation of polyphosphoinositide-specific phospholipase C activity by purified Gq.

PKC-alpha regulates cardiac contractility and propensity toward heart failure

Count-based differential expression analysis of RNA sequencing data using R and Bioconductor

HTSeq--a Python framework to work with high-throughput sequencing data

Phospholipase Cβ1b directly binds the SH3 domain of Shank3 for targeting and activation in cardiomyocytes.

P304

e0158317

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P31

scholarly article

P356

10.1371/JOURNAL.PONE.0158317

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Scot J Matkovich

P577

2016-06-30T00:00:00Z

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P698

27359099

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P932

4928941

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