Impaired antibody synthesis after spinal cord injury is level dependent and is due to sympathetic nervous system dysregulation. - Wikidata - awgldk - TriplyDB

Impaired antibody synthesis after spinal cord injury is level dependent and is due to sympathetic nervous system dysregulation.

Impaired antibody synthesis after spinal cord injury is level dependent and is due to sympathetic nervous system dysregulation.

http://www.wikidata.org/entity/Q36066082

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Neuroendocrine regulation of inflammation Multiple organ dysfunction and systemic inflammation after spinal cord injury: a complex relationship B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.The SCIentinel study--prospective multicenter study to define the spinal cord injury-induced immune depression syndrome (SCI-IDS)--study protocol and interim feasibility data.Adrenergic-mediated loss of splenic marginal zone B cells contributes to infection susceptibility after stroke Divergent effects of norepinephrine, dopamine and substance P on the activation, differentiation and effector functions of human cytotoxic T lymphocytes Fenbendazole improves pathological and functional recovery following traumatic spinal cord injury.Assessment of depression in a rodent model of spinal cord injury.The paradox of chronic neuroinflammation, systemic immune suppression, autoimmunity after traumatic chronic spinal cord injury Inducible protein-10, a potential driver of neurally controlled interleukin-10 and morbidity in human blunt trauma.Development of protective autoimmunity by immunization with a neural-derived peptide is ineffective in severe spinal cord injury.Chronic thoracic spinal cord injury impairs CD8+ T-cell function by up-regulating programmed cell death-1 expression.Genome-wide gene expression profiling of stress response in a spinal cord clip compression injury model Spinal cord injury causes chronic liver pathology in rats.Chronic spinal cord injury impairs primary antibody responses but spares existing humoral immunity in mice Stress exacerbates neuron loss and microglia proliferation in a rat model of excitotoxic lower motor neuron injury Antibody profiling identifies novel antigenic targets in spinal cord injury patients.The Transcriptional Response of Neurotrophins and Their Tyrosine Kinase Receptors in Lumbar Sensorimotor Circuits to Spinal Cord Contusion is Affected by Injury Severity and Survival Time.Stress-induced effects, which inhibit host defenses, alter leukocyte trafficking CD11d Antibody Treatment Improves Recovery in Spinal Cord-Injured Mice.CD11d integrin blockade reduces the systemic inflammatory response syndrome after spinal cord injury.Silencing spinal interneurons inhibits immune suppressive autonomic reflexes caused by spinal cord injury.Chronic spinal cord injury attenuates influenza virus-specific antiviral immunity.Autonomic dysreflexia causes chronic immune suppression after spinal cord injury Mechanisms and implications of adaptive immune responses after traumatic spinal cord injury Spinal cord injury-induced immune deficiency syndrome enhances infection susceptibility dependent on lesion level Traumatic brain injury-induced alterations in peripheral immunity Spinal cord injury, immunodepression, and antigenic challenge.Impaired immune responses following spinal cord injury lead to reduced ability to control viral infection.Surgical denervation of ocular sympathetic afferents decreases local transforming growth factor-beta and abolishes immune privilege Stress hormones collaborate to induce lymphocyte apoptosis after high level spinal cord injury Activation of the hypothalamic-pituitary-adrenal (HPA) axis contributes to the immunosuppression of mice infected with Angiostrongylus cantonensis.Spinal cord injury modulates the lung inflammatory response in mechanically ventilated rats: a comparative animal study.Impaired toll like receptor-7 and 9 induced immune activation in chronic spinal cord injured patients contributes to immune dysfunction.Preclinical Efficacy Failure of Human CNS-Derived Stem Cells for Use in the Pathway Study of Cervical Spinal Cord Injury.Opioid administration following spinal cord injury: implications for pain and locomotor recovery.Immune dysfunction and chronic inflammation following spinal cord injury.Spinal Cord Injury Suppresses Cutaneous Inflammation: Implications for Peripheral Wound Healing.Autonomic dysreflexia after spinal cord injury: Systemic pathophysiology and methods of management.Mechanisms and Therapeutic Relevance of Neuro-immune Communication.

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P2860

Impaired antibody synthesis after spinal cord injury is level dependent and is due to sympathetic nervous system dysregulation.

http://www.wikidata.org/entity/Q36066082

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J.EXPNEUROL.2007.05.019

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Experimental Neurology

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Kurt M Lucin

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Phillip G Popovich

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T Bucky Jones

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Virginia M Sanders

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William B Malarkey

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P2860

Ventilator-associated pneumonia

Vaccination with a Nogo-A-derived peptide after incomplete spinal-cord injury promotes recovery via a T-cell-mediated neuroprotective response: comparison with other myelin antigens

Known and plausible modulators of depressed immune functions following spinal cord injuries.

Hemolytic plaque formation by leukocytes in vitro. Control by vasoactive hormones

Protective effects of a glucocorticoid on downregulation of pulmonary beta 2-adrenergic receptors in vivo.

Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.

Physiological functions of glucocorticoids in stress and their relation to pharmacological actions.

Perspective: hypothesis: serum IgG antibody is sufficient to confer protection against infectious diseases by inactivating the inoculum.

Pathological CNS autoimmune disease triggered by traumatic spinal cord injury: implications for autoimmune vaccine therapy.

Risk assessment in immunotoxicology. II. Relationships between immune and host resistance tests.

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