Frequency of down-regulation of individual HLA-A and -B alleles in cervical carcinomas in relation to TAP-1 expression. - Wikidata - awgldk - TriplyDB

Frequency of down-regulation of individual HLA-A and -B alleles in cervical carcinomas in relation to TAP-1 expression.

Frequency of down-regulation of individual HLA-A and -B alleles in cervical carcinomas in relation to TAP-1 expression.

http://www.wikidata.org/entity/Q36081450

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Comprehensive genomic characterization of head and neck squamous cell carcinomas Genetic variants in TAP are associated with high-grade cervical neoplasia.Human leukocyte antigens and cancer: is it in our genes?Development of a novel multiplex PCR assay to detect functional subtypes of KIR3DL1 alleles The role of cytotoxic T-lymphocytes in the prevention and immune surveillance of tumors--lessons from normal and immunodeficient mice.Immune selection in neoplasia: towards a microevolutionary model of cancer development.Risk of invasive cervical cancer associated with polymorphic HLA DR/DQ haplotypes.Impaired expression of proteasome subunits and human leukocyte antigens class I in human colon cancer cells.Molecular mechanisms of HLA class I antigen abnormalities following viral infection and transformation.Generating HPV specific T helper cells for the treatment of HPV induced malignancies using TCR gene transfer.Evolving immunotherapeutic strategies in bladder and renal cancer Immunosuppressive tumor microenvironment in cervical cancer patients Association of antigen processing machinery and HLA class I defects with clinicopathological outcome in cervical carcinoma.Multiple genetic alterations cause frequent and heterogeneous human histocompatibility leukocyte antigen class I loss in cervical cancer.Assessment of cytotoxic T-lymphocyte phenotype using the specific markers granzyme B and TIA-1 in cervical neoplastic lesions.An immunohistochemical study of altered immunomodulatory molecule expression in head and neck squamous cell carcinoma.Metastatic MHC class I-negative mouse cells derived by transformation with human papillomavirus type 16 Immune concept of human papillomaviruses and related antigens in local cancer milieu of human cervical neoplasia.Advances in dendritic-cell-based therapeutic vaccines for cervical cancer.HPV infection: immunological aspects and their utility in future therapy Suppression of the CD8 T cell response by human papillomavirus type 16 E7 occurs in Langerhans cell-depleted mice.Classical and non-classical HLA class I aberrations in primary cervical squamous- and adenocarcinomas and paired lymph node metastases.Immunotherapy of MHC class I-deficient tumors.Major histocompatibility antigens and antigen-processing molecules in retinoblastoma.Different N-terminal isoforms of Oct-1 control expression of distinct sets of genes and their high levels in Namalwa Burkitt's lymphoma cells affect a wide range of cellular processes.Analysis of Class I Major Histocompatibility Complex Gene Transcription in Human Tumors Caused by Human Papillomavirus Infection.Induction of MHC class I molecule cell surface expression and epigenetic activation of antigen-processing machinery components in a murine model for human papilloma virus 16-associated tumours.Inhibitory effects of unmethylated CpG oligodeoxynucleotides on MHC class I-deficient and -proficient HPV16-associated tumours.Down-regulation of MHC class I by bovine papillomavirus E5 oncoproteins.[HPV-associated head and neck cancer : mutational signature and genomic aberrations].HLA and cancer: implications for cancer immunotherapy and vaccination.Identification of key amino acid residues that determine the ability of high risk HPV16-E7 to dysregulate major histocompatibility complex class I expression.HPV 16 infection and progression of cervical intra-epithelial neoplasia: analysis of HLA polymorphism and HPV 16 E6 sequence variants.Heterogeneity of HLA and EBER expression in Epstein-Barr virus-associated nasopharyngeal carcinoma.Single nucleotide polymorphisms in antigen processing machinery component ERAP1 significantly associate with clinical outcome in cervical carcinoma.HLA class I phenotype and genotype alterations in cervical carcinomas and derivative cell lines.Transcriptional regulation of the major histocompatibility complex (MHC) class I heavy chain, TAP1 and LMP2 genes by the human papillomavirus (HPV) type 6b, 16 and 18 E7 oncoproteins.Higher HLA class I expression in renal cell carcinoma than in autologous normal tissue.E5 protein of human papillomavirus type 16 selectively downregulates surface HLA class I.

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P2860

Frequency of down-regulation of individual HLA-A and -B alleles in cervical carcinomas in relation to TAP-1 expression.

http://www.wikidata.org/entity/Q36081450

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Assembly and function of the two ABC transporter proteins encoded in the human major histocompatibility complex

In search of the 'missing self': MHC molecules and NK cell recognition

Nomenclature for factors of the HLA system, 1991. WHO Nomenclature Committee for factors of the HLA system

Human immunodeficiency virus genetic variation that can escape cytotoxic T cell recognition

Multiple HLA A11-restricted cytotoxic T-lymphocyte epitopes of different immunogenicities in the Epstein-Barr virus-encoded nuclear antigen 4.

HLA-A2 molecules in an antigen-processing mutant cell contain signal sequence-derived peptides.

Proteolysis, proteasomes and antigen presentation.

A molecular model of MHC class-I-restricted antigen processing.

The role of surface HLA-A,B,C molecules in tumour immunity.

Lessons from T cell responses to virus induced tumours for cancer eradication in general.

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