about
Context-Dependent Regulation of Autophagy by IKK-NF-κB Signaling: Impact on the Aging ProcessRole of E2-Ub-conjugating enzymes during skeletal muscle atrophyThe TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and diseaseCellular and molecular mechanisms of muscle atrophyMolecular pathways leading to loss of skeletal muscle mass in cancer cachexia--can findings from animal models be translated to humans?Clinical classification of cancer cachexia: phenotypic correlates in human skeletal muscleTranslational studies in older men using testosterone to treat sarcopenia.Skeletal muscle atrophy and the E3 ubiquitin ligases MuRF1 and MAFbx/atrogin-1PGC1α plays a critical role in TWEAK-induced cardiac dysfunction.Novel TAZ modulators enhance myogenic differentiation and muscle regeneration.Gene expression changes controlling distinct adaptations in the heart and skeletal muscle of a hibernating mammalMuscle inflammation susceptibility: a prognostic index of recovery potential after hip arthroplasty?Expression of TRAF6 and ubiquitin mRNA in skeletal muscle of gastric cancer patients.Muscle RANK is a key regulator of Ca2+ storage, SERCA activity, and function of fast-twitch skeletal muscles.TRAF molecules in cell signaling and in human diseasesTWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity.Systematic protein-protein interaction and pathway analyses in the idiopathic inflammatory myopathies.TWEAK/Fn14 pathway is a novel mediator of retinal neovascularization.New roles for Smad signaling and phosphatidic acid in the regulation of skeletal muscle mass.Expression of tumor necrosis factor-like weak inducer of apoptosis and fibroblast growth factor-inducible 14 in patients with polymyositis and dermatomyositis.BGP-15 Protects against Oxaliplatin-Induced Skeletal Myopathy and Mitochondrial Reactive Oxygen Species Production in Mice.Mechanisms for fiber-type specificity of skeletal muscle atrophy.Protein breakdown in muscle wasting: role of autophagy-lysosome and ubiquitin-proteasomeA further TWEAK to multiple sclerosis pathophysiology.Novel investigational drugs mimicking exercise for the treatment of cachexia.The impact of postexercise essential amino acid ingestion on the ubiquitin proteasome and autophagosomal-lysosomal systems in skeletal muscle of older men.Long-term exercise training prevents mammary tumorigenesis-induced muscle wasting in rats through the regulation of TWEAK signalling.Disrupted Skeletal Muscle Mitochondrial Dynamics, Mitophagy, and Biogenesis during Cancer Cachexia: A Role for Inflammation.MicroRNA-351 inhibits denervation-induced muscle atrophy by targeting TRAF6Absence of 4-1BB reduces obesity-induced atrophic response in skeletal muscle.It's all about talking: two-way communication between proteasomal and lysosomal degradation pathways via ubiquitin.Skeletal Muscle Function During the Progression of Cancer Cachexia in the Male ApcMin/+ Mouse.Muscle Fn14 gene expression is associated with fat-free mass retention during energy deficit at high altitude
P2860
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P2860
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
TWEAK and TRAF6 regulate skeletal muscle atrophy.
@ast
TWEAK and TRAF6 regulate skeletal muscle atrophy.
@en
type
label
TWEAK and TRAF6 regulate skeletal muscle atrophy.
@ast
TWEAK and TRAF6 regulate skeletal muscle atrophy.
@en
prefLabel
TWEAK and TRAF6 regulate skeletal muscle atrophy.
@ast
TWEAK and TRAF6 regulate skeletal muscle atrophy.
@en
P2860
P1476
TWEAK and TRAF6 regulate skeletal muscle atrophy
@en
P2093
Pradyut K Paul
Shephali Bhatnagar
P2860
P304
P356
10.1097/MCO.0B013E328351C3FC
P50
P577
2012-05-01T00:00:00Z