Cardiac mTOR protects the heart against ischemia-reperfusion injury - Wikidata - awgldk - TriplyDB

Cardiac mTOR protects the heart against ischemia-reperfusion injury

Cardiac mTOR protects the heart against ischemia-reperfusion injury

http://www.wikidata.org/entity/Q36115340

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Klotho and phosphate are modulators of pathologic uremic cardiac remodeling.Leucine-enriched essential amino acids attenuate inflammation in rat muscle and enhance muscle repair after eccentric contraction Impairment of Excitation-Contraction Coupling in Right Ventricular Hypertrophied Muscle with Fibrosis Induced by Pulmonary Artery Banding Three-dimensional myocardial scarring along myofibers after coronary ischemia-reperfusion revealed by computerized images of histological assays.Moderate lifelong overexpression of tuberous sclerosis complex 1 (TSC1) improves health and survival in mice Rapamycin extends life and health in C57BL/6 mice Deficiency of cardiac Acyl-CoA synthetase-1 induces diastolic dysfunction, but pathologic hypertrophy is reversed by rapamycin mTORC2 phosphorylation of Akt1: a possible mechanism for hydrogen sulfide-induced cardioprotection.Late-life rapamycin treatment reverses age-related heart dysfunction.Mechanistic target of rapamycin complex 2 protects the heart from ischemic damage Sevoflurane postconditioning protects rat hearts against ischemia-reperfusion injury via the activation of PI3K/AKT/mTOR signaling bFGF regulates autophagy and ubiquitinated protein accumulation induced by myocardial ischemia/reperfusion via the activation of the PI3K/Akt/mTOR pathway.Identification of protein network alterations upon retinal ischemia-reperfusion injury by quantitative proteomics using a Rattus norvegicus model Health Effects of Long-Term Rapamycin Treatment: The Impact on Mouse Health of Enteric Rapamycin Treatment from Four Months of Age throughout Life Cardiac mTOR rescues the detrimental effects of diet-induced obesity in the heart after ischemia-reperfusion.Loss of long-chain acyl-CoA synthetase isoform 1 impairs cardiac autophagy and mitochondrial structure through mechanistic target of rapamycin complex 1 activation.Rapamycin protects against myocardial ischemia-reperfusion injury through JAK2-STAT3 signaling pathway.N-n-butyl haloperidol iodide protects cardiomyocytes against hypoxia/reoxygenation injury by inhibiting autophagy.Targets, trafficking, and timing of cardiac autophagy Cardioprotection by Klotho through downregulation of TRPC6 channels in the mouse heart.CITED4 induces physiologic hypertrophy and promotes functional recovery after ischemic injury.Danshensu alleviates cardiac ischaemia/reperfusion injury by inhibiting autophagy and apoptosis via activation of mTOR signalling.Metabolomics reveals the mechanisms for the cardiotoxicity of Pinelliae Rhizoma and the toxicity-reducing effect of processing Reperfusion Therapy with Rapamycin Attenuates Myocardial Infarction through Activation of AKT and ERK.Mammalian target of rapamycin signaling in cardiac physiology and disease.The cardioprotection of the insulin-mediated PI3K/Akt/mTOR signaling pathway.MicroRNA-144 is a circulating effector of remote ischemic preconditioning.Insights for Oxidative Stress and mTOR Signaling in Myocardial Ischemia/Reperfusion Injury under Diabetes.The mTOR Signaling Pathway in Myocardial Dysfunction in Type 2 Diabetes Mellitus.Role of mechanistic target of rapamycin (mTOR) in renal function and ischaemia-reperfusion induced kidney injury.Early and short-term triiodothyronine supplementation prevents adverse post-ischemic cardiac remodeling: role of transforming growth factor-β1 and anti-fibrotic miRNA signaling.Impact of caloric restriction on myocardial ischaemia/reperfusion injury and new therapeutic options to mimic its effects The AMPK Agonist PT1 and mTOR Inhibitor 3HOI-BA-01 Protect Cardiomyocytes After Ischemia Through Induction of Autophagy.Overexpression of microRNA-99a attenuates heart remodelling and improves cardiac performance after myocardial infarction.Nicotinamide Adenine Dinucleotide Protects against Spinal Cord Ischemia Reperfusion Injury-Induced Apoptosis by Blocking Autophagy.Inhibition of mammalian target of rapamycin protects against reperfusion injury in diabetic heart through STAT3 signaling.Protective Effects of the Mechanistic Target of Rapamycin against Excess Iron and Ferroptosis in Cardiomyocytes.Salidroside suppressing LPS-induced myocardial injury by inhibiting ROS-mediated PI3K/Akt/mTOR pathway in vitro and in vivo.Influence of the Novel ATP-Competitive Dual mTORC1/2 Inhibitor AZD2014 on Immune Cell Populations and Heart Allograft Rejection.Hyperbaric oxygen protects against myocardial reperfusion injury via the inhibition of inflammation and the modulation of autophagy.

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Cardiac mTOR protects the heart against ischemia-reperfusion injury

http://www.wikidata.org/entity/Q36115340

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Cardiac mTOR protects the heart against ischemia-reperfusion injury

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Cardiac mTOR protects the heart against ischemia-reperfusion injury

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type

label

Cardiac mTOR protects the heart against ischemia-reperfusion injury

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Cardiac mTOR protects the heart against ischemia-reperfusion injury

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Cardiac mTOR protects the heart against ischemia-reperfusion injury

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Cardiac mTOR protects the heart against ischemia-reperfusion injury

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AJPHEART.00241.2012

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American Journal of Physiology - Heart and Circulatory Physiology

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Cardiac mTOR protects the heart against ischemia-reperfusion injury

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P2093

Anthony Rosenzweig

http://www.w3.org/2001/XMLSchema#string

Brendan T Inouye

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Chun-Yang Xiao

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Marielle Scherrer-Crosbie

http://www.w3.org/2001/XMLSchema#string

Masaya Takahashi

http://www.w3.org/2001/XMLSchema#string

Takashi Matsui

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Toshinori Aoyagi

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Yoichiro Kusakari

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P2860

Raptor, a binding partner of target of rapamycin (TOR), mediates TOR action

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

mTOR: from growth signal integration to cancer, diabetes and ageing

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

SIN1/MIP1 maintains rictor-mTOR complex integrity and regulates Akt phosphorylation and substrate specificity

Rapamycin treatment augments both protein ubiquitination and Akt activation in pressure-overloaded rat myocardium

mTORC1 senses lysosomal amino acids through an inside-out mechanism that requires the vacuolar H(+)-ATPase

PI3K rescues the detrimental effects of chronic Akt activation in the heart during ischemia/reperfusion injury

The cytoprotective effects of tumor necrosis factor are conveyed through tumor necrosis factor receptor-associated factor 2 in the heart.

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10.1152/AJPHEART.00241.2012

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1

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2012-05-04T00:00:00Z

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22561297

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reperfusion injury

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3404649

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