Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
about
High levels of RAD51 perturb DNA replication elongation and cause unscheduled origin firing due to impaired CHK1 activation.Hippo pathway and protection of genome stability in response to DNA damage.DNA methylation-based immune response signature improves patient diagnosis in multiple cancers.The MRE11-RAD50-NBS1 Complex Conducts the Orchestration of Damage Signaling and Outcomes to Stress in DNA Replication and Repair.
P2860
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
description
2015 nî lūn-bûn
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2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
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2015年论文
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name
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
@ast
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
@en
type
label
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
@ast
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
@en
prefLabel
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
@ast
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
@en
P2860
P1433
P1476
Safeguarding genome stability: RASSF1A tumor suppressor regulates BRCA2 at stalled forks.
@en
P2093
Dafni Eleftheria Pefani
Eric O'Neill
P2860
P304
P356
10.1080/15384101.2015.1035845
P577
2015-01-01T00:00:00Z