A role for Sp and nuclear receptor transcription factors in a cardiac hypertrophic growth program.
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Role of BAF60a/BAF60c in chromatin remodeling and hepatic lipid metabolismA critical role for the peroxisome proliferator-activated receptor alpha (PPARalpha) in the cellular fasting response: the PPARalpha-null mouse as a model of fatty acid oxidation disordersAltered molecular response to adrenoreceptor-induced cardiac hypertrophy in Egr-1-deficient miceSp3 proteins negatively regulate beta myosin heavy chain gene expression during skeletal muscle inactivityRare Copy Number Variants Identified Suggest the Regulating Pathways in Hypertension-Related Left Ventricular HypertrophySequencing of mRNA identifies re-expression of fetal splice variants in cardiac hypertrophy.Co-regulation of tissue-specific alternative human carnitine palmitoyltransferase Ibeta gene promoters by fatty acid enzyme substrate.What we know and do not know about sex and cardiac disease.Prenatal Hypoxia Reduces Mitochondrial Protein Levels and Cytochrome c Oxidase Activity in Offspring Guinea Pig HeartsMetabolic cardiomyopathies.MEK1 inhibits cardiac PPARα activity by direct interaction and prevents its nuclear localization.Therapeutic potential of CPT I inhibitors: cardiac gene transcription as a target.Energy metabolic reprogramming in the hypertrophied and early stage failing heart: a multisystems approach.Insulin signaling coordinately regulates cardiac size, metabolism, and contractile protein isoform expressionGlucose metabolism and cardiac hypertrophyPPAR-alpha effects on the heart and other vascular tissues.A critical role for peroxisomal proliferator-activated receptor-alpha nuclear receptors in the development of cardiomyocyte degeneration and necrosis.Left ventricular remodeling in swine after myocardial infarction: a transcriptional genomics approach.Maintaining ancient organelles: mitochondrial biogenesis and maturation.Transcriptional control of cardiac fuel metabolism and mitochondrial functionMitochondrial energy metabolism in heart failure: a question of balance.The PPAR genes, cardiovascular disease and the emergence of PPAR pharmacogenetics.Regulation of the sarcoplasmic reticulum Ca2+-ATPase expression in the hypertrophic and failing heart.Return to the fetal gene program: a suggested metabolic link to gene expression in the heart.Metabolic reserve of the heart: the forgotten link between contraction and coronary flow.Deactivation of peroxisome proliferator-activated receptor-alpha during cardiac hypertrophic growthMetabolic gene remodeling and mitochondrial dysfunction in failing right ventricular hypertrophy secondary to pulmonary arterial hypertension.Metabolic remodelling of the failing heart: beneficial or detrimental?Peroxisome proliferator-activated receptor gamma coactivator-1 promotes cardiac mitochondrial biogenesis.Potential of metabolic agents as adjunct therapies in heart failure.Cytoskeletal protein kinases: titin and its relations in mechanosensing.GATA-4 and serum response factor regulate transcription of the muscle-specific carnitine palmitoyltransferase I beta in rat heart.Peroxisome proliferator-activated receptor-α expression induces alterations in cardiac myofilaments in a pressure-overload model of hypertrophy.Cardiac nuclear receptors: architects of mitochondrial structure and function.O-GlcNAcylation and cardiovascular disease.Metabolic Reprogramming via PPARα Signaling in Cardiac Hypertrophy and Failure From Metabolomics to Epigenetics.Expression of lipogenic genes is upregulated in the heart with exercise training-induced but not pressure overload-induced left ventricular hypertrophy.The coactivator PGC-1 cooperates with peroxisome proliferator-activated receptor alpha in transcriptional control of nuclear genes encoding mitochondrial fatty acid oxidation enzymes.Increased COUP-TFII expression in adult hearts induces mitochondrial dysfunction resulting in heart failure.Increased reactive oxygen species production down-regulates peroxisome proliferator-activated alpha pathway in C2C12 skeletal muscle cells.
P2860
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P2860
A role for Sp and nuclear receptor transcription factors in a cardiac hypertrophic growth program.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
A role for Sp and nuclear rece ...... c hypertrophic growth program.
@ast
A role for Sp and nuclear rece ...... c hypertrophic growth program.
@en
type
label
A role for Sp and nuclear rece ...... c hypertrophic growth program.
@ast
A role for Sp and nuclear rece ...... c hypertrophic growth program.
@en
prefLabel
A role for Sp and nuclear rece ...... c hypertrophic growth program.
@ast
A role for Sp and nuclear rece ...... c hypertrophic growth program.
@en
P2093
P2860
P356
P1476
A role for Sp and nuclear rece ...... c hypertrophic growth program.
@en
P2093
P2860
P304
P356
10.1073/PNAS.94.12.6438
P407
P577
1997-06-01T00:00:00Z