Mutations and A/I hypermutations in measles virus persistent infections.
about
Expression and regulation by interferon of a double-stranded-RNA-specific adenosine deaminase from human cells: evidence for two forms of the deaminaseAntiviral actions of interferonsAdenosine-to-inosine RNA editing and human diseaseUnusual distribution of mutations associated with serial bottleneck passages of human immunodeficiency virus type 1Enhancement of replication of RNA viruses by ADAR1 via RNA editing and inhibition of RNA-activated protein kinaseAdenosine deaminases acting on RNA (ADARs) are both antiviral and proviralNuclear antisense RNA induces extensive adenosine modifications and nuclear retention of target transcriptsTranscription inhibition and other properties of matrix proteins expressed by M genes cloned from measles viruses and diseased human brain tissue.Productive measles virus brain infection and apoptosis in CD46 transgenic miceComparison of predicted amino acid sequences of measles virus strains in the Edmonston vaccine lineage.No evidence of measles virus in stapes samples from patients with otosclerosisAntisense RNA: function and fate of duplex RNA in cells of higher eukaryotes.Interplay between innate immunity and negative-strand RNA viruses: towards a rational modelPassage of a Sendai virus recombinant in embryonated chicken eggs leads to markedly rapid accumulation of U-to-C transitions in a limited region of the viral genomeThe zalpha domain of the editing enzyme dsRNA adenosine deaminase binds left-handed Z-RNA as well as Z-DNA.Mechanism of interferon action: functionally distinct RNA-binding and catalytic domains in the interferon-inducible, double-stranded RNA-specific adenosine deaminaseCell fusion by the envelope glycoproteins of persistent measles viruses which caused lethal human brain disease.Characteristics of viruses derived from nude mice with persistent measles virus infectionADARs: viruses and innate immunity.Preferential selection of adenosines for modification by double-stranded RNA adenosine deaminase.Gamma interferon is a major suppressive factor produced by activated human peripheral blood lymphocytes that is able to inhibit foamy virus-induced cytopathic effects.Double-stranded RNA adenosine deaminase ADAR-1-induced hypermutated genomes among inactivated seasonal influenza and live attenuated measles virus vaccines.Large-population passages of vesicular stomatitis virus in interferon-treated cells select variants of only limited resistance.Antigenic determinants of measles virus hemagglutinin associated with neurovirulence.Genetic bottlenecks and population passages cause profound fitness differences in RNA viruses.Mutagenesis-induced, large fitness variations with an invariant arenavirus consensus genomic nucleotide sequence.High pathogenicity of wild-type measles virus infection in CD150 (SLAM) transgenic mice.ADAR1 enhances HTLV-1 and HTLV-2 replication through inhibition of PKR activity.
P2860
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P2860
Mutations and A/I hypermutations in measles virus persistent infections.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Mutations and A/I hypermutations in measles virus persistent infections.
@ast
Mutations and A/I hypermutations in measles virus persistent infections.
@en
type
label
Mutations and A/I hypermutations in measles virus persistent infections.
@ast
Mutations and A/I hypermutations in measles virus persistent infections.
@en
prefLabel
Mutations and A/I hypermutations in measles virus persistent infections.
@ast
Mutations and A/I hypermutations in measles virus persistent infections.
@en
P1476
Mutations and A/I hypermutations in measles virus persistent infections.
@en
P2093
M A Billeter
R Cattaneo
P577
1992-01-01T00:00:00Z