Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
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A tyrosine-phosphorylated carboxy-terminal peptide of the fibroblast growth factor receptor (Flg) is a binding site for the SH2 domain of phospholipase C-gamma 1Human carcinoembryonic antigen, an intercellular adhesion molecule, blocks fusion and differentiation of rat myoblastsExpression of the murine fibroblast growth factor 5 gene in the adult central nervous systemTranscription of the muscle regulatory gene Myf4 is regulated by serum components, peptide growth factors and signaling pathways involving G proteinsCharacterization of recombinant human fibroblast growth factor (FGF)-10 reveals functional similarities with keratinocyte growth factor (FGF-7)Participation of multiple factors, including proliferin, in the inhibition of myogenic differentiationMitogen stimulation affects contractile protein mRNA abundance and translation in embryonic quail myocytes.Differential regulation of skeletal alpha-actin transcription in cardiac muscle by two fibroblast growth factors.Peptide growth factors can provoke "fetal" contractile protein gene expression in rat cardiac myocytes.Molecular cloning and characterization of the ryanodine receptor/junctional channel complex cDNA from skeletal muscle sarcoplasmic reticulum.An activated c-Ha-ras allele blocks the induction of muscle-specific genes whose expression is contingent on mitogen withdrawal.Control of differentiation in BC3H1 muscle cellsControl of myogenic differentiation by fibroblast growth factor is mediated by position in the G1 phase of the cell cycle.Regulation of myogenic differentiation by type beta transforming growth factor.Growth factor control of skeletal muscle differentiation: commitment to terminal differentiation occurs in G1 phase and is repressed by fibroblast growth factorEpidermal growth factor controls smooth muscle alpha-isoactin expression in BC3H1 cellsDexamethasone-dependent inhibition of differentiation of C2 myoblasts bearing steroid-inducible N-ras oncogenes.A growth factor-repressible gene associated with protein kinase C-mediated inhibition of adipocyte differentiationCell surface fibroblast growth factor and epidermal growth factor receptors are permanently lost during skeletal muscle terminal differentiation in culture.Growth factors, signaling pathways, and the regulation of proliferation and differentiation in BC3H1 muscle cells. II. Two signaling pathways distinguished by pertussis toxin and a potential role for the ras oncogene.Growth factors, signaling pathways, and the regulation of proliferation and differentiation in BC3H1 muscle cells. I. A pertussis toxin-sensitive pathway is involved.The expression of sarcomeric muscle-specific contractile protein genes in BC3H1 cells: BC3H1 cells resemble skeletal myoblasts that are defective for commitment to terminal differentiation.Aberrant regulation of MyoD1 contributes to the partially defective myogenic phenotype of BC3H1 cells.Cell cycle commitment of rat muscle satellite cells.Interferon gamma inhibits both proliferation and expression of differentiation-specific alpha-smooth muscle actin in arterial smooth muscle cells.The ryanodine receptor/junctional channel complex is regulated by growth factors in a myogenic cell line.Repression of myogenic differentiation by aFGF, bFGF, and K-FGF is dependent on cellular heparan sulfateFibroblast growth factor and transforming growth factor beta repress transcription of the myogenic regulatory gene MyoD1A ras-dependent pathway abolishes activity of a muscle-specific enhancer upstream from the muscle creatine kinase gene.Identification of upstream and intragenic regulatory elements that confer cell-type-restricted and differentiation-specific expression on the muscle creatine kinase geneAutonomous expression of c-myc in BC3H1 cells partially inhibits but does not prevent myogenic differentiation.The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation.Mitogens and protein synthesis inhibitors induce ornithine decarboxylase gene transcription through separate mechanisms in the BC3H1 muscle cell line.Expression and possible functions of the FGF-5 gene.Metalloendoprotease inhibitors that block fusion also prevent biochemical differentiation in L6 myoblasts.Clinical applications of heparin-binding growth factors.Transformation by myc prevents fusion but not biochemical differentiation of C2C12 myoblasts: mechanisms of phenotypic correction in mixed culture with normal cells.Myogenic differentiation triggered by antisense acidic fibroblast growth factor RNA.Differentiation of muscle, fat, cartilage, and bone from progenitor cells present in a bone-derived clonal cell population: effect of dexamethasone.Differential effects of fibroblast growth factor and tumor promoters on the initiation and maintenance of adipocyte differentiation.
P2860
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P2860
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
description
1985 nî lūn-bûn
@nan
1985年の論文
@ja
1985年論文
@yue
1985年論文
@zh-hant
1985年論文
@zh-hk
1985年論文
@zh-mo
1985年論文
@zh-tw
1985年论文
@wuu
1985年论文
@zh
1985年论文
@zh-cn
name
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
@ast
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
@en
type
label
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
@ast
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
@en
prefLabel
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
@ast
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
@en
P2093
P2860
P356
P1476
Control by fibroblast growth factor of differentiation in the BC3H1 muscle cell line.
@en
P2093
P2860
P304
P356
10.1083/JCB.100.5.1540
P407
P577
1985-05-01T00:00:00Z