Fibroblast growth factor and transforming growth factor beta repress transcription of the myogenic regulatory gene MyoD1
about
Inhibition of myogenesis by transforming growth factor beta is density-dependent and related to the translocation of transcription factor MEF2 to the cytoplasmDevelopmentally regulated expression and localization of fibroblast growth factor receptors in the human muscleAlteration of homeobox gene expression by N-ras transformation of PA-1 human teratocarcinoma cellsThe basic helix-loop-helix transcription factor Mist1 functions as a transcriptional repressor of myoDTranscription of the muscle regulatory gene Myf4 is regulated by serum components, peptide growth factors and signaling pathways involving G proteinsInflammation induced loss of skeletal muscleTGF-beta inhibits muscle differentiation through functional repression of myogenic transcription factors by Smad3MyoD is indispensable for muscle-specific alternative splicing in mouse mitochondrial ATP synthase gamma-subunit pre-mRNADifferential trans activation associated with the muscle regulatory factors MyoD1, myogenin, and MRF4.Characterisation of a genomic clone covering the structural mouse MyoD1 gene and its promoter regionTGF-β1 enhances contractility in engineered skeletal muscleDifferential regulation of skeletal alpha-actin transcription in cardiac muscle by two fibroblast growth factors.Influence of PDGF-BB on proliferation and transition through the MyoD-myogenin-MEF2A expression program during myogenesis in mouse C2 myoblasts.Pregnancy-associated plasma protein-A regulates myoblast proliferation and differentiation through an insulin-like growth factor-dependent mechanism.Targeted expression of transforming growth factor-beta 1 in intracardiac grafts promotes vascular endothelial cell DNA synthesis.Peptide growth factors can provoke "fetal" contractile protein gene expression in rat cardiac myocytes.Dihydropyridine receptor gene expression is regulated by inhibitors of myogenesis and is relatively insensitive to denervation.Identification of novel MyoD gene targets in proliferating myogenic stem cellsSkeletal muscle tissue engineering: methods to form skeletal myotubes and their applications.TGF-β inhibits muscle differentiation by blocking autocrine signaling pathways initiated by IGF-II.Expression of myogenic factors in denervated chicken breast muscle: isolation of the chicken Myf5 gene.Stem cell activation in skeletal muscle regeneration.The anabolic/androgenic steroid nandrolone exacerbates gene expression modifications induced by mutant SOD1 in muscles of mice models of amyotrophic lateral sclerosis.TGFbeta1 regulation of vimentin gene expression during differentiation of the C2C12 skeletal myogenic cell line requires Smads, AP-1 and Sp1 family members.Expression of transforming growth factor-beta isoforms in small round cell tumors of childhood. An immunohistochemical studyBasic fibroblast growth factor has a differential effect on MyoD conversion of cultured aortic smooth muscle cells from newborn and adult rats.Bone morphogenetic protein-2 converts the differentiation pathway of C2C12 myoblasts into the osteoblast lineage.Lysophosphatidic acid and bFGF control different modes in proliferating myoblastsCadherins promote skeletal muscle differentiation in three-dimensional cultures.MyoD induces myogenic differentiation through cooperation of its NH2- and COOH-terminal regions.The ryanodine receptor/junctional channel complex is regulated by growth factors in a myogenic cell line.Inhibition of myogenesis by the H-ras oncogene: implication of a role for protein kinase C.Ras p21Val inhibits myogenesis without altering the DNA binding or transcriptional activities of the myogenic basic helix-loop-helix factors.Signaling through mitogen-activated protein kinase and Rac/Rho does not duplicate the effects of activated Ras on skeletal myogenesisTransforming growth factor-beta-regulated miR-24 promotes skeletal muscle differentiation.Muscle-specific expression of the troponin I gene requires interactions between helix-loop-helix muscle regulatory factors and ubiquitous transcription factorsTransforming growth factor beta induces myoblast differentiation in the presence of mitogensAngiotensin II inhibits satellite cell proliferation and prevents skeletal muscle regeneration.Hepatic fibrosis, glomerulosclerosis, and a lipodystrophy-like syndrome in PEPCK-TGF-beta1 transgenic mice.Defective skeletal muscle growth in lamin A/C-deficient mice is rescued by loss of Lap2α.
P2860
Q22003948-E35A2BB7-6FD4-43D3-AAAE-C563E6815004Q24311344-F36AA751-615D-439F-8E2A-0A5E2A445E13Q24338771-E9506132-5D7D-496A-991D-BA663FE8DCB1Q24533171-CD7E66E2-4503-4738-ADB9-FBD302D305C0Q24642495-BB0E3BFE-1641-4B79-B339-9F03C3DF970CQ28083759-C4FB3F43-E825-4A3D-81E6-E0A5DD58124CQ28364253-46691E9E-976F-4444-80D5-7F1C75FFB375Q28506896-C9F85994-AA28-480C-A415-56D8CC3ECA61Q30356643-A0593D44-C00A-45C4-B939-94A2A72F89C8Q33286464-E664F674-45AE-491C-8359-045737F0C37AQ33673547-F630A692-B63D-4B8D-A878-26E05EF81E94Q33790096-90DABDB1-8AE2-4AF8-B6B9-1B37DFC67F65Q33894854-BB7DC4A8-152D-418D-BD81-01B89EA595B0Q33989224-BF419EE2-CEBA-4754-91A0-A49480AF04D4Q34193341-12F83BF1-0BCA-4E2C-BF0E-6CE7952BE253Q34243039-23FD1266-E527-480D-9E2B-6528CB0156D4Q34245224-85AD8559-C1D9-44FD-BF46-4BFFD7883B3FQ34285149-9A920BDD-1DF1-4C62-8EC1-E224B7FB3B95Q34322871-D318FB49-95A2-4875-BFDD-E195746B5A79Q34943015-448EEF06-ED7F-4FAD-A0E2-2B86E9778DBBQ34984073-4452CFBB-AF65-4CC8-94A9-55FD6829DC6FQ35547307-25144212-DFE9-48E9-9975-D3C30F2F907BQ35737086-D462F058-4B84-4121-B067-443A2896A39AQ35758216-3BD660ED-8AD0-4E9C-9F37-41D4A529CF61Q35832578-A5B81018-13DB-427E-92C9-8409CEF76B3AQ35832854-3C9D5948-C00D-453A-85A1-4FB7B9551D84Q36235105-51F30766-DDAD-4148-8875-32D2E562B300Q36236438-9855A648-398C-4750-B28C-15941CB5E9D2Q36254787-E323A722-0991-4156-A041-56AFADE50E64Q36320492-9204DE24-804E-46A3-BFB1-DCA52D926A04Q36530035-543EBE89-CE0C-4B15-BAD4-4D2D357A236CQ36532786-58965678-797B-4CEC-B4DE-229DD3E34769Q36554923-BF7D14FD-C3A0-4AC7-8A86-E333A568DB34Q36568977-EFB48E7B-C7BC-4004-B964-5723413A66BEQ36649628-2C9A164B-EADC-445E-ADF8-EAD2562840DCQ36680396-B22B2839-5660-4135-B40B-5CFB73CE2F45Q37047765-D84A0523-E0EB-46CB-9D24-A333979B0FE4Q37099711-02DFD046-8348-4FF0-A679-1518ED47262FQ37375446-E5E29CE6-1CA5-4C3B-8BDC-BA887D50FFCDQ37463359-A5808C45-41BA-4C86-8E67-2E23CA2E2200
P2860
Fibroblast growth factor and transforming growth factor beta repress transcription of the myogenic regulatory gene MyoD1
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Fibroblast growth factor and t ...... myogenic regulatory gene MyoD1
@ast
Fibroblast growth factor and t ...... myogenic regulatory gene MyoD1
@en
type
label
Fibroblast growth factor and t ...... myogenic regulatory gene MyoD1
@ast
Fibroblast growth factor and t ...... myogenic regulatory gene MyoD1
@en
prefLabel
Fibroblast growth factor and t ...... myogenic regulatory gene MyoD1
@ast
Fibroblast growth factor and t ...... myogenic regulatory gene MyoD1
@en
P2093
P2860
P356
P1476
Fibroblast growth factor and t ...... myogenic regulatory gene MyoD1
@en
P2093
E J Taparowsky
S F Konieczny
S J Rhodes
T B Vaidya
P2860
P304
P356
10.1128/MCB.9.8.3576
P407
P577
1989-08-01T00:00:00Z