Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation.
about
Btf, a novel death-promoting transcriptional repressor that interacts with Bcl-2-related proteins.Induced expression of PD-1, a novel member of the immunoglobulin gene superfamily, upon programmed cell deathA new translational regulator with homology to eukaryotic translation initiation factor 4GA novel form of DAP5 protein accumulates in apoptotic cells as a result of caspase cleavage and internal ribosome entry site-mediated translationDo all programmed cell deaths occur via apoptosis?DIO-1 is a gene involved in onset of apoptosis in vitro, whose misexpression disrupts limb developmentDisruption of epithelial cell-matrix interactions induces apoptosisInhibition of axotomy-induced neuronal apoptosis by extracellular delivery of a Bcl-XL fusion proteinNeurturin shares receptors and signal transduction pathways with glial cell line-derived neurotrophic factor in sympathetic neuronsTau protein kinase I is essential for amyloid beta-protein-induced neurotoxicityIntrinsic and extrinsic pathway signaling during neuronal apoptosis: lessons from the analysis of mutant miceCritical function of endogenous XIAP in regulating caspase activation during sympathetic neuronal apoptosisbcl-x is expressed in embryonic and postnatal neural tissues and functions to prevent neuronal cell deathExpression of the SM-20 gene promotes death in nerve growth factor-dependent sympathetic neuronsIntra-axonal translation and retrograde trafficking of CREB promotes neuronal survivalWithdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell deathAlternative programs of cell death in developing retinal tissueCHOP/GADD153 is a mediator of apoptotic death in substantia nigra dopamine neurons in an in vivo neurotoxin model of parkinsonismThe small GTP-binding protein Cdc42 is required for nerve growth factor withdrawal-induced neuronal deathDeath Receptor 6 Promotes Wallerian Degeneration in Peripheral Axons.The specific FKBP38 inhibitor N-(N',N'-dimethylcarboxamidomethyl)cycloheximide has potent neuroprotective and neurotrophic properties in brain ischemia.Potential mechanism of cell death in the developing rat brain induced by propofol anesthesia.Alterations of intracellular calcium homeostasis and mitochondrial function are involved in ruthenium red neurotoxicity in primary cortical cultures.SM-20 is a novel mitochondrial protein that causes caspase-dependent cell death in nerve growth factor-dependent neurons.Extracts of Actinobacillus actinomycetemcomitans induce apoptotic cell death in human osteoblastic MG63 cells.Novel characteristics of glutamate-induced cell death in primary septohippocampal cultures: relationship to calpain and caspase-3 protease activation.TNF-alpha stimulates caspase-3 activation and apoptotic cell death in primary septo-hippocampal cultures.The modular systems biology approach to investigate the control of apoptosis in Alzheimer's disease neurodegeneration.IL3-dependent cells die by apoptosis on removal of their growth factor.The herpes simplex virus type 1 regulatory protein ICP27 is required for the prevention of apoptosis in infected human cells.Gene activation is required for developmentally programmed cell death.Serum and transforming growth factor beta regulate glial fibrillary acidic protein in serum-free-derived mouse embryo cellsApoptotic and anti-apoptotic synaptic signaling mechanisms.Depletion of calcium stores in injured sensory neurons: anatomic and functional correlatesComparative expressed-sequence-tag analysis of differential gene expression profiles in PC-12 cells before and after nerve growth factor treatment.Global analysis of gene expression in NGF-deprived sympathetic neurons identifies molecular pathways associated with cell death.Human clusterin gene expression is confined to surviving cells during in vitro programmed cell death.Translational control of cell fate: availability of phosphorylation sites on translational repressor 4E-BP1 governs its proapoptotic potency.Role of Ca2+ channels in the ability of membrane depolarization to prevent neuronal death induced by trophic-factor deprivation: evidence that levels of internal Ca2+ determine nerve growth factor dependence of sympathetic ganglion cells.Apoptosis and dependence receptors: a molecular basis for cellular addiction.
P2860
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P2860
Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Inhibitors of protein synthesi ...... rve growth factor deprivation.
@ast
Inhibitors of protein synthesi ...... rve growth factor deprivation.
@en
type
label
Inhibitors of protein synthesi ...... rve growth factor deprivation.
@ast
Inhibitors of protein synthesi ...... rve growth factor deprivation.
@en
prefLabel
Inhibitors of protein synthesi ...... rve growth factor deprivation.
@ast
Inhibitors of protein synthesi ...... rve growth factor deprivation.
@en
P2093
P2860
P356
P1476
Inhibitors of protein synthesi ...... rve growth factor deprivation.
@en
P2093
D P Martin
E M Johnson
J G Carter
P S DiStefano
R E Schmidt
P2860
P304
P356
10.1083/JCB.106.3.829
P407
P577
1988-03-01T00:00:00Z