Disparate interaction of peptide ligand with nascent versus mature class I major histocompatibility complex molecules: comparisons of peptide binding to alternative forms of Ld in cell lysates and the cell surface.
about
The Peptide-Receptive Transition State of MHC Class I Molecules: Insight from Structure and Molecular DynamicsUbe2j2 ubiquitinates hydroxylated amino acids on ER-associated degradation substrates.Enhancing the immunogenicity of exogenous hepatitis B surface antigen-based vaccines for MHC-I-restricted T cells.Class I MHC molecules: assembly and antigen presentation.Alternative pathways for processing exogenous and endogenous antigens that can generate peptides for MHC class I-restricted presentation.Opposite effects of endogenous peptide-MHC class I on T cell activity in the presence and absence of CD8.Analysis of major histocompatibility complex class I folding: novel insights into intermediate formsThe optimization of peptide cargo bound to MHC class I molecules by the peptide-loading complex.Recognition of open conformers of classical MHC by chaperones and monoclonal antibodies.A Novel MHC-I Surface Targeted for Binding by the MCMV m06 Immunoevasin Revealed by Solution NMR.Major histocompatibility complex class II-restricted presentation of an internally synthesized antigen displays cell-type variability and segregates from the exogenous class II and endogenous class I presentation pathwaysThe class I-b molecule Qa-1 forms heterodimers with H-2Ld and a novel 50-kD glycoprotein encoded centromeric to I-E betaRestoration of cytomegalovirus antigen presentation by gamma interferon combats viral escape.Extensive peptide ligand exchange by surface class I major histocompatibility complex molecules independent of exogenous beta 2-microglobulin.The natural killer cell receptor Ly-49A recognizes a peptide-induced conformational determinant on its major histocompatibility complex class I ligand.Availability of endogenous peptides limits expression of an M3a-Ld major histocompatibility complex class I chimera.Impaired intracellular transport and cell surface expression of nonpolymorphic HLA-E: evidence for inefficient peptide binding.Physical association of the K3 protein of gamma-2 herpesvirus 68 with major histocompatibility complex class I molecules with impaired peptide and beta(2)-microglobulin assembly.Evidence for MR1 antigen presentation to mucosal-associated invariant T cells.Exogenous peptides enter the endoplasmic reticulum of TAP-deficient cells and induce the maturation of nascent MHC class I molecules.Definition and transfer of a serological epitope specific for peptide-empty forms of MHC class I.Model for the in vivo assembly of nascent Ld class I molecules and for the expression of unfolded Ld molecules at the cell surface.A soluble major histocompatibility complex class I peptide-binding platform undergoes a conformational change in response to peptide epitopes.The Role of Molecular Flexibility in Antigen Presentation and T Cell Receptor-Mediated Signaling
P2860
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P2860
Disparate interaction of peptide ligand with nascent versus mature class I major histocompatibility complex molecules: comparisons of peptide binding to alternative forms of Ld in cell lysates and the cell surface.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Disparate interaction of pepti ...... lysates and the cell surface.
@ast
Disparate interaction of pepti ...... lysates and the cell surface.
@en
type
label
Disparate interaction of pepti ...... lysates and the cell surface.
@ast
Disparate interaction of pepti ...... lysates and the cell surface.
@en
prefLabel
Disparate interaction of pepti ...... lysates and the cell surface.
@ast
Disparate interaction of pepti ...... lysates and the cell surface.
@en
P2093
P2860
P356
P1476
Disparate interaction of pepti ...... lysates and the cell surface.
@en
P2093
P2860
P304
P356
10.1084/JEM.175.1.191
P407
P577
1992-01-01T00:00:00Z