Model for the in vivo assembly of nascent Ld class I molecules and for the expression of unfolded Ld molecules at the cell surface.
about
Amyloid precursor-like protein 2 increases the endocytosis, instability, and turnover of the H2-K(d) MHC class I moleculeSpecificity of amyloid precursor-like protein 2 interactions with MHC class I moleculesThe Peptide-Receptive Transition State of MHC Class I Molecules: Insight from Structure and Molecular DynamicsThe three-dimensional structure of an H-2Ld-peptide complex explains the unique interaction of Ld with beta-2 microglobulin and peptideMechanism for amyloid precursor-like protein 2 enhancement of major histocompatibility complex class I molecule degradation.Enhancing the immunogenicity of exogenous hepatitis B surface antigen-based vaccines for MHC-I-restricted T cells.Requirements for the selective degradation of endoplasmic reticulum-resident major histocompatibility complex class I proteins by the viral immune evasion molecule mK3.Class I MHC molecules: assembly and antigen presentation.Alternative pathways for processing exogenous and endogenous antigens that can generate peptides for MHC class I-restricted presentation.Opposite effects of endogenous peptide-MHC class I on T cell activity in the presence and absence of CD8.Analysis of major histocompatibility complex class I folding: novel insights into intermediate formsNewly discovered viral E3 ligase pK3 induces endoplasmic reticulum-associated degradation of class I major histocompatibility proteins and their membrane-bound chaperonesThe optimization of peptide cargo bound to MHC class I molecules by the peptide-loading complex.Recognition of open conformers of classical MHC by chaperones and monoclonal antibodies.The class I-b molecule Qa-1 forms heterodimers with H-2Ld and a novel 50-kD glycoprotein encoded centromeric to I-E betaPeptide influences the folding and intracellular transport of free major histocompatibility complex class I heavy chains.Intermediates in the assembly and degradation of class I major histocompatibility complex (MHC) molecules probed with free heavy chain-specific monoclonal antibodies.Human major histocompatibility complex (MHC) class I molecules with disulfide traps secure disease-related antigenic peptides and exclude competitor peptidesBiosynthesis of HLA-C heavy chains in melanoma cells with multiple defects in the expression of HLA-A, -B, -C molecules.MR1 uses an endocytic pathway to activate mucosal-associated invariant T cellsRegulation of NK cell responsiveness to achieve self-tolerance and maximal responses to diseased target cellsThe same major histocompatibility complex polymorphism involved in control of HIV influences peptide binding in the mouse H-2Ld system.The amyloid precursor-like protein 2 and the adenoviral E3/19K protein both bind to a conformational site on H-2Kd and regulate H-2Kd expression.Physical association of the K3 protein of gamma-2 herpesvirus 68 with major histocompatibility complex class I molecules with impaired peptide and beta(2)-microglobulin assembly.Antigenic drift as a mechanism for tumor evasion of destruction by cytolytic T lymphocytesEffect of a tapasin mutant on the assembly of the mouse MHC class I molecule H2-K(d).Comparative analysis of the impact of a free cysteine in tapasin on the maturation and surface expression of murine MHC class I allotypes.Influence of the tapasin C terminus on the assembly of MHC class I allotypes.Evidence for MR1 antigen presentation to mucosal-associated invariant T cells.Definition and transfer of a serological epitope specific for peptide-empty forms of MHC class I.Single H2Kb, H2Db and double H2KbDb knockout mice: peripheral CD8+ T cell repertoire and anti-lymphocytic choriomeningitis virus cytolytic responses.Model for the interaction of gammaherpesvirus 68 RING-CH finger protein mK3 with major histocompatibility complex class I and the peptide-loading complex.Alloreactivity, antigen recognition and T-cell selection: three diverse T-cell recognition problems with a common solution.Major histocompatibility complex class I viral antigen processing in the secretory pathway defined by the trans-Golgi network protease furin.The crystal structure of H-2D(b) complexed with a partial peptide epitope suggests a major histocompatibility complex class I assembly intermediate.
P2860
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P2860
Model for the in vivo assembly of nascent Ld class I molecules and for the expression of unfolded Ld molecules at the cell surface.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Model for the in vivo assembly ...... molecules at the cell surface.
@en
type
label
Model for the in vivo assembly ...... molecules at the cell surface.
@en
prefLabel
Model for the in vivo assembly ...... molecules at the cell surface.
@en
P2093
P2860
P356
P1476
Model for the in vivo assembly ...... molecules at the cell surface.
@en
P2093
P2860
P304
P356
10.1084/JEM.178.6.2035
P407
P577
1993-12-01T00:00:00Z