about
Class I-restricted cross-presentation of exogenous self-antigens leads to deletion of autoreactive CD8(+) T cellsImmunologic aspect of ovarian cancer and p53 as tumor antigen.The Role of Invariant Natural Killer T Cells in Dendritic Cell Licensing, Cross-Priming, and Memory CD8(+) T Cell GenerationThe activation and inactivation of mature CD4 T cells: a case for peripheral self-nonself discriminationCompartmentalization of T lymphocytes to the site of disease: intrahepatic CD4+ T cells specific for the protein NS4 of hepatitis C virus in patients with chronic hepatitis CAntigen presentation and the regulation of CD4 memory generation to influenza.Administration of superantigens protects mice from lethal Listeria monocytogenes infection by enhancing cytotoxic T cells.The role of co-inhibitory signals in spontaneous tolerance of weakly mismatched transplantsRole of autoreactive CD8+ T cells in organ-specific autoimmune diseases: insight from transgenic mouse models.Transplantation tolerance-where do we stand?Approaches to improve engineered vaccines for human immunodeficiency virus and other viruses that cause chronic infections.CD40 ligand-mediated interactions are involved in the generation of memory CD8(+) cytotoxic T lymphocytes (CTL) but are not required for the maintenance of CTL memory following virus infection.CD4+ T cell effects on CD8+ T cell location defined using bioluminescence.Review article: molecular signals and genetic reprogramming in peripheral T-cell differentiation.Donor lymphocyte infusions to treat hematologic malignancies in relapse after allogeneic blood or marrow transplantation.Epitopes on the beta subunit of human muscle acetylcholine receptor recognized by CD4+ cells of myasthenia gravis patients and healthy subjects.Killing of dendritic cells: a life cut short or a purposeful death?Concurrent interaction of DCs with CD4(+) and CD8(+) T cells improves secondary CTL expansion: It takes three to tango.A two-step, two-signal model for the primary activation of precursor helper T cellsThe role of dendritic cell subsets in selection between tolerance and immunity.CD4+ T cells play a critical role in the generation of primary and memory antitumor immune responses elicited by SA-4-1BBL and TAA-based vaccines in mouse tumor models.Antigen presenting B cells facilitate CD4 T cell cooperation resulting in enhanced generation of effector and memory CD4 T cells.Vaccination with a recombinant vaccinia virus encoding a "self" antigen induces autoimmune vitiligo and tumor cell destruction in mice: requirement for CD4(+) T lymphocytes.Targeting the tumor microenvironment to enhance antitumor immune responsesHelper T cells, dendritic cells and CTL Immunity.Immature dendritic cells acquire CD8(+) cytotoxic T lymphocyte priming capacity upon activation by T helper cell-independent or -dependent stimuli.Lymphocyte proliferation in mice congenitally deficient in T-cell receptor alpha beta + cells.Thymus epithelium induces tissue-specific toleranceInduction of long-term H-Y-specific tolerance in female mice given male lymphoid cells while transiently depleted of CD4+ or CD8+ T cells.Anergy and exhaustion are independent mechanisms of peripheral T cell toleranceBone marrow-generated dendritic cells pulsed with a class I-restricted peptide are potent inducers of cytotoxic T lymphocytes.Successful T cell priming in B cell-deficient mice.Long-lasting CD8 T cell memory in the absence of CD4 T cells or B cells.CD8(+) T cells mediate CD40-independent maturation of dendritic cells in vivoUncoupling of proliferative potential and gain of effector function by CD8(+) T cells responding to self-antigens.In vivo cytotoxic T lymphocyte elicitation by mycobacterial heat shock protein 70 fusion proteins maps to a discrete domain and is CD4(+) T cell independentCD40-independent pathways of T cell help for priming of CD8(+) cytotoxic T lymphocytes.Induction of a CD8+ cytotoxic T lymphocyte response by cross-priming requires cognate CD4+ T cell help.CD4+ T cell help impairs CD8+ T cell deletion induced by cross-presentation of self-antigens and favors autoimmunity.Viral and bacterial infections interfere with peripheral tolerance induction and activate CD8+ T cells to cause immunopathology
P2860
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P2860
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
A fail-safe mechanism for maintaining self-tolerance
@ast
A fail-safe mechanism for maintaining self-tolerance
@en
type
label
A fail-safe mechanism for maintaining self-tolerance
@ast
A fail-safe mechanism for maintaining self-tolerance
@en
prefLabel
A fail-safe mechanism for maintaining self-tolerance
@ast
A fail-safe mechanism for maintaining self-tolerance
@en
P2860
P356
P1476
A fail-safe mechanism for maintaining self-tolerance
@en
P2093
Matzinger P
P2860
P304
P356
10.1084/JEM.176.2.553
P407
P577
1992-08-01T00:00:00Z