about
Classifying leukemia types with chromatin conformation data.E2F-1 directly regulates thrombospondin 1 expression.Cytogenetic as an important tool for diagnosis and prognosis for patients with hypocellular primary myelodysplastic syndrome.Genetic abnormalities and challenges in the treatment of acute myeloid leukemia.MLL fusions: pathways to leukemiaCongenital Acute Myeloid Leukemia with Unique Translocation t(11;19)(q23;p13.3).Challenges and strategies for generating therapeutic patient-specific hemangioblasts and hematopoietic stem cells from human pluripotent stem cellsPharmacogenetics of alkylator-associated acute myeloid leukemia.Acute leukemia in early childhood.Aberrant chromatin at genes encoding stem cell regulators in human mixed-lineage leukemia.RAS oncogene suppression induces apoptosis followed by more differentiated and less myelosuppressive disease upon relapse of acute myeloid leukemia.Diagnostic microarrays in hematologic oncology: applications of high- and low-density arrays.The molecular signature of oncofusion proteins in acute myeloid leukemia.Insights into the cellular origin and etiology of the infant pro-B acute lymphoblastic leukemia with MLL-AF4 rearrangement.Genomic characterization of acute leukemias.The mixed-lineage leukemia fusion partner AF4 stimulates RNA polymerase II transcriptional elongation and mediates coordinated chromatin remodeling.HOX proteins and leukemia.Bimodal degradation of MLL by SCFSkp2 and APCCdc20 assures cell cycle execution: a critical regulatory circuit lost in leukemogenic MLL fusions.t(4;11) leukemias display addiction to MLL-AF4 but not to AF4-MLLHistory of Maternal Fetal Loss and Childhood Leukaemia Risk in Subsequent Offspring: Differentials by Miscarriage or Stillbirth History and Disease Subtype.Elongation factor ELL (Eleven-Nineteen Lysine-rich Leukemia) acts as a transcription factor for direct thrombospondin-1 regulation.Ligand-independent FLT3 activation does not cooperate with MLL-AF4 to immortalize/transform cord blood CD34+ cells.The silent mutational landscape of infant MLL-AF4 pro-B acute lymphoblastic leukemia.TOP3A, a new partner gene fused to MLL in an adult patient with de novo acute myeloid leukaemia
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P2860
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Molecular pathogenesis of MLL-associated leukemias.
@ast
Molecular pathogenesis of MLL-associated leukemias.
@en
type
label
Molecular pathogenesis of MLL-associated leukemias.
@ast
Molecular pathogenesis of MLL-associated leukemias.
@en
prefLabel
Molecular pathogenesis of MLL-associated leukemias.
@ast
Molecular pathogenesis of MLL-associated leukemias.
@en
P2860
P356
P1476
Molecular pathogenesis of MLL-associated leukemias.
@en
P2093
Mariko Eguchi
Minenori Eguchi-Ishimae
P2860
P2888
P356
10.1532/IJH97.05042
P577
2005-07-01T00:00:00Z
P6179
1033955269