The End2 mutation in CHO cells slows the exit of transferrin receptors from the recycling compartment but bulk membrane recycling is unaffected.
about
Rab10 regulates membrane transport through early endosomes of polarized Madin-Darby canine kidney cellsNeonatal Fc receptor mediates internalization of Fc in transfected human endothelial cellsActin dynamics regulate multiple endosomal steps during Kaposi's sarcoma-associated herpesvirus entry and trafficking in endothelial cellsGPI anchoring leads to sphingolipid-dependent retention of endocytosed proteins in the recycling endosomal compartment.Modulation of toxin stability by 4-phenylbutyric acid and negatively charged phospholipidsMegalencephalic leukoencephalopathy with subcortical cysts protein-1 modulates endosomal pH and protein trafficking in astrocytes: relevance to MLC disease pathogenesisA novel choline cotransporter sequestration compartment in cholinergic neurons revealed by selective endosomal ablation.Cholesterol-dependent retention of GPI-anchored proteins in endosomes.Clathrin hub expression affects early endosome distribution with minimal impact on receptor sorting and recycling.Receptor trafficking via the perinuclear recycling compartment accompanied by cell division is necessary for permanent neurotensin cell sensitization and leads to chronic mitogen-activated protein kinase activation.Glucose availability and glycolytic metabolism dictate glycosphingolipid levelsThe recycling endosome of Madin-Darby canine kidney cells is a mildly acidic compartment rich in raft componentsDegradation of Alzheimer's amyloid fibrils by microglia requires delivery of ClC-7 to lysosomes.Activation of microglia acidifies lysosomes and leads to degradation of Alzheimer amyloid fibrils.Endocytosed cation-independent mannose 6-phosphate receptor traffics via the endocytic recycling compartment en route to the trans-Golgi network and a subpopulation of late endosomes.A distinct class of intracellular storage vesicles, identified by expression of the glucose transporter GLUT4.An endocytosed TGN38 chimeric protein is delivered to the TGN after trafficking through the endocytic recycling compartment in CHO cells.Transferrin receptor containing the SDYQRL motif of TGN38 causes a reorganization of the recycling compartment but is not targeted to the TGNARF6 targets recycling vesicles to the plasma membrane: insights from an ultrastructural investigationEndocytic sorting of lipid analogues differing solely in the chemistry of their hydrophobic tails.Evidence for nonvectorial, retrograde transferrin trafficking in the early endosomes of HEp2 cells.Defective acidification in human breast tumor cells and implications for chemotherapy.Oligomerized transferrin receptors are selectively retained by a lumenal sorting signal in a long-lived endocytic recycling compartment.The position of lysosomes within the cell determines their luminal pHEndosome acidification and receptor trafficking: bafilomycin A1 slows receptor externalization by a mechanism involving the receptor's internalization motif.Toxicity mechanisms of amphotericin B and its neutralization by conjugation with arabinogalactanStimulation-dependent recycling of integrin beta1 regulated by ARF6 and Rab11.Apical and basolateral endocytic pathways of MDCK cells meet in acidic common endosomes distinct from a nearly-neutral apical recycling endosome.Definition of distinct compartments in polarized Madin-Darby canine kidney (MDCK) cells for membrane-volume sorting, polarized sorting and apical recycling.Lysosomal accumulation of drugs in drug-sensitive MES-SA but not multidrug-resistant MES-SA/Dx5 uterine sarcoma cells.The receptor recycling pathway contains two distinct populations of early endosomes with different sorting functions.Differential targeting of glucosylceramide and galactosylceramide analogues after synthesis but not during transcytosis in Madin-Darby canine kidney cells.The kinetic aspects of intracellular fluorescence labeling with TMA-DPH support the maturation model for endocytosis in L929 cells.Live-cell Microscopy and Fluorescence-based Measurement of Luminal pH in Intracellular Organelles.Basic fibroblast growth factor stimulates surface expression and activity of Na(+)/H(+) exchanger NHE3 via mechanism involving phosphatidylinositol 3-kinase.Two DNA nanomachines map pH changes along intersecting endocytic pathways inside the same cell.Identification of an insulin-responsive, slow endocytic recycling mechanism in Chinese hamster ovary cells.Biogenesis of Salmonella typhimurium-containing vacuoles in epithelial cells involves interactions with the early endocytic pathway.Sterol, protein and lipid trafficking in Chinese hamster ovary cells with Niemann-Pick type C1 defect.
P2860
Q24321358-FABA8015-C6EA-4E50-A135-7ACC6388C7F0Q24651849-D6AB815E-7AC1-4A77-8350-203691F321A9Q27316598-14998C76-1F66-4C62-8C42-88F8AC10BCDAQ28361864-6791BCC4-8D98-4F66-B1A8-20414A5B832DQ28476692-5CD976A4-99DA-496D-9675-F0CFFB9B7948Q30577254-2E9AE956-4028-4295-B71C-0D628B66B450Q33651909-A7ED27DE-5CC8-4864-978B-7DFC361E191EQ33889272-BF9A1FD0-7D53-41A3-84DB-FFA53197931CQ33946749-D3EDC2FC-CFD6-4782-ADA6-9B58939736BCQ34286857-986FB27C-68B9-4426-A90F-5800C2284C71Q34498625-1394C46E-1C16-412C-881C-868578FD2BD3Q34745959-09F4EE97-C5EC-4293-8332-B045DB738707Q34977292-F97E6C9F-76F6-42E7-A6EF-4B975C659C60Q35723681-DB79B8EB-564D-42FD-92FE-E069A2757F04Q35802669-8D6E3F6F-0DAD-4F53-B422-BB8D0DA8578DQ36001945-44F8C60A-3C5B-4725-AC23-603187D288BFQ36255652-2690DC63-DED9-42EB-907F-92ADDCA008B1Q36257746-66CD4615-5799-43E7-84F2-3923F4A50DD0Q36274890-C5B4F1F5-7555-4AE3-8EE8-6061389E45D2Q36293356-DDA51441-9218-4391-8266-48C0B06A1E3CQ36382394-0D569297-48F9-47C0-9869-CE1290B2E067Q36400861-C1DE5DEF-C070-4BD4-8FF8-08A299C12816Q36535694-3E581E46-D28E-494B-A4F2-39C011A7D30FQ36687639-8862AA73-C0DD-4B23-806F-EDC46481197AQ37376783-FE3EAF4A-53FF-4437-9E9E-0FC3DE039F66Q39294022-155F1EB0-B55E-46F8-A2C8-35763CDB9534Q40607521-2C92EAC6-EAE6-40D5-9F81-E2AD65D00B71Q40826327-5E57DB1E-58E3-492D-B9AC-A1F62B0E447BQ40826380-19443A8B-107F-41C2-9338-220EC5EE960AQ40871608-7EF46BC0-93AD-4376-AD66-621673CEFC2AQ40962892-1A848581-E432-4302-8A58-68D5C4812091Q41277635-70819CC3-2672-4903-A2D5-A7D5D57F5524Q41469115-603D9098-7B1F-4680-A738-194D1D5394D7Q41487914-9A8C5B70-31B4-4FE5-AA9A-A8B4769E9E67Q42484860-A2CEBCF7-99CD-4E1A-8500-0032750E60DAQ44810253-FA98710F-D320-49DC-B3B6-CB8A30C526FAQ47891245-C1B5D404-E1B2-4601-A221-90644B2EDDC0Q50117184-9D298709-EC68-4990-8BB7-A6606BE743C2Q51801566-04C8F8BF-BAC1-4F74-BB6B-CA70E5760EDD
P2860
The End2 mutation in CHO cells slows the exit of transferrin receptors from the recycling compartment but bulk membrane recycling is unaffected.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
The End2 mutation in CHO cells ...... brane recycling is unaffected.
@ast
The End2 mutation in CHO cells ...... brane recycling is unaffected.
@en
type
label
The End2 mutation in CHO cells ...... brane recycling is unaffected.
@ast
The End2 mutation in CHO cells ...... brane recycling is unaffected.
@en
prefLabel
The End2 mutation in CHO cells ...... brane recycling is unaffected.
@ast
The End2 mutation in CHO cells ...... brane recycling is unaffected.
@en
P2093
P2860
P356
P1476
The End2 mutation in CHO cells ...... brane recycling is unaffected.
@en
P2093
F R Maxfield
J F Presley
L S Johnson
T E McGraw
P2860
P304
P356
10.1083/JCB.122.6.1231
P407
P577
1993-09-01T00:00:00Z