Separate cis-acting DNA elements of the mouse pro-alpha 1(I) collagen promoter direct expression of reporter genes to different type I collagen-producing cells in transgenic mice
about
FIAT represses ATF4-mediated transcription to regulate bone mass in transgenic miceRNA protein interactions governing expression of the most abundant protein in human body, type I collagenExtra-cellular matrix in vascular networksWnt/beta-catenin signaling is sufficient and necessary for synovial joint formationCthrc1 is a positive regulator of osteoblastic bone formationRegulation of collagen type I in vascular smooth muscle cells by competition between Nkx2.5 and deltaEF1/ZEB1The high mobility group transcription factor Sox8 is a negative regulator of osteoblast differentiationSignalling mediated by the endoplasmic reticulum stress transducer OASIS is involved in bone formationCloning and characterization of a transcription factor that binds to the proximal promoters of the two mouse type I collagen genesc-Krox binds to several sites in the promoter of both mouse type I collagen genes. Structure/function study and developmental expression analysisSP7 inhibits osteoblast differentiation at a late stage in miceEvidence that fibroblasts derive from epithelium during tissue fibrosisNegative regulation of bone formation by the transmembrane Wnt antagonist Kremen-2.Postnatally induced inactivation of Osterix in osteoblasts results in the reduction of bone formation and maintenance.Positive regulation of adult bone formation by osteoblast-specific transcription factor osterix.A gene-targeting approach identifies a function for the first intron in expression of the alpha1(I) collagen gene.Maintenance of Bone Homeostasis by DLL1-Mediated Notch SignalingOsteoblast-specific expression of the fibrous dysplasia (FD)-causing mutation Gsα(R201C) produces a high bone mass phenotype but does not reproduce FD in the mouse.Cbfa1 contributes to the osteoblast-specific expression of type I collagen genes.Leukaemogenesis induced by an activating β-catenin mutation in osteoblasts.Pregnancy-associated plasma protein-A increases osteoblast proliferation in vitro and bone formation in vivoSrc family kinase/abl inhibitor dasatinib suppresses proliferation and enhances differentiation of osteoblasts.Overexpression of Bcl2 in osteoblasts inhibits osteoblast differentiation and induces osteocyte apoptosis.Mutant DLX 3 disrupts odontoblast polarization and dentin formation.EGR1 and EGR2 involvement in vertebrate tendon differentiation.Monitoring collagen transcription by vascular smooth muscle cells in fibrin-based tissue constructs.High bone resorption in adult aging transgenic mice overexpressing cbfa1/runx2 in cells of the osteoblastic lineage.ERRγ is not required for skeletal development but is a RUNX2-dependent negative regulator of postnatal bone formation in male mice.Adjacent DNA sequences modulate Sox9 transcriptional activation at paired Sox sites in three chondrocyte-specific enhancer elements.Transgenic over-expression of growth differentiation factor 11 propeptide in skeleton results in transformation of the seventh cervical vertebra into a thoracic vertebraInhibition of leukemia cell engraftment and disease progression in mice by osteoblastsSmad1 plays an essential role in bone development and postnatal bone formation.Cartilage-specific overexpression of ERRγ results in Chondrodysplasia and reduced chondrocyte proliferation.Osteoblast menin regulates bone mass in vivo.Transgenic mice expressing a ligand-inducible cre recombinase in osteoblasts and odontoblasts: a new tool to examine physiology and disease of postnatal bone and toothRunx2 inhibits chondrocyte proliferation and hypertrophy through its expression in the perichondriumA cell-autonomous requirement for neutral sphingomyelinase 2 in bone mineralization.The Sox2 high mobility group transcription factor inhibits mature osteoblast function in transgenic miceDominance of SOX9 function over RUNX2 during skeletogenesis.The impact of osteoblastic differentiation on osteosarcomagenesis in the mouse.
P2860
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P2860
Separate cis-acting DNA elements of the mouse pro-alpha 1(I) collagen promoter direct expression of reporter genes to different type I collagen-producing cells in transgenic mice
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Separate cis-acting DNA elemen ...... ucing cells in transgenic mice
@ast
Separate cis-acting DNA elemen ...... ucing cells in transgenic mice
@en
type
label
Separate cis-acting DNA elemen ...... ucing cells in transgenic mice
@ast
Separate cis-acting DNA elemen ...... ucing cells in transgenic mice
@en
prefLabel
Separate cis-acting DNA elemen ...... ucing cells in transgenic mice
@ast
Separate cis-acting DNA elemen ...... ucing cells in transgenic mice
@en
P2093
P2860
P356
P1476
Separate cis-acting DNA elemen ...... ucing cells in transgenic mice
@en
P2093
B de Crombrugghe
H Eberspaecher
P2860
P304
P356
10.1083/JCB.129.5.1421
P407
P577
1995-06-01T00:00:00Z