Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
about
A novel Rb- and p300-binding protein inhibits transactivation by MyoDAdenovirus E1A inhibits cardiac myocyte-specific gene expression through its amino terminusInvolvement of cell cycle elements, cyclin-dependent kinases, pRb, and E2F x DP, in B-amyloid-induced neuronal death.Enhanced proliferation of monolayer cultures of embryonic stem (ES) cell-derived cardiomyocytes following acute loss of retinoblastoma.Phosphorylation-dependent degradation of p300 by doxorubicin-activated p38 mitogen-activated protein kinase in cardiac cells.p300 protein as a coactivator of GATA-5 in the transcription of cardiac-restricted atrial natriuretic factor gene.p300 is required for MyoD-dependent cell cycle arrest and muscle-specific gene transcriptionBax-mediated cell death by the Gax homeoprotein requires mitogen activation but is independent of cell cycle activityFOG-2 competes with GATA-4 for transcriptional coactivator p300 and represses hypertrophic responses in cardiac myocytes.Transgenic expression in mouse lung reveals distinct biological roles for the adenovirus type 5 E1A 243- and 289-amino-acid proteinsp21(CIP1) Controls proliferating cell nuclear antigen level in adult cardiomyocytesRepression of c-Myc and inhibition of G1 exit in cells conditionally overexpressing p300 that is not dependent on its histone acetyltransferase activityAntisense-mediated depletion of p300 in human cells leads to premature G1 exit and up-regulation of c-MYC.Cardiac regeneration based on mechanisms of cardiomyocyte proliferation and differentiationCyclin-dependent kinases participate in death of neurons evoked by DNA-damaging agentsCardiac myocyte cell cycle control in development, disease, and regeneration.Caspase inhibition protects against reovirus-induced myocardial injury in vitro and in vivoBinucleation of cardiomyocytes: the transition from a proliferative to a terminally differentiated state.Mechanisms of Cardiomyocyte Proliferation and Differentiation in Development and Regeneration.Cell cycle withdrawal promotes myogenic induction of Akt, a positive modulator of myocyte survival.Reconstitution of cyclin D1-associated kinase activity drives terminally differentiated cells into the cell cycle.Caspase activation and specific cleavage of substrates after coxsackievirus B3-induced cytopathic effect in HeLa cellsEffects of depletion of CREB-binding protein on c-Myc regulation and cell cycle G1-S transition.Apoptotic response to growth factor deprivation involves cooperative interactions between c-Fos and p300.Adenoviral delivery of human CDC5 promotes G2/M progression and cell division in neonatal ventricular cardiomyocytes.Effects of fullerenol C60(OH)24 nanoparticles on a single-dose doxorubicin-induced cardiotoxicity in pigs: an ultrastructural study.
P2860
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P2860
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
@ast
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
@en
type
label
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
@ast
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
@en
prefLabel
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
@ast
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
@en
P2860
P356
P1476
Induction of DNA synthesis and apoptosis in cardiac myocytes by E1A oncoprotein.
@en
P2093
P2860
P304
P356
10.1083/JCB.133.2.325
P407
P577
1996-04-01T00:00:00Z