Cell cycle withdrawal promotes myogenic induction of Akt, a positive modulator of myocyte survival.
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Live cell imaging reveals marked variability in myoblast proliferation and fateIntegrin alpha 2 beta 1 promotes activation of protein phosphatase 2A and dephosphorylation of Akt and glycogen synthase kinase 3 beta.UVA-induced cell cycle progression is mediated by a disintegrin and metalloprotease/epidermal growth factor receptor/AKT/Cyclin D1 pathways in keratinocytesDistinct biological activities of C3 and ADP-ribosyltransferase-deficient C3-E174Qp38 and extracellular signal-regulated kinases regulate the myogenic program at multiple stepsPositive regulation of skeletal myogenesis by R-RasThe Akt-regulated forkhead transcription factor FOXO3a controls endothelial cell viability through modulation of the caspase-8 inhibitor FLIPInvolvement of DNase gamma in apoptosis associated with myogenic differentiation of C2C12 cellsAkt-mediated cardiomyocyte survival pathways are compromised by G alpha q-induced phosphoinositide 4,5-bisphosphate depletionAmphoterin stimulates myogenesis and counteracts the antimyogenic factors basic fibroblast growth factor and S100B via RAGE bindingThrombin induces expression of FGF-2 via activation of PI3K-Akt-Fra-1 signaling axis leading to DNA synthesis and motility in vascular smooth muscle cellsmTORC1 serves ER stress-triggered apoptosis via selective activation of the IRE1-JNK pathwayTrefoil factor 3 stimulates human and rodent pancreatic islet beta-cell replication with retention of function.Interrogating signaling nodes involved in cellular transformations using kinase activity probes.Reactive oxygen species stimulate VEGF production from C(2)C(12) skeletal myotubes through a PI3K/Akt pathway.Insulin signaling leading to proliferation, survival, and membrane ruffling in C2C12 myoblasts.Akt signaling regulates side population cell phenotype via Bcrp1 translocation.Transforming growth factor-beta-activated kinase 1 is an essential regulator of myogenic differentiation.MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptomsActivation of AMPK and inactivation of Akt result in suppression of mTOR-mediated S6K1 and 4E-BP1 pathways leading to neuronal cell death in in vitro models of Parkinson's diseaseADAM12 and alpha9beta1 integrin are instrumental in human myogenic cell differentiation.Immunosuppressive cyclosporin A activates AKT in keratinocytes through PTEN suppression: implications in skin carcinogenesis.IGF-1 prevents ANG II-induced skeletal muscle atrophy via Akt- and Foxo-dependent inhibition of the ubiquitin ligase atrogin-1 expression.Ectopic expression of E2F1 stimulates beta-cell proliferation and function.Insulin-like growth factor-mediated muscle cell survival: central roles for Akt and cyclin-dependent kinase inhibitor p21Endothelial nitric-oxide synthase (eNOS) is activated through G-protein-coupled receptor kinase-interacting protein 1 (GIT1) tyrosine phosphorylation and Src proteinInsulin-like 6 is induced by muscle injury and functions as a regenerative factor.The FOXO3a transcription factor regulates cardiac myocyte size downstream of AKT signaling.Granulocyte colony stimulating factor reduces brain injury in a cardiopulmonary bypass-circulatory arrest model of ischemia in a newborn pigletPTEN positively regulates UVB-induced DNA damage repair.Mechanical stimuli and IL-13 interact at integrin adhesion complexes to regulate expression of smooth muscle myosin heavy chain in airway smooth muscle tissue.Permissive roles of phosphatidyl inositol 3-kinase and Akt in skeletal myocyte maturationGrowth of limb muscle is dependent on skeletal-derived Indian hedgehog.Brahma is required for cell cycle arrest and late muscle gene expression during skeletal myogenesisRNA-seq and metabolomic analyses of Akt1-mediated muscle growth reveals regulation of regenerative pathways and changes in the muscle secretome.Serum withdrawal-induced accumulation of phosphoinositide 3-kinase lipids in differentiating 3T3-L6 myoblasts: distinct roles for Ship2 and PTEN.Deficiency of the cyclin kinase inhibitor p21(WAF-1/CIP-1) promotes apoptosis of activated/memory T cells and inhibits spontaneous systemic autoimmunity.Retinoid X Receptor-selective Signaling in the Regulation of Akt/Protein Kinase B Isoform-specific Expression.Mitophagy is required for mitochondrial biogenesis and myogenic differentiation of C2C12 myoblasts.Unscheduled Akt-triggered activation of cyclin-dependent kinase 2 as a key effector mechanism of apoptin's anticancer toxicity.
P2860
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P2860
Cell cycle withdrawal promotes myogenic induction of Akt, a positive modulator of myocyte survival.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Cell cycle withdrawal promotes ...... modulator of myocyte survival.
@en
Cell cycle withdrawal promotes ...... modulator of myocyte survival.
@nl
type
label
Cell cycle withdrawal promotes ...... modulator of myocyte survival.
@en
Cell cycle withdrawal promotes ...... modulator of myocyte survival.
@nl
prefLabel
Cell cycle withdrawal promotes ...... modulator of myocyte survival.
@en
Cell cycle withdrawal promotes ...... modulator of myocyte survival.
@nl
P2093
P2860
P356
P1476
Cell cycle withdrawal promotes ...... modulator of myocyte survival.
@en
P2093
P2860
P304
P356
10.1128/MCB.19.7.5073
P407
P577
1999-07-01T00:00:00Z