Skeletal muscle cells lacking the retinoblastoma protein display defects in muscle gene expression and accumulate in S and G2 phases of the cell cycle.
about
Cloning and characterization of a novel Kruppel-associated box family transcriptional repressor that interacts with the retinoblastoma gene product, RBMutagenesis of the pRB pocket reveals that cell cycle arrest functions are separable from binding to viral oncoproteinsCells degrade a novel inhibitor of differentiation with E1A-like properties upon exiting the cell cycleA novel Rb- and p300-binding protein inhibits transactivation by MyoDTranscription coactivator TRAP220 is required for PPAR gamma 2-stimulated adipogenesisThe Notch coactivator, MAML1, functions as a novel coactivator for MEF2C-mediated transcription and is required for normal myogenesisA novel synaptobrevin/VAMP homologous protein (VAMP5) is increased during in vitro myogenesis and present in the plasma membraneThe retinoblastoma tumor suppressor protein targets distinct general transcription factors to regulate RNA polymerase III gene expressionCoupling of the cell cycle and myogenesis through the cyclin D1-dependent interaction of MyoD with cdk4A Suv39h-dependent mechanism for silencing S-phase genes in differentiating but not in cycling cells.Molecular basis for impaired muscle differentiation in myotonic dystrophyHMG box transcriptional repressor HBP1 maintains a proliferation barrier in differentiated liver tissueRetinoblastoma protein enhances the fidelity of chromosome segregation mediated by hsHec1pArrest of G(1)-S progression by the p53-inducible gene PC3 is Rb dependent and relies on the inhibition of cyclin D1 transcriptionTumor suppressors: enhancers or suppressors of regeneration?Satellite cells: regenerative mechanisms and applicability in muscular dystrophyPhysical interaction between pRb and cdk9/cyclinT2 complexActivation of MyoD-dependent transcription by cdk9/cyclin T2Synergistic role of E1A-binding proteins and tissue-specific transcription factors in differentiationGenetic interaction between Rb and K-ras in the control of differentiation and tumor suppressionThe winged-helix/forkhead protein myocyte nuclear factor beta (MNF-beta) forms a co-repressor complex with mammalian sin3BInterplay between the retinoblastoma protein and LEK1 specifies stem cells toward the cardiac lineageThe retinoblastoma gene pathway regulates the postmitotic state of hair cells of the mouse inner ear.Cyclin D-cdk4 activity modulates the subnuclear localization and interaction of MEF2 with SRC-family coactivators during skeletal muscle differentiationLamin A/C and emerin are critical for skeletal muscle satellite cell differentiationTranscription factor epiprofin is essential for tooth morphogenesis by regulating epithelial cell fate and tooth numberSubcellular compartmentalization of E2F family members is required for maintenance of the postmitotic state in terminally differentiated muscleEffects of p21(Cip1/Waf1) at both the G1/S and the G2/M cell cycle transitions: pRb is a critical determinant in blocking DNA replication and in preventing endoreduplicationA novel EID-1 family member, EID-2, associates with histone deacetylases and inhibits muscle differentiation.Selection of a dominant negative retinoblastoma protein (RB) inhibiting satellite myoblast differentiation implies an indirect interaction between MyoD and RBA pRb-independent mechanism preserves the postmitotic state in terminally differentiated skeletal muscle cells.MyoD stimulates RB promoter activity via the CREB/p300 nuclear transduction pathway.RETINOBLASTOMA-RELATED protein stimulates cell differentiation in the Arabidopsis root meristem by interacting with cytokinin signaling.The retinoblastoma tumor-suppressor gene, the exception that proves the ruleMaged1, a new regulator of skeletal myogenic differentiation and muscle regeneration.Developmental defects and tumor predisposition in Rb mutant mice.The retinoblastoma gene family in differentiation and development.Satellite cells and the muscle stem cell nicheBAR the door: cancer suppression by amphiphysin-like genesMammalian myotube dedifferentiation induced by newt regeneration extract
P2860
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P2860
Skeletal muscle cells lacking the retinoblastoma protein display defects in muscle gene expression and accumulate in S and G2 phases of the cell cycle.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Skeletal muscle cells lacking ...... d G2 phases of the cell cycle.
@ast
Skeletal muscle cells lacking ...... d G2 phases of the cell cycle.
@en
type
label
Skeletal muscle cells lacking ...... d G2 phases of the cell cycle.
@ast
Skeletal muscle cells lacking ...... d G2 phases of the cell cycle.
@en
prefLabel
Skeletal muscle cells lacking ...... d G2 phases of the cell cycle.
@ast
Skeletal muscle cells lacking ...... d G2 phases of the cell cycle.
@en
P2093
P2860
P356
P1476
Skeletal muscle cells lacking ...... d G2 phases of the cell cycle.
@en
P2093
A B Lassar
B G Novitch
G J Mulligan
P2860
P304
P356
10.1083/JCB.135.2.441
P407
P577
1996-10-01T00:00:00Z