about
Definition of treatment goals for moderate to severe psoriasis: a European consensusThe expression of IL-20 and IL-24 and their shared receptors are increased in rheumatoid arthritis and spondyloarthropathyCharacterization of the interleukin-17 isoforms and receptors in lesional psoriatic skinComparative Analysis of Two Gene-Targeting Approaches Challenges the Tumor-Suppressive Role of the Protein Kinase MK5/PRAKPharmacological undertreatment of coronary risk factors in patients with psoriasis: observational study of the Danish nationwide registriesLow-dose (10-Gy) total skin electron beam therapy for cutaneous T-cell lymphoma: an open clinical study and pooled data analysis.The p38 MAPK regulates IL-24 expression by stabilization of the 3' UTR of IL-24 mRNA.Staphylococcal enterotoxins stimulate lymphoma-associated immune dysregulation.Arachidonic acid metabolism in skin health and disease.Dimethylfumarate specifically inhibits the mitogen and stress-activated kinases 1 and 2 (MSK1/2): possible role for its anti-psoriatic effect.Anti-inflammatory effect of a retrovirus-derived immunosuppressive peptide in mouse models.Treatment of plaque psoriasis with an ointment formulation of the Janus kinase inhibitor, tofacitinib: a Phase 2b randomized clinical trial.Pathway Analysis of Skin from Psoriasis Patients after Adalimumab Treatment Reveals New Early Events in the Anti-Inflammatory Mechanism of Anti-TNF-αIκBζ is a key driver in the development of psoriasis.STAT2 is involved in the pathogenesis of psoriasis by promoting CXCL11 and CCL5 production by keratinocytes.Staphylococcal enterotoxin A (SEA) stimulates STAT3 activation and IL-17 expression in cutaneous T-cell lymphomaPatient Preferences for Topical Psoriasis Treatments are Diverse and Difficult to Predict.Bacterial toxins fuel disease progression in cutaneous T-cell lymphoma.The caspase-cleaved form of LYN mediates a psoriasis-like inflammatory syndrome in mice.Hospital-diagnosed atopic dermatitis and long-term risk of myocardial infarction: a population-based follow-up study.Investigating heredity in cutaneous T-cell lymphoma in a unique cohort of Danish twins.Malignant inflammation in cutaneous T-cell lymphoma-a hostile takeoverLeptin deficiency in mice counteracts imiquimod (IMQ)-induced psoriasis-like skin inflammation while leptin stimulation induces inflammation in human keratinocytes.Tumor necrosis factor-alpha-induced CTACK/CCL27 (cutaneous T-cell-attracting chemokine) production in keratinocytes is controlled by nuclear factor kappaB.Tumor necrosis factor α-mediated induction of interleukin 17C in human keratinocytes is controlled by nuclear factor κB.1alpha,25(OH)(2)D(3) regulates NF-kappaB DNA binding activity in cultured normal human keratinocytes through an increase in IkappaBalpha expression.Associations between functional polymorphisms and response to biological treatment in Danish patients with psoriasis.Identification of key research needs for topical therapy treatment of psoriasis - a consensus paper by the International Psoriasis Council.Jak3, STAT3, and STAT5 inhibit expression of miR-22, a novel tumor suppressor microRNA, in cutaneous T-Cell lymphomaDisease activity in and quality of life of patients with psoriatic arthritis mutilans: the Nordic PAM Study.Characteristics of patients receiving ustekinumab compared with secukinumab for treatment of moderate-to-severe plaque psoriasis - nationwide results from the DERMBIO registry.Tumour necrosis factor-α plays a significant role in the Aldara-induced skin inflammation in mice.Reformulations of well-known active ingredients in the topical treatment of psoriasis vulgaris can improve clinical outcomes for patients.MicroRNA normalization candidates for quantitative reverse-transcriptase polymerase chain reaction in real time in lesional and nonlesional psoriatic skin.STAT5 induces miR-21 expression in cutaneous T cell lymphoma.Low-dose total skin electron beam therapy as a debulking agent for cutaneous T-cell lymphoma: an open-label prospective phase II study.MK2 regulates the early stages of skin tumor promotion.IL-8 and p53 are inversely regulated through JNK, p38 and NF-kappaB p65 in HepG2 cells during an inflammatory response.Immune responses and parasitological observations induced during probiotic treatment with medicinal Trichuris suis ova in a healthy volunteer.Risk of Acute Myocardial Infarction or Stroke in Patients with Mycosis Fungoides and Parapsoriasis.
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Lars Iversen
@ast
Lars Iversen
@en
Lars Iversen
@es
Lars Iversen
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type
label
Lars Iversen
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Lars Iversen
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Lars Iversen
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Lars Iversen
@nl
prefLabel
Lars Iversen
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Lars Iversen
@en
Lars Iversen
@es
Lars Iversen
@nl
P106
P21
P31
P496
0000-0003-1816-4508