ASC Induces Apoptosis via Activation of Caspase-9 by Enhancing Gap Junction-Mediated Intercellular Communication.

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Intercellular competition and the inevitability of multicellular aging.CD69 enhances immunosuppressive function of regulatory T-cells and attenuates colitis by prompting IL-10 production

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ASC Induces Apoptosis via Activation of Caspase-9 by Enhancing Gap Junction-Mediated Intercellular Communication.

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http://www.wikidata.org/entity/Q36240325

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2017 nî lūn-bûn

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2017年の論文

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ASC Induces Apoptosis via Acti ...... d Intercellular Communication.

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ASC Induces Apoptosis via Acti ...... d Intercellular Communication.

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ASC Induces Apoptosis via Acti ...... d Intercellular Communication.

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ASC Induces Apoptosis via Acti ...... d Intercellular Communication.

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ASC Induces Apoptosis via Acti ...... d Intercellular Communication.

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ASC Induces Apoptosis via Acti ...... d Intercellular Communication.

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ASC Induces Apoptosis via Acti ...... d Intercellular Communication.

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Akira Kobayashi

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Chifumi Fujii

http://www.w3.org/2001/XMLSchema#string

Kensuke Ito

http://www.w3.org/2001/XMLSchema#string

Michiko Takeoka

http://www.w3.org/2001/XMLSchema#string

Nagisa Okada

http://www.w3.org/2001/XMLSchema#string

Shigeaki Hida

http://www.w3.org/2001/XMLSchema#string

Shin-Ichi Miyagawa

http://www.w3.org/2001/XMLSchema#string

Shun'ichiro Taniguchi

http://www.w3.org/2001/XMLSchema#string

Takashi Sakaizawa

http://www.w3.org/2001/XMLSchema#string

Tomio Matsumura

http://www.w3.org/2001/XMLSchema#string

P2860

ASC, a novel 22-kDa protein, aggregates during apoptosis of human promyelocytic leukemia HL-60 cells

ASC is an activating adaptor for NF-kappa B and caspase-8-dependent apoptosis

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

ASC is a Bax adaptor and regulates the p53-Bax mitochondrial apoptosis pathway

Cadherin-11 mediates contact inhibition of locomotion during Xenopus neural crest cell migration

TMS1, a novel proapoptotic caspase recruitment domain protein, is a target of methylation-induced gene silencing in human breast cancers

Activation of a caspase-9-mediated apoptotic pathway by subcellular redistribution of the novel caspase recruitment domain protein TMS1

Effects of methylation on expression of TMS1/ASC in human breast cancer cells

A molecular platform in neurons regulates inflammation after spinal cord injury

Contact inhibition and high cell density deactivate the mammalian target of rapamycin pathway, thus suppressing the senescence program

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e0169340

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scholarly article

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10.1371/JOURNAL.PONE.0169340

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English

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Masato Kitazawa

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2017-01-05T00:00:00Z

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28056049

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5215782

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ASC Induces Apoptosis via Activation of Caspase-9 by Enhancing Gap Junction-Mediated Intercellular Communication.

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P2860

P2860

ASC Induces Apoptosis via Activation of Caspase-9 by Enhancing Gap Junction-Mediated Intercellular Communication.

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