Changes in atherosclerotic plaques induced by inhalation of diesel exhaust. - Wikidata - awgldk - TriplyDB

Changes in atherosclerotic plaques induced by inhalation of diesel exhaust.

Changes in atherosclerotic plaques induced by inhalation of diesel exhaust.

http://www.wikidata.org/entity/Q36244638

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The Apoe(-/-) mouse model: a suitable model to study cardiovascular and respiratory diseases in the context of cigarette smoke exposure and harm reduction Mechanisms linking traffic-related air pollution and atherosclerosis Circulating factors induce coronary endothelial cell activation following exposure to inhaled diesel exhaust and nitrogen dioxide in humans: evidence from a novel translational in vitro model Particle-induced pulmonary acute phase response correlates with neutrophil influx linking inhaled particles and cardiovascular risk Engine exhaust particulate and gas phase contributions to vascular toxicity Diesel exhaust particulate increases the size and complexity of lesions in atherosclerotic mice Altered nitric oxide bioavailability contributes to diesel exhaust inhalation-induced cardiovascular dysfunction in man Contribution of lung macrophages to the inflammatory responses induced by exposure to air pollutants Air pollution, vascular disease and thrombosis: linking clinical data and pathogenic mechanisms.A case-control study of medium-term exposure to ambient nitrogen dioxide pollution and hospitalization for stroke CD36-dependent 7-ketocholesterol accumulation in macrophages mediates progression of atherosclerosis in response to chronic air pollution exposure.In utero exposure to diesel exhaust air pollution promotes adverse intrauterine conditions, resulting in weight gain, altered blood pressure, and increased susceptibility to heart failure in adult mice.Exposure to diesel exhaust up-regulates iNOS expression in ApoE knockout mice Nitrative stress, oxidative stress and plasma endothelin levels after inhalation of particulate matter and ozone.The biological effects of subacute inhalation of diesel exhaust following addition of cerium oxide nanoparticles in atherosclerosis-prone mice.Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.Heme oxygenase-1 protects endothelial cells from the toxicity of air pollutant chemicals Endothelial inflammatory transcriptional responses to an altered plasma exposome following inhalation of diesel emissions Identification of chemical components of combustion emissions that affect pro-atherosclerotic vascular responses in mice.CD36 mediates endothelial dysfunction downstream of circulating factors induced by O3 exposure.Surface area-dependence of gas-particle interactions influences pulmonary and neuroinflammatory outcomes.In utero and early life exposure to diesel exhaust air pollution increases adult susceptibility to heart failure in mice.Cardiovascular effects of particulate air pollution exposure: time course and underlying mechanisms.From particles to patients: oxidative stress and the cardiovascular effects of air pollution.Oxidatively damaged DNA in animals exposed to particles.Public health and components of particulate matter: the changing assessment of black carbon.The role of oxidative stress in the cardiovascular actions of particulate air pollution.Diesel engine exhaust: basis for occupational exposure limit value.Atherosclerosis and vasomotor dysfunction in arteries of animals after exposure to combustion-derived particulate matter or nanomaterials.Foam cell formation by particulate matter (PM) exposure: a review.Oxidative Stress and Cardiovascular Risk: Obesity, Diabetes, Smoking, and Pollution: Part 3 of a 3-Part Series.Diesel exhaust inhalation induces heat shock protein 70 expression in vivo.In Utero Exposure of Hyperlipidemic Mice to Diesel Exhaust: Lack of Effects on Atherosclerosis in Adult Offspring Fed a Regular Chow Diet.Inhalation of diesel exhaust does not exacerbate cardiac hypertrophy or heart failure in two mouse models of cardiac hypertrophy.Early kidney damage induced by subchronic exposure to PM2.5 in rats.A finite element investigation on plaque vulnerability in realistic healthy and atherosclerotic human coronary arteries.Exposure to traffic-generated air pollutants mediates alterations in brain microvascular integrity in wildtype mice on a high-fat diet.Transcriptional profiling of human bronchial epithelial cell BEAS-2B exposed to diesel and biomass ultrafine particles.

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Changes in atherosclerotic plaques induced by inhalation of diesel exhaust.

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Changes in atherosclerotic plaques induced by inhalation of diesel exhaust.

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J.ATHEROSCLEROSIS.2011.02.019

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Atherosclerosis

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Changes in atherosclerotic plaques induced by inhalation of diesel exhaust.

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Cornelis van Breemen

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Grace Yang

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Hisashi Suzuki

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Michael E Rosenfeld

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Ni Bai

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Stephan F van Eeden

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Takashi Kido

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Terrance J Kavanagh

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P2860

Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species

Ambient air pollution and atherosclerosis in Los Angeles

Atherosclerosis — An Inflammatory Disease

Comparative effects of inhaled diesel exhaust and ambient fine particles on inflammation, atherosclerosis, and vascular dysfunction

Particulate matter air pollution and cardiovascular disease: An update to the scientific statement from the American Heart Association

Air pollution and cardiovascular disease: a statement for healthcare professionals from the Expert Panel on Population and Prevention Science of the American Heart Association

Reduction in fine particulate air pollution and mortality: Extended follow-up of the Harvard Six Cities study

Cytokines involved in the systemic inflammatory response induced by exposure to particulate matter air pollutants (PM(10))

Exposure to traffic and the onset of myocardial infarction

Particulate air pollution induces progression of atherosclerosis

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Joel D. Kaufman

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2011-03-02T00:00:00Z

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National Institute for Occupational Safety and Health

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atherosclerosis

cardiovascular disease

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4631611

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