In vivo analysis of intestinal permeability following hemorrhagic shock

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The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.Enteral tranexamic acid attenuates vasopressor resistance and changes in α1-adrenergic receptor expression in hemorrhagic shock.

P2860

Q40304817-0222E9A7-5D12-41B3-A425-D0AE38BA4B32 Q46813964-5AD6B780-6AAC-4727-BE20-21F3C1D0BEC6

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In vivo analysis of intestinal permeability following hemorrhagic shock

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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In vivo analysis of intestinal permeability following hemorrhagic shock

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In vivo analysis of intestinal permeability following hemorrhagic shock

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Item

label

In vivo analysis of intestinal permeability following hemorrhagic shock

@ast

In vivo analysis of intestinal permeability following hemorrhagic shock

@en

prefLabel

In vivo analysis of intestinal permeability following hemorrhagic shock

@ast

In vivo analysis of intestinal permeability following hemorrhagic shock

@en

P1476

In vivo analysis of intestinal permeability following hemorrhagic shock

@en

P2093

Erik B Kistler

http://www.w3.org/2001/XMLSchema#string

Marisol Chang

http://www.w3.org/2001/XMLSchema#string

Michael Richter

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Rafi Mazor

http://www.w3.org/2001/XMLSchema#string

Tom Alsaigh

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P2860

Breakdown of mucin as barrier to digestive enzymes in the ischemic rat small intestine

Protease activity increases in plasma, peritoneal fluid, and vital organs after hemorrhagic shock in rats

Vascular permeability, vascular hyperpermeability and angiogenesis

Enzymatic digestion of the milk protein beta-casein releases potent chemotactic peptide(s) for monocytes and macrophages.

Analysis of leukocyte rolling and migration--using inhibitors in the undisturbed microcirculation of the rat mesentery--on inflammatory stimulation

Disruption of the mucosal barrier during gut ischemia allows entry of digestive enzymes into the intestinal wall.

Impaired small-bowel barrier integrity in the presence of lumenal pancreatic digestive enzymes leads to circulatory shock

On the absorption of bacterial endotoxin from the gastro-intestinal tract of the normal and shocked animal

The gut is a motor of organ system dysfunction.

Generation of in vivo activating factors in the ischemic intestine by pancreatic enzymes.

P304

287-295

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P31

scholarly article

P356

10.5492/WJCCM.V4.I4.287

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P478

http://www.w3.org/2001/XMLSchema#string

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2015-11-04T00:00:00Z

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283618054

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P698

26557479

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P932

4631874

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In vivo analysis of intestinal permeability following hemorrhagic shock

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P2860

P2860

In vivo analysis of intestinal permeability following hemorrhagic shock

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type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P932