An efficient and versatile system for acute and chronic modulation of renal tubular function in transgenic mice. - Wikidata - awgldk - TriplyDB

An efficient and versatile system for acute and chronic modulation of renal tubular function in transgenic mice.

An efficient and versatile system for acute and chronic modulation of renal tubular function in transgenic mice.

http://www.wikidata.org/entity/Q36247097

about

Renal tubular HIF-2α expression requires VHL inactivation and causes fibrosis and cysts Loss of Renal Tubular PGC-1α Exacerbates Diet-Induced Renal Steatosis and Age-Related Urinary Sodium Excretion in Mice An Efficient and Versatile System for Visualization and Genetic Modification of Dopaminergic Neurons in Transgenic Mice Nephron-specific deletion of the prorenin receptor causes a urine concentration defect.GATA2 regulates body water homeostasis through maintaining aquaporin 2 expression in renal collecting ducts.MDM2 prevents spontaneous tubular epithelial cell death and acute kidney injury.mTORC1 maintains renal tubular homeostasis and is essential in response to ischemic stress.Acute Vhl gene inactivation induces cardiac HIF-dependent erythropoietin gene expression.Tubular overexpression of transforming growth factor-beta1 induces autophagy and fibrosis but not mesenchymal transition of renal epithelial cells Epithelial Notch signaling regulates interstitial fibrosis development in the kidneys of mice and humans Inducible kidney-specific Sgk1 knockout mice show a salt-losing phenotype.Renal tubular NEDD4-2 deficiency causes NCC-mediated salt-dependent hypertension Transformation of the fallopian tube secretory epithelium leads to high-grade serous ovarian cancer in Brca;Tp53;Pten models.Synchronized renal tubular cell death involves ferroptosis.Strict control of transgene expression in a mouse model for sensitive biological applications based on RMCE compatible ES cells.Disruption of the endothelin A receptor in the nephron causes mild fluid volume expansion.PGC1α drives NAD biosynthesis linking oxidative metabolism to renal protection.Hyperkalemic hypertension-associated cullin 3 promotes WNK signaling by degrading KLHL3 Nedd4-2 modulates renal Na+-Cl- cotransporter via the aldosterone-SGK1-Nedd4-2 pathway.Tubulovascular cross-talk by vascular endothelial growth factor a maintains peritubular microvasculature in kidney.Epithelial Sodium Channel-Mediated Sodium Transport Is Not Dependent on the Membrane-Bound Serine Protease CAP2/Tmprss4.Generation and Characterisation of a Pax8-CreERT2 Transgenic Line and a Slc22a6-CreERT2 Knock-In Line for Inducible and Specific Genetic Manipulation of Renal Tubular Epithelial Cells.Hypoxia-inducible factor signaling in the development of kidney fibrosis Cystogenesis and elongated primary cilia in Tsc1-deficient distal convoluted tubules.Glomerular parietal epithelial cells contribute to adult podocyte regeneration in experimental focal segmental glomerulosclerosis It's Totally Tubular....Riding The New Wave of Ovarian Cancer Research.Transforming growth factor β-1 stimulates profibrotic epithelial signaling to activate pericyte-myofibroblast transition in obstructive kidney fibrosis.The serine protease hepsin mediates urinary secretion and polymerisation of Zona Pellucida domain protein uromodulin.Possible role for nephron-derived angiotensinogen in angiotensin-II dependent hypertension.Paracrine Wnt1 Drives Interstitial Fibrosis without Inflammation by Tubulointerstitial Cross-Talk.mTORC1-mediated inhibition of polycystin-1 expression drives renal cyst formation in tuberous sclerosis complex.Epithelial cell TGFβ signaling induces acute tubular injury and interstitial inflammation.Renal Deletion of 12 kDa FK506-Binding Protein Attenuates Tacrolimus-Induced Hypertension.Inhibition of histone deacetylase 6 activity reduces cyst growth in polycystic kidney disease Renal tubular epithelial cell prorenin receptor regulates blood pressure and sodium transport.Diethylstilbestrol induces vaginal adenosis by disrupting SMAD/RUNX1-mediated cell fate decision in the Müllerian duct epithelium.Ovarian hormones through Wnt signalling regulate the growth of human and mouse ovarian cancer initiating lesions Hyperactivation of Nrf2 in early tubular development induces nephrogenic diabetes insipidus.Lineage-tracing methods and the kidney.Epithelial-mesenchymal transition (EMT) in kidney fibrosis: fact or fantasy?

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An efficient and versatile system for acute and chronic modulation of renal tubular function in transgenic mice.

http://www.wikidata.org/entity/Q36247097

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

An efficient and versatile sys ...... r function in transgenic mice.

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An efficient and versatile sys ...... r function in transgenic mice.

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type

label

An efficient and versatile sys ...... r function in transgenic mice.

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An efficient and versatile sys ...... r function in transgenic mice.

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prefLabel

An efficient and versatile sys ...... r function in transgenic mice.

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An efficient and versatile sys ...... r function in transgenic mice.

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Nature Medicine

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Adam B Glick

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Anna Jauch

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David J Kwiatkowski

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Dean W Felsher

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Hermann Bujard

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Hermann-Josef Gröne

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Jürgen Horst

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Kai Schönig

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Magnus von Knebel Doeberitz

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Manja Bode

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P2860

PAX8, a human paired box gene: isolation and expression in developing thyroid, kidney and Wilms' tumors

Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

Exploring the sequence space for tetracycline-dependent transcriptional activators: novel mutations yield expanded range and sensitivity

Generalized lacZ expression with the ROSA26 Cre reporter strain

A mouse model of TSC1 reveals sex-dependent lethality from liver hemangiomas, and up-regulation of p70S6 kinase activity in Tsc1 null cells

Follicular cells of the thyroid gland require Pax8 gene function

Transcriptional activation by tetracyclines in mammalian cells

Pax8, a murine paired box gene expressed in the developing excretory system and thyroid gland

A germ-line Tsc1 mutation causes tumor development and embryonic lethality that are similar, but not identical to, those caused by Tsc2 mutation in mice

Reversible tumorigenesis by MYC in hematopoietic lineages.

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979-984

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10.1038/NM.1865

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9

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14

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23191159

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18724376

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3446847

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