Dysregulated interferon-gamma responses during lethal cytomegalovirus brain infection of IL-10-deficient mice.
about
Neuropathogenesis of congenital cytomegalovirus infection: disease mechanisms and prospects for interventionImmunobiology of congenital cytomegalovirus infection of the central nervous system—the murine cytomegalovirus modelTregs Modulate Lymphocyte Proliferation, Activation, and Resident-Memory T-Cell Accumulation within the Brain during MCMV InfectionPersistent humoral immune responses in the CNS limit recovery of reactivated murine cytomegalovirusThe PD-1: PD-L1 pathway promotes development of brain-resident memory T cells following acute viral encephalitis.Reduced lymphocyte infiltration during cytomegalovirus brain infection of interleukin-10-deficient mice.Glial cells suppress postencephalitic CD8+ T lymphocytes through PD-L1.Excess neutrophil infiltration during cytomegalovirus brain infection of interleukin-10-deficient miceInterleukin-10 repletion suppresses pro-inflammatory cytokines and decreases liver pathology without altering viral replication in murine cytomegalovirus (MCMV)-infected IL-10 knockout mice.Infiltrating regulatory B cells control neuroinflammation following viral brain infectionFunctional characterization of mouse spinal cord infiltrating CD8+ lymphocytes.Memory T cells persisting in the brain following MCMV infection induce long-term microglial activation via interferon-γ.Chronic reactive gliosis following regulatory T cell depletion during acute MCMV encephalitis.Glial cell activation, recruitment, and survival of B-lineage cells following MCMV brain infectionCytomegalovirus infection and interferon-gamma modulate major histocompatibility complex class I expression on neural stem cells.Prolonged microglial cell activation and lymphocyte infiltration following experimental herpes encephalitisModulation of Microglial Cell Fcγ Receptor Expression Following Viral Brain Infection.Ethanol-Induced TLR4/NLRP3 Neuroinflammatory Response in Microglial Cells Promotes Leukocyte Infiltration Across the BBB.Nitrosative damage during retrovirus infection-induced neuropathic pain.Reactive glia promote development of CD103+ CD69+ CD8+ T-cells through programmed cell death-ligand 1 (PD-L1).
P2860
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P2860
Dysregulated interferon-gamma responses during lethal cytomegalovirus brain infection of IL-10-deficient mice.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Dysregulated interferon-gamma ...... ction of IL-10-deficient mice.
@ast
Dysregulated interferon-gamma ...... ction of IL-10-deficient mice.
@en
type
label
Dysregulated interferon-gamma ...... ction of IL-10-deficient mice.
@ast
Dysregulated interferon-gamma ...... ction of IL-10-deficient mice.
@en
prefLabel
Dysregulated interferon-gamma ...... ction of IL-10-deficient mice.
@ast
Dysregulated interferon-gamma ...... ction of IL-10-deficient mice.
@en
P2093
P2860
P1433
P1476
Dysregulated interferon-gamma ...... ction of IL-10-deficient mice.
@en
P2093
Genya Gekker
James R Lokensgard
Joseph M Palmquist
Shuxian Hu
Thomas Bakken
P2860
P304
P356
10.1016/J.VIRUSRES.2007.05.022
P577
2007-07-12T00:00:00Z