Quantitative liver proteomics identifies FGF19 targets that couple metabolism and proliferation.

about

Peroxisomal Acyl-CoA Oxidase Type 1: Anti-Inflammatory and Anti-Aging Properties with a Special Emphasis on Studies with LPS and Argan Oil as a Model Transposable to Aging.

P2860

Q54986126-073A514B-B398-41CC-9F2C-1890F1C3F468

P2860

Quantitative liver proteomics identifies FGF19 targets that couple metabolism and proliferation.

Item

http://www.wikidata.org/entity/Q36274229

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

Quantitative liver proteomics ...... metabolism and proliferation.

@ast

Quantitative liver proteomics ...... metabolism and proliferation.

@en

type

Item

label

Quantitative liver proteomics ...... metabolism and proliferation.

@ast

Quantitative liver proteomics ...... metabolism and proliferation.

@en

prefLabel

Quantitative liver proteomics ...... metabolism and proliferation.

@ast

Quantitative liver proteomics ...... metabolism and proliferation.

@en

P1476

Quantitative liver proteomics ...... e metabolism and proliferation

@en

P2093

Alexandra Milona

http://www.w3.org/2001/XMLSchema#string

Boudewijn M T Burgering

http://www.w3.org/2001/XMLSchema#string

Vittoria Massafra

http://www.w3.org/2001/XMLSchema#string

P2860

Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis

beta ig-h3: a transforming growth factor-beta-responsive gene encoding a secreted protein that inhibits cell attachment in vitro and suppresses the growth of CHO cells in nude mice

FGF19 as a postprandial, insulin-independent activator of hepatic protein and glycogen synthesis

A mouse model of hepatocellular carcinoma: ectopic expression of fibroblast growth factor 19 in skeletal muscle of transgenic mice

HER-2 overexpression differentially alters transforming growth factor-beta responses in luminal versus mesenchymal human breast cancer cells

Dysregulated metabolism contributes to oncogenesis

Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

Global quantification of mammalian gene expression control

Convergence of genes implicated in Alzheimer's disease on the cerebral cholesterol shuttle: APP, cholesterol, lipoproteins, and atherosclerosis

Regulation of amphiregulin gene expression by β-catenin signaling in human hepatocellular carcinoma cells: a novel crosstalk between FGF19 and the EGFR system

P304

e0171185

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PONE.0171185

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Harmjan Vos

Saskia van Mil

P577

2017-02-08T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28178326

http://www.w3.org/2001/XMLSchema#string

P932

5298232

http://www.w3.org/2001/XMLSchema#string

Quantitative liver proteomics identifies FGF19 targets that couple metabolism and proliferation.

about

P2860

P2860

Quantitative liver proteomics identifies FGF19 targets that couple metabolism and proliferation.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932