Don't lose heart--therapeutic value of apoptosis prevention in the treatment of cardiovascular disease. - Wikidata - awgldk - TriplyDB

Don't lose heart--therapeutic value of apoptosis prevention in the treatment of cardiovascular disease.

Don't lose heart--therapeutic value of apoptosis prevention in the treatment of cardiovascular disease.

http://www.wikidata.org/entity/Q36275682

about

miR-30 regulates mitochondrial fission through targeting p53 and the dynamin-related protein-1 pathway Gene Expression Signatures of Coronary Heart Disease Small molecules, big effects: the role of microRNAs in regulation of cardiomyocyte death Heart repair and regeneration: recent insights from zebrafish studies Transcription factor Foxo3a prevents apoptosis by regulating calcium through the apoptosis repressor with caspase recruitment domain Leptin protects H9c2 rat cardiomyocytes from H2O2-induced apoptosis Mitofusin 1 is negatively regulated by microRNA 140 in cardiomyocyte apoptosis.Leptin protects cardiomyocytes from serum-deprivation-induced apoptosis by increasing anti-oxidant defence.Cellular stress responses: cell survival and cell death Sex-related resistance to myocardial ischemia-reperfusion injury is associated with high constitutive ARC expression Wearable multi-channel microelectrode membranes for elucidating electrophysiological phenotypes of injured myocardium.Cardiac remodeling in obesity.Trade-offs between cancer and other diseases: do they exist and influence longevity?Electrocardiogram signals to assess zebrafish heart regeneration: implication of long QT intervals.Cardioprotective effects of voluntary exercise in a rat model: role of matrix metalloproteinase-2.HSP70 inhibits stress-induced cardiomyocyte apoptosis by competitively binding to FAF1.Flexible microelectrode arrays to interface epicardial electrical signals with intracardial calcium transients in zebrafish hearts.Electrical and Mechanical Strategies to Enable Cardiac Repair and Regeneration.Cell death in the cardiovascular system.Morphological aspects of apoptosis in heart diseases Cell death in health and disease The paradigm of postconditioning to protect the heart.Features of cardiomyocyte proliferation and its potential for cardiac regeneration.The therapeutic effect of rosuvastatin on cardiac remodelling from hypertrophy to fibrosis during the end-stage hypertension in rats.Distinct mechanisms of cardiomyocyte apoptosis induced by doxorubicin and hypoxia converge on mitochondria and are inhibited by Bcl-xL Mechanisms of exercise-induced cardioprotection.Mitochondrial calcium homeostasis as potential target for mitochondrial medicine.Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury.Protective approaches against myocardial ischemia reperfusion injury Effect of PKC inhibitor on experimental autoimmune myocarditis in Lewis rats Antiapoptotic actions of methyl gallate on neonatal rat cardiac myocytes exposed to H2O2 Apoptosis: a mapped path to cell death Biological targets for isatin and its analogues: Implications for therapy.Plasma concentrations of tumour necrosis factor-alpha, tumour necrosis factor-related apoptosis-inducing ligand, and FasLigand/CD95L in patients with Chagas cardiomyopathy correlate with left ventricular dysfunction.Suppression of phosphorylated MAPK and caspase 3 by carbon dioxide.

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P2860

Don't lose heart--therapeutic value of apoptosis prevention in the treatment of cardiovascular disease.

http://www.wikidata.org/entity/Q36275682

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2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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Don't lose heart--therapeutic ...... ent of cardiovascular disease.

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J.1582-4934.2005.TB00492.X

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Journal of Cellular and Molecular Medicine

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Don't lose heart--therapeutic ...... ment of cardiovascular disease

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Angela M Duffy

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Janice L V Reeve

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Timothy O'Brien

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P2860

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases

X-linked IAP is a direct inhibitor of cell-death proteases

Solution structure of human survivin and its binding interface with Smac/Diablo

Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Abeta-induced cell death

Essential myosin light chain as a target for caspase-3 in failing myocardium

BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

Negative regulation of the Apaf-1 apoptosome by Hsp70

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

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apoptotic process

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