HMGB1 mediates cognitive impairment in sepsis survivors. - Wikidata - awgldk - TriplyDB

HMGB1 mediates cognitive impairment in sepsis survivors.

HMGB1 mediates cognitive impairment in sepsis survivors.

http://www.wikidata.org/entity/Q36283400

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Neuroimmune Function and the Consequences of Alcohol Exposure Mechanisms of chemotherapy-induced behavioral toxicities Understanding Long-Term Outcomes Following Sepsis: Implications and Challenges Mechanisms of neuroimmune gene induction in alcoholism Amyloid β: one of three danger-associated molecules that are secondary inducers of the proinflammatory cytokines that mediate Alzheimer's disease α7 nicotinic acetylcholine receptor signaling inhibits inflammasome activation by preventing mitochondrial DNA release.Surgery upregulates high mobility group box-1 and disrupts the blood-brain barrier causing cognitive dysfunction in aged rats.Systemic inflammation and the brain: novel roles of genetic, molecular, and environmental cues as drivers of neurodegeneration.HMGB1 mediates splenomegaly and expansion of splenic CD11b+ Ly-6C(high) inflammatory monocytes in murine sepsis survivors.Brain region-specific alterations in the gene expression of cytokines, immune cell markers and cholinergic system components during peripheral endotoxin-induced inflammation.Sirt1 restrains lung inflammasome activation in a murine model of sepsis.Blood-Brain Barrier Deterioration and Hippocampal Gene Expression in Polymicrobial Sepsis: An Evaluation of Endothelial MyD88 and the Vagus Nerve Cecal Ligation and Puncture Results in Long-Term Central Nervous System Myeloid Inflammation.High-mobility group box 1 mediates persistent splenocyte priming in sepsis survivors: evidence from a murine model Low molecular weight heparin prevents lipopolysaccharide induced-hippocampus-dependent cognitive impairments in mice.High Mobility Group Box Protein 1 (HMGB1): The Prototypical Endogenous Danger Molecule Neural circuitry and immunity.HMGB1 mediates anemia of inflammation in murine sepsis survivors.Could Biomarkers Direct Therapy for the Septic Patient?The Enemy within: Innate Surveillance-Mediated Cell Death, the Common Mechanism of Neurodegenerative Disease.Protein kinase G oxidation is a major cause of injury during sepsis.Treatment with anti-HMGB1 monoclonal antibody does not affect lupus nephritis in MRL/lpr mice.Systemic HMGB1 Neutralization Prevents Postoperative Neurocognitive Dysfunction in Aged Rats.Acteoside improves survival in cecal ligation and puncture-induced septic mice via blocking of high mobility group box 1 release.Preclinical models of overwhelming sepsis implicate the neural system that encodes contextual fear memory.JAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation.Neuroimmune basis of alcoholic brain damage.Co-morbidity and systemic inflammation as drivers of cognitive decline: new experimental models adopting a broader paradigm in dementia research.The brain at risk: the sepsis syndrome and lessons from preclinical experiments.Beneficial effects of ulinastatin on gut barrier function in sepsis Immunity and Inflammation in Epilepsy.Regulation of Posttranslational Modifications of HMGB1 During Immune Responses Progress in Clinical Encapsulated Islet Xenotransplantation.Stimulation of the α7 nicotinic acetylcholine receptor protects against neuroinflammation after tibia fracture and endotoxemia in mice Brain Barrier Breakdown as a Cause and Consequence of Neuroinflammation in Sepsis.Understanding brain dysfunction in sepsis.Caspr2-reactive antibody cloned from a mother of an ASD child mediates an ASD-like phenotype in mice.Is there a role for high mobility group box 1 and the receptor for advanced glycation end products in the genesis of long-term cognitive impairment in sepsis survivors?Early antagonism of cerebral high mobility group box-1 protein is benefit for sepsis induced brain injury.Response to “HMGB1 Mediates Cognitive Impairment in Sepsis Survivors”.

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P2860

HMGB1 mediates cognitive impairment in sepsis survivors.

http://www.wikidata.org/entity/Q36283400

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2012年學術文章

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2012年學術文章

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name

HMGB1 mediates cognitive impairment in sepsis survivors.

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HMGB1 mediates cognitive impairment in sepsis survivors.

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type

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HMGB1 mediates cognitive impairment in sepsis survivors.

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HMGB1 mediates cognitive impairment in sepsis survivors.

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prefLabel

HMGB1 mediates cognitive impairment in sepsis survivors.

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HMGB1 mediates cognitive impairment in sepsis survivors.

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MOLMED.2012.00195

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Molecular Medicine

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HMGB1 mediates cognitive impairment in sepsis survivors.

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Betty Diamond

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Mahendar Ochani

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Meghan Dancho

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S I Valdes-Ferrer

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Sangeeta S Chavan

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Sergio Robbiati

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P2860

Long-term cognitive impairment and functional disability among survivors of severe sepsis

Recombinant HMGB1 with cytokine-stimulating activity

Interleukin-1 receptor accessory protein organizes neuronal synaptogenesis as a cell adhesion molecule

Nonresolving inflammation

Synaptic scaling mediated by glial TNF-alpha

Tumor necrosis factor-alpha triggers a cytokine cascade yielding postoperative cognitive decline

Behavioral and functional analysis of mouse phenotype: SHIRPA, a proposed protocol for comprehensive phenotype assessment.

HMGB1 as a late mediator of lethal systemic inflammation.

Reversing established sepsis with antagonists of endogenous high-mobility group box 1.

A genomic storm in critically injured humans

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