Pharmacological dissection and distribution of NaN/Nav1.9, T-type Ca2+ currents, and mechanically activated cation currents in different populations of DRG neurons.
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Piezo proteins are pore-forming subunits of mechanically activated channels.Piezo1 and Piezo2 are essential components of distinct mechanically activated cation channelsTouch sense: functional organization and molecular determinants of mechanosensitive receptorsSensory mechanotransduction at membrane-matrix interfacesRedox-Dependent Modulation of T-Type Ca(2+) Channels in Sensory Neurons Contributes to Acute Anti-Nociceptive Effect of Substance PNeurotransmitter modulation of small-conductance Ca2+-activated K+ channels by regulation of Ca2+ gatingEthanol inhibition of a T-type Ca²+ channel through activity of protein kinase CImmunohistological demonstration of CaV3.2 T-type voltage-gated calcium channel expression in soma of dorsal root ganglion neurons and peripheral axons of rat and mouse.Presynaptic Cav3.2 channels regulate excitatory neurotransmission in nociceptive dorsal horn neuronsTTA-P2 is a potent and selective blocker of T-type calcium channels in rat sensory neurons and a novel antinociceptive agent.T-type voltage-gated calcium channels as targets for the development of novel pain therapies.Upregulation of the T-type calcium current in small rat sensory neurons after chronic constrictive injury of the sciatic nerveNeuropathic pain: role for presynaptic T-type channels in nociceptive signaling.Voltage-activated calcium currents in octopus cells of the mouse cochlear nucleus.CRMP-2 peptide mediated decrease of high and low voltage-activated calcium channels, attenuation of nociceptor excitability, and anti-nociception in a model of AIDS therapy-induced painful peripheral neuropathyNav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation.1,4-Dihydropyridine derivatives with T-type calcium channel blocking activity attenuate inflammatory and neuropathic pain.Antisense-mediated knockdown of Na(V)1.8, but not Na(V)1.9, generates inhibitory effects on complete Freund's adjuvant-induced inflammatory pain in rat.Molecular mechanisms of mechanotransduction in mammalian sensory neurons.Inflammatory signals enhance piezo2-mediated mechanosensitive currents.Morphology and intrinsic excitability of regenerating sensory and motor neurons grown on a line micropatternDiffering neurophysiologic mechanosensory input from glabrous and hairy skin in juvenile rats.TRPA1 has a key role in the somatic pro-nociceptive actions of hydrogen sulfidePiezo proteins: regulators of mechanosensation and other cellular processesUp-regulation of tetrodotoxin-sensitive sodium currents by prostaglandin E₂ in type-4 rat dorsal root ganglion cells.Redox and nitric oxide-mediated regulation of sensory neuron ion channel functionFunctional properties and toxin pharmacology of a dorsal root ganglion sodium channel viewed through its voltage sensorsExpression and Regulation of Cav3.2 T-Type Calcium Channels during Inflammatory Hyperalgesia in Mouse Dorsal Root Ganglion Neurons.Upregulation of T-type Ca2+ channels in long-term diabetes determines increased excitability of a specific type of capsaicin-insensitive DRG neurons.Animal toxins can alter the function of Nav1.8 and Nav1.9.Inflammatory mediators increase Nav1.9 current and excitability in nociceptors through a coincident detection mechanism.Tetrodotoxin-resistant voltage-dependent sodium channels in identified muscle afferent neuronsInflammatory mediator bradykinin increases population of sensory neurons expressing functional T-type Ca(2+) channels.Regulation of Piezo2 Mechanotransduction by Static Plasma Membrane Tension in Primary Afferent NeuronsEvolutionary Specialization of Tactile Perception in Vertebrates.Nociceptive neurons differentially express fast and slow T-type Ca²⁺ currents in different types of diabetic neuropathy.T-type calcium channels in chronic pain: mouse models and specific blockers.Transduction and encoding sensory information by skin mechanoreceptors.Regulation of the T-type Ca(2+) channel Cav3.2 by hydrogen sulfide: emerging controversies concerning the role of H2 S in nociception.Activation of transient receptor potential ankyrin 1 induces CGRP release from spinal cord synaptosomes.
P2860
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P2860
Pharmacological dissection and distribution of NaN/Nav1.9, T-type Ca2+ currents, and mechanically activated cation currents in different populations of DRG neurons.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Pharmacological dissection and ...... nt populations of DRG neurons.
@ast
Pharmacological dissection and ...... nt populations of DRG neurons.
@en
type
label
Pharmacological dissection and ...... nt populations of DRG neurons.
@ast
Pharmacological dissection and ...... nt populations of DRG neurons.
@en
prefLabel
Pharmacological dissection and ...... nt populations of DRG neurons.
@ast
Pharmacological dissection and ...... nt populations of DRG neurons.
@en
P2093
P2860
P356
P1476
Pharmacological dissection and ...... nt populations of DRG neurons.
@en
P2093
Bertrand Coste
Marcel Crest
Patrick Delmas
P2860
P356
10.1085/JGP.200609665
P577
2007-01-01T00:00:00Z