RET is a potential tumor suppressor gene in colorectal cancer. - Wikidata - awgldk - TriplyDB

RET is a potential tumor suppressor gene in colorectal cancer.

RET is a potential tumor suppressor gene in colorectal cancer.

http://www.wikidata.org/entity/Q36301488

about

Genomic diversity of colorectal cancer: Changing landscape and emerging targets Common fragile sites: genomic hotspots of DNA damage and carcinogenesis Differences in DNA methylation signatures reveal multiple pathways of progression from adenoma to colorectal cancer NTRK3 is a potential tumor suppressor gene commonly inactivated by epigenetic mechanisms in colorectal cancer HPIP is upregulated in colorectal cancer and regulates colorectal cancer cell proliferation, apoptosis and invasion.Oncodomains: A protein domain-centric framework for analyzing rare variants in tumor samples.Context is everything for dependence receptors in colorectal cancer.Functional variants at the 11q13 risk locus for breast cancer regulate cyclin D1 expression through long-range enhancers.Analysis of RET promoter CpG island methylation using methylation-specific PCR (MSP), pyrosequencing, and methylation-sensitive high-resolution melting (MS-HRM): impact on stage II colon cancer patient outcome.Altered RECQ Helicase Expression in Sporadic Primary Colorectal Cancers.miR-181b functions as an oncomiR in colorectal cancer by targeting PDCD4 Hyper-Methylated Loci Persisting from Sessile Serrated Polyps to Serrated Cancers.Dependence receptors and colorectal cancer.Epigenetic silencing of TPM2 contributes to colorectal cancer progression upon RhoA activation.Promoter CpG island methylation of RET predicts poor prognosis in stage II colorectal cancer patients Maintaining the unmethylated state.The GDNF Family: A Role in Cancer?Targeted molecular profiling of rare genetic alterations in colorectal cancer using next-generation sequencing.The Generation and Validation of a 20-Genes Model Influencing the Prognosis of Colorectal Cancer.NIT1 suppresses tumour proliferation by activating the TGFβ1-Smad2/3 signalling pathway in colorectal cancer.Next-generation sequencing analysis of receptor-type tyrosine kinase genes in surgically resected colon cancer: identification of gain-of-function mutations in the RET proto-oncogene.The immunohistochemical profile of granular cell (Abrikossoff) tumor suggests an endomesenchymal origin.MiR-1260b inhibitor enhances the chemosensitivity of colorectal cancer cells to fluorouracil by targeting PDCD4/IGF1 CHSY1 promoted proliferation and suppressed apoptosis in colorectal cancer through regulation of the NFκB and/or caspase-3/7 signaling pathway

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RET is a potential tumor suppressor gene in colorectal cancer.

http://www.wikidata.org/entity/Q36301488

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

@zh-hant

name

RET is a potential tumor suppressor gene in colorectal cancer.

@ast

RET is a potential tumor suppressor gene in colorectal cancer.

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type

label

RET is a potential tumor suppressor gene in colorectal cancer.

@ast

RET is a potential tumor suppressor gene in colorectal cancer.

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prefLabel

RET is a potential tumor suppressor gene in colorectal cancer.

@ast

RET is a potential tumor suppressor gene in colorectal cancer.

@en

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RET is a potential tumor suppressor gene in colorectal cancer.

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D Il Park

http://www.w3.org/2001/XMLSchema#string

J Wang

http://www.w3.org/2001/XMLSchema#string

K D Tsuchiya

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P Welcsh

http://www.w3.org/2001/XMLSchema#string

R Fausel

http://www.w3.org/2001/XMLSchema#string

S Dzieciatkowski

http://www.w3.org/2001/XMLSchema#string

S Kanngurn

http://www.w3.org/2001/XMLSchema#string

W M Grady

http://www.w3.org/2001/XMLSchema#string

Y Luo

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P2860

The RET proto-oncogene induces apoptosis: a novel mechanism for Hirschsprung disease

The full oncogenic activity of Ret/ptc2 depends on tyrosine 539, a docking site for phospholipase Cgamma

GFRalpha-2 and GFRalpha-3 are two new receptors for ligands of the GDNF family

Persephin, a novel neurotrophic factor related to GDNF and neurturin

Quantitative high-throughput analysis of DNA methylation patterns by base-specific cleavage and mass spectrometry

Inactivation of the UNC5C Netrin-1 receptor is associated with tumor progression in colorectal malignancies

The consensus coding sequences of human breast and colorectal cancers

The GDNF family: signalling, biological functions and therapeutic value

Artemin is oncogenic for human mammary carcinoma cells

RET tyrosine kinase signaling in development and cancer

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2037-2047

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scholarly article

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10.1038/ONC.2012.225

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16

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32

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2012-07-02T00:00:00Z

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228103022

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colorectal cancer

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