Analysis of Venezuelan equine encephalitis virus capsid protein function in the inhibition of cellular transcription
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Mosquito immunity against arbovirusesAlphavirus Infection: Host Cell Shut-Off and Inhibition of Antiviral ResponsesThe C-terminal 18 Amino Acid Region of Dengue Virus NS5 Regulates its Subcellular Localization and Contains a Conserved Arginine Residue Essential for Infectious Virus ProductionIRES-dependent replication of Venezuelan equine encephalitis virus makes it highly attenuated and incapable of replicating in mosquito cellsA viral RNA structural element alters host recognition of nonself RNAPrinciples of antidote pharmacology: an update on prophylaxis, post-exposure treatment recommendations and research initiatives for biological agentsThe ubiquitin proteasome system plays a role in venezuelan equine encephalitis virus infectionSelective Inhibitor of Nuclear Export (SINE) Compounds Alter New World Alphavirus Capsid Localization and Reduce Viral Replication in Mammalian CellsModulation of GSK-3β activity in Venezuelan equine encephalitis virus infectionMutation of the N-Terminal Region of Chikungunya Virus Capsid Protein: Implications for Vaccine DesignThe role of interferon antagonist, non-structural proteins in the pathogenesis and emergence of arboviruses.Random insertion mutagenesis of sindbis virus nonstructural protein 2 and selection of variants incapable of downregulating cellular transcriptionIdentification of Western equine encephalitis virus structural proteins that confer protection after DNA vaccination.Identification of Interactions between Sindbis Virus Capsid Protein and Cytoplasmic vRNA as Novel Virulence DeterminantsEnhancement of protein expression by alphavirus replicons by designing self-replicating subgenomic RNAs.Genetic and anatomic determinants of enzootic Venezuelan equine encephalitis virus infection of Culex (Melanoconion) taeniopus.IRES-driven expression of the capsid protein of the Venezuelan equine encephalitis virus TC-83 vaccine strain increases its attenuation and safetyEncapsidation of host-derived factors correlates with enhanced infectivity of Sindbis virusDifferentiation of neurons restricts Arbovirus replication and increases expression of the alpha isoform of IRF-7.Venezuelan equine encephalitis virus variants lacking transcription inhibitory functions demonstrate highly attenuated phenotype.A DNA vaccine for venezuelan equine encephalitis virus delivered by intramuscular electroporation elicits high levels of neutralizing antibodies in multiple animal models and provides protective immunity to mice and nonhuman primates.Design of chimeric alphaviruses with a programmed, attenuated, cell type-restricted phenotype.Conservation of a packaging signal and the viral genome RNA packaging mechanism in alphavirus evolution.Chimeric Chikungunya viruses are nonpathogenic in highly sensitive mouse models but efficiently induce a protective immune response.The combined use of alphavirus replicons and pseudoinfectious particles for the discovery of antivirals derived from natural products.Noncapped Alphavirus Genomic RNAs and Their Role during Infection.New World and Old World Alphaviruses Have Evolved to Exploit Different Components of Stress Granules, FXR and G3BP Proteins, for Assembly of Viral Replication Complexes.Neurotropic arboviruses induce interferon regulatory factor 3-mediated neuronal responses that are cytoprotective, interferon independent, and inhibited by Western equine encephalitis virus capsid.A five-amino-acid deletion of the eastern equine encephalitis virus capsid protein attenuates replication in mammalian systems but not in mosquito cells.Eastern and Venezuelan equine encephalitis viruses differ in their ability to infect dendritic cells and macrophages: impact of altered cell tropism on pathogenesis.Similarities and differences in antagonism of neuron alpha/beta interferon responses by Venezuelan equine encephalitis and Sindbis alphaviruses.Rubella virus capsid protein: a small protein with big functions.Non-encapsidation activities of the capsid proteins of positive-strand RNA viruses.The role of the IKK complex in viral infections.Host translation shutoff mediated by non-structural protein 2 is a critical factor in the antiviral state resistance of Venezuelan equine encephalitis virus.The SD1 Subdomain of Venezuelan Equine Encephalitis Virus Capsid Protein Plays a Critical Role in Nucleocapsid and Particle Assembly.Both RIG-I and MDA5 detect alphavirus replication in concentration-dependent mode.Interferon-stimulated poly(ADP-Ribose) polymerases are potent inhibitors of cellular translation and virus replicationVenezuelan equine encephalitis virus nsP2 protein regulates packaging of the viral genome into infectious virions.Pseudoinfectious Venezuelan equine encephalitis virus: a new means of alphavirus attenuation.
P2860
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P2860
Analysis of Venezuelan equine encephalitis virus capsid protein function in the inhibition of cellular transcription
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Analysis of Venezuelan equine ...... tion of cellular transcription
@ast
Analysis of Venezuelan equine ...... tion of cellular transcription
@en
type
label
Analysis of Venezuelan equine ...... tion of cellular transcription
@ast
Analysis of Venezuelan equine ...... tion of cellular transcription
@en
prefLabel
Analysis of Venezuelan equine ...... tion of cellular transcription
@ast
Analysis of Venezuelan equine ...... tion of cellular transcription
@en
P2093
P2860
P50
P356
P1433
P1476
Analysis of Venezuelan equine ...... tion of cellular transcription
@en
P2093
Natalia Garmashova
Svetlana Atasheva
Wenli Kang
P2860
P304
13552-13565
P356
10.1128/JVI.01576-07
P407
P577
2007-10-03T00:00:00Z