The C5a receptor has a key role in immune complex glomerulonephritis in complement factor H-deficient mice
about
Complement regulation in renal disease modelsLoss of CD11b exacerbates murine complement-mediated tubulointerstitial nephritis.CD11b is protective in complement-mediated immune complex glomerulonephritis.C5a Induces the Synthesis of IL-6 and TNF-α in Rat Glomerular Mesangial Cells through MAPK Signaling Pathways.Abrogation of immune complex glomerulonephritis by native carboxypeptidase and pharmacological antagonism of the C5a receptor.Getting over our Immune-Complex - C5a receptor blockade is the answer.Many drugs for many targets: novel treatments for complement-mediated glomerular disease.
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The C5a receptor has a key role in immune complex glomerulonephritis in complement factor H-deficient mice
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The C5a receptor has a key rol ...... lement factor H-deficient mice
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The C5a receptor has a key rol ...... lement factor H-deficient mice
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The C5a receptor has a key rol ...... lement factor H-deficient mice
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The C5a receptor has a key rol ...... lement factor H-deficient mice
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The C5a receptor has a key rol ...... lement factor H-deficient mice
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The C5a receptor has a key rol ...... lement factor H-deficient mice
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The C5a receptor has a key rol ...... lement factor H-deficient mice
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Jessy J Alexander
Lee Chaves
Richard J Quigg
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10.1038/KI.2012.249
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2012-07-25T00:00:00Z