Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis - Wikidata - awgldk - TriplyDB

Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

http://www.wikidata.org/entity/Q36344904

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Recent Progress in Stem Cell Therapy for Diabetic Nephropathy FSGS: Diagnosis and Diagnostic Work-Up Tipping the balance from angiogenesis to fibrosis in CKD Primary proximal tubule injury leads to epithelial cell cycle arrest, fibrosis, vascular rarefaction, and glomerulosclerosis Severe renal mass reduction impairs recovery and promotes fibrosis after AKI Improved mitochondrial function underlies the protective effect of pirfenidone against tubulointerstitial fibrosis in 5/6 nephrectomized rats Sphingosine Kinase 2 Deficiency Attenuates Kidney Fibrosis via IFN-γ.The proximal tubule is the primary target of injury and progression of kidney disease: role of the glomerulotubular junction.Progression after AKI: Understanding Maladaptive Repair Processes to Predict and Identify Therapeutic Treatments.Therapeutic translation in acute kidney injury: the epithelial/endothelial axis Hypoxia, HIF, and Associated Signaling Networks in Chronic Kidney Disease.Potential Role of Serum and Urinary Biomarkers in Diagnosis and Prognosis of Diabetic Nephropathy.Urinary Tamm-Horsfall protein, albumin, vitamin D-binding protein, and retinol-binding protein as early biomarkers of chronic kidney disease in dogs Using 2-photon microscopy to understand albuminuria.Kidney pericytes: roles in regeneration and fibrosis.Blood kidney injury molecule-1 is a biomarker of acute and chronic kidney injury and predicts progression to ESRD in type I diabetes Sonic hedgehog is a novel tubule-derived growth factor for interstitial fibroblasts after kidney injury.Chronic kidney disease: Mesoamerican nephropathy--new clues to the cause.CCAAT-enhancer binding protein delta (C/EBPδ) attenuates tubular injury and tubulointerstitial fibrogenesis during chronic obstructive nephropathy.Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.Epithelial cells as active player in fibrosis: findings from an in vitro model.Transcriptome analysis of proximal tubular cells (HK-2) exposed to urines of type 1 diabetes patients at risk of early progressive renal function decline.Significance of urinary full-length megalin in patients with IgA nephropathy.Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.KIM-1-mediated phagocytosis reduces acute injury to the kidney.Hyperbaric oxygen therapy (HBOT) suppresses biomarkers of cell stress and kidney injury in diabetic mice Specific macrophage subtypes influence the progression of rhabdomyolysis-induced kidney injury Analysis of nephron composition and function in the adult zebrafish kidney Mild ischemic injury leads to long-term alterations in the kidney: amelioration by spironolactone administration.Hepatocyte Nuclear Factor-1β Induces Redifferentiation of Dedifferentiated Tubular Epithelial Cells.Simultaneous deletion of Bax and Bak is required to prevent apoptosis and interstitial fibrosis in obstructive nephropathy Meclizine Preconditioning Protects the Kidney Against Ischemia-Reperfusion Injury.TMIGD1 is a novel adhesion molecule that protects epithelial cells from oxidative cell injury Can we target tubular damage to prevent renal function decline in diabetes?Repeated administration of low-dose cisplatin in mice induces fibrosis.Cellular mechanisms of tissue fibrosis. 3. Novel mechanisms of kidney fibrosis.Proximal tubule-specific overexpression of netrin-1 suppresses acute kidney injury-induced interstitial fibrosis and glomerulosclerosis through suppression of IL-6/STAT3 signaling Kidney tubules: intertubular, vascular, and glomerular cross-talk.Kielin/chordin-like protein attenuates both acute and chronic renal injury Regenerative medicine for the kidney: stem cell prospects & challenges.

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Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

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Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

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Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

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Kidney International

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Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

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Benjamin D Humphreys

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Chang Wang

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Gabriela Campanholle

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Ivica Grgic

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Joseph V Bonventre

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Takaharu Ichimura

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Vanesa Bijol

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Venkata S Sabbisetti

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P2860

c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation

Six2 defines and regulates a multipotent self-renewing nephron progenitor population throughout mammalian kidney development

Generation and evolution of atubular glomeruli in the progression of renal disorders.

Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis

Acute kidney injury: a springboard for progression in chronic kidney disease

Renal-associated TLR2 mediates ischemia/reperfusion injury in the kidney

Tubular overexpression of transforming growth factor-beta1 induces autophagy and fibrosis but not mesenchymal transition of renal epithelial cells

HMGB1 contributes to kidney ischemia reperfusion injury.

Regression of microalbuminuria in type 1 diabetes is associated with lower levels of urinary tubular injury biomarkers, kidney injury molecule-1, and N-acetyl-β-D-glucosaminidase.

The Ngal reporter mouse detects the response of the kidney to injury in real time.

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10.1038/KI.2012.20

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2

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