A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
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Ripped to deathDeath receptor-ligand systems in cancer, cell death, and inflammationUse of high frequency ultrasound to monitor cervical lymph node alterations in miceRelocalization of apoptosis-inducing factor in photoreceptor apoptosis induced by retinal detachment in vivoThe effector T cell response to influenza infectionFas-mediated apoptosis regulates the composition of peripheral alphabeta T cell repertoire by constitutively purging out double negative T cellsSystemic FasL and TRAIL neutralisation reduce leishmaniasis induced skin ulceration.Major histocompatibility complex class I-specific cytolytic T cells, derived from gld mice, lacking Thy-1, CD4, and CD8.Comparison of immunoperoxidase staining with indirect immunofluorescence, ELISA, and Western blotting assays for detecting anti-HTLV-I antibodies in systemic lupus erythematosus.Human TCR-alpha beta+ CD4- CD8- T cells can derive from CD8+ T cells and display an inflammatory effector phenotypeLessons from BXSB and related mouse models.Genetic basis of the complex pathological manifestations of collagen disease: lessons from MRL/lpr and related mouse models.The nuclear factor NF-kappaB pathway in inflammationThe Yaa gene abrogates the major histocompatibility complex association of murine lupus in (NZB x BXSB)F1 hybrid miceOverexpression of src family gene for tyrosine-kinase p59fyn in CD4-CD8- T cells of mice with a lymphoproliferative disorderT-cell receptor genes in autoimmune mice: T-cell subsets have unexpected T-cell receptor gene programs.Studies of the pathogenesis of angioimmunoblastic lymphadenopathyTargeting Fas in osteoresorptive disorders.Activation-induced cell death: the controversial role of Fas and Fas ligand in immune privilege and tumour counterattack.Triggers for anti-chromatin autoantibody production in SLE.Lupus Nephritis: Animal Modeling of a Complex Disease Syndrome Pathology.Double negative (DN) αβ T cells: misperception and overdue recognitionSequence and organization of the mouse poly (ADP-ribose) polymerase gene.Infiltration of circulating myeloid cells through CD95L contributes to neurodegeneration in miceA role for tumor necrosis factor-alpha in remodeling the splenic marginal zone during Leishmania donovani infection.Analysis of granulomatous arteritis in MRL/Mp autoimmune disease mice bearing lymphoproliferative genes. The use of mouse genetics to dissociate the development of arteritis and glomerulonephritisC-reactive protein as a marker for inflammatory bowel disease.Increased protection from vaccinia virus infection in mice genetically prone to lymphoproliferative disorders.The regulation and activation of lupus-associated B cells.Impaired apoptosis and immune senescence - cause or effect?Inhibition of apoptosis by BCL2 prevents leukemic transformation of a murine myelodysplastic syndromeActivation of natural killer T cells in NZB/W mice induces Th1-type immune responses exacerbating lupusAbnormalities induced by the mutant gene, lpr. Patterns of disease and expression of murine leukemia viruses in SJL/J mice homozygous and heterozygous for lpr.A new allele of the lpr locus, lprcg, that complements the gld gene in induction of lymphadenopathy in the mouseCharacterization of Lyt-2-, L3T4- class I-specific cytolytic clones in C3H-gld/gld mice. Implications for functions of accessory molecules and programmed development.Genetic analysis of autoimmune gld mice. I. Identification of a restriction fragment length polymorphism closely linked to the gld mutation within a conserved linkage group.Cytotoxic T cells deficient in both functional fas ligand and perforin show residual cytolytic activity yet lose their capacity to induce lethal acute graft-versus-host diseaseTumor necrosis factor sustains the generalized lymphoproliferative disorder (gld) phenotype.Impaired clearance of apoptotic cells promotes synergy between atherogenesis and autoimmune diseaseMultiple endogenous xenotropic and mink cell focus-forming murine leukemia virus-related transcripts are induced by polyclonal immune activators
P2860
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P2860
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年学术文章
@wuu
1984年学术文章
@zh-cn
1984年学术文章
@zh-hans
1984年学术文章
@zh-my
1984年学术文章
@zh-sg
1984年學術文章
@yue
1984年學術文章
@zh
1984年學術文章
@zh-hant
name
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
@ast
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
@en
type
label
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
@ast
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
@en
prefLabel
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
@ast
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
@en
P2093
P2860
P356
P1476
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.
@en
P2093
P2860
P356
10.1084/JEM.159.1.1
P407
P577
1984-01-01T00:00:00Z