Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
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A role for IgG immune complexes during infection with the intracellular pathogen Leishmania.iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant miceAmastigote Load and Cell Surface Phenotype of Infected Cells from Lesions and Lymph Nodes of Susceptible and Resistant Mice Infected with Leishmania majorConstitutive and adaptive detoxification of nitric oxide in Escherichia coli. Role of nitric-oxide dioxygenase in the protection of aconitaseRoles of endogenous gamma interferon and macrophage microbicidal mechanisms in host response to chemotherapy in experimental visceral leishmaniasisGranule-dependent killing of Toxoplasma gondii by CD8+ T cellsReactive oxygen intermediates, nitrite and IFN-gamma in Indian visceral leishmaniasisMycobacterium indicus pranii (Mw) re-establishes host protective immune response in Leishmania donovani infected macrophages: critical role of IL-12Response to reactive nitrogen intermediates in Mycobacterium tuberculosis: induction of the 16-kilodalton alpha-crystallin homolog by exposure to nitric oxide donorsRapid healing of cutaneous leishmaniasis by high-frequency electrocauterization and hydrogel wound care with or without DAC N-055: a randomized controlled phase IIa trial in KabulMice lacking inducible nitric oxide synthase demonstrate impaired killing of Porphyromonas gingivalisIdentification of nitric oxide synthase as a protective locus against tuberculosisThe immunology of Leishmania/HIV co-infection.Collective nitric oxide production provides tissue-wide immunity during Leishmania infection.Fibroblasts as host cells in latent leishmaniosisMolecular blocking of CD23 supports its role in the pathogenesis of arthritis.Proteophosophoglycans regurgitated by Leishmania-infected sand flies target the L-arginine metabolism of host macrophages to promote parasite survivalEvolution of lesion formation, parasitic load, immune response, and reservoir potential in C57BL/6 mice following high- and low-dose challenge with Leishmania major.Skin-resident CD4+ T cells protect against Leishmania major by recruiting and activating inflammatory monocytes.Alcoholic Fractions F5 and F6 from Withania somnifera Leaves Show a Potent Antileishmanial and Immunomodulatory Activities to Control Experimental Visceral Leishmaniasis.Phlebotomus papatasi saliva inhibits protein phosphatase activity and nitric oxide production by murine macrophages.CD40 signaling in macrophages induces activity against an intracellular pathogen independently of gamma interferon and reactive nitrogen intermediatesIn vivo formation of electron paramagnetic resonance-detectable nitric oxide and of nitrotyrosine is not impaired during murine leishmaniasis.T-Cell hyporesponsiveness induced by activated macrophages through nitric oxide production in mice infected with Mycobacterium tuberculosis.Subunit vaccination of mice against new world cutaneous leishmaniasis: comparison of three proteins expressed in amastigotes and six adjuvants.Leishmania major reaches distant cutaneous sites where it persists transiently while persisting durably in the primary dermal site and its draining lymph node: a study with laboratory mice.T cells that react to the immunodominant Leishmania major LACK antigen prevent early dissemination of the parasite in susceptible BALB/c miceEarly enhanced Th1 response after Leishmania amazonensis infection of C57BL/6 interleukin-10-deficient mice does not lead to resolution of infection.Expression of inducible nitric oxide synthase in skin lesions of patients with american cutaneous leishmaniasis.Progressive visceral leishmaniasis is driven by dominant parasite-induced STAT6 activation and STAT6-dependent host arginase 1 expression.An in vitro model of antibody-enhanced killing of the intracellular parasite Leishmania amazonensisIdentification of a compensatory mutant (lpg2-REV) of Leishmania major able to survive as amastigotes within macrophages without LPG2-dependent glycoconjugates and its significance to virulence and immunization strategiesOxidative stress markers are increased since early stages of infection in syphilitic patients.What is the role of nitric oxide in murine and human host defense against tuberculosis?Current knowledge.Predominant role of endothelial nitric oxide synthase in vascular endothelial growth factor-induced angiogenesis and vascular permeability.Role of cytokines in the innate immune response to intracellular pathogensNitric oxide-peroxynitrite-poly(ADP-ribose) polymerase pathway in the skin.Mapping the genes for susceptibility and response to Leishmania tropica in mouseDifferent genetic control of cutaneous and visceral disease after Leishmania major infection in mice.Deletion of IL-4 receptor alpha on dendritic cells renders BALB/c mice hypersusceptible to Leishmania major infection.
P2860
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P2860
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
@ast
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
@en
type
label
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
@ast
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
@en
prefLabel
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
@ast
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
@en
P2093
P2860
P356
P1476
Reactivation of latent leishmaniasis by inhibition of inducible nitric oxide synthase.
@en
P2093
Donhauser N
Röllinghoff M
P2860
P304
P356
10.1084/JEM.183.4.1501
P407
P577
1996-04-01T00:00:00Z