Mode of action: neurotoxicity induced by thyroid hormone disruption during development--hearing loss resulting from exposure to PHAHs.
about
Developmental neurotoxicity of polybrominated diphenyl ether (PBDE) flame retardantsEnvironmental exposure to organochlorine pesticides and deficits in cochlear status in childrenIPCS framework for analyzing the relevance of a noncancer mode of action for humansPutative adverse outcome pathways relevant to neurotoxicityMinding the calcium store: Ryanodine receptor activation as a convergent mechanism of PCB toxicitySerum PCB concentrations and cochlear function in 12-year-old children.Short-term exposure to triclosan decreases thyroxine in vivo via upregulation of hepatic catabolism in Young Long-Evans ratsHuman cost burden of exposure to endocrine disrupting chemicals. A critical review.Prenatal and postnatal serum PCB concentrations and cochlear function in children at 45 months of age.Effects of developmental exposure to polychlorinated biphenyls and/or polybrominated diphenyl ethers on cochlear functionInternational STakeholder NETwork (ISTNET): creating a developmental neurotoxicity (DNT) testing road map for regulatory purposesTiered High-Throughput Screening Approach to Identify Thyroperoxidase Inhibitors Within the ToxCast Phase I and II Chemical Libraries.Effects of prenatal exposure to diesel exhaust particles on postnatal development, behavior, genotoxicity and inflammation in miceDevelopmental exposure to polychlorinated biphenyls interferes with experience-dependent dendritic plasticity and ryanodine receptor expression in weanling ratsExcitatory and inhibitory synaptic transmission is differentially influenced by two ortho-substituted polychlorinated biphenyls in the hippocampal slice preparationThyroid-disrupting chemicals: interpreting upstream biomarkers of adverse outcomes.Comparative hazard characterization in food toxicology.Developmental neurotoxicity testing: a path forward.Prenatal stress and development: beyond the single cause and effect paradigm.Environmental factors for the development of fetal urinary malformations.DPOAEs in infants developmentally exposed to PCBs show two differently time spaced exposure sensitive windows.Developmental exposure to PCBs alters the activation of the auditory cortex in response to GABAA antagonism.The effects of amitrole on thyroglobulin and iodide uptake in FRTL-5 cells.Thyroid Allostasis-Adaptive Responses of Thyrotropic Feedback Control to Conditions of Strain, Stress, and Developmental Programming.PCB exposure and cochlear function at age 6 years.In vitro perturbations of targets in cancer hallmark processes predict rodent chemical carcinogenesis.Brominated flame retardants as possible endocrine disrupters.Anthropogenic flank attack on polar bears: interacting consequences of climate warming and pollutant exposure
P2860
Q24678057-05E949FA-BBAB-44DF-98B4-A2885584C822Q27313926-6745DBED-D922-42A8-9C87-78A00468B5BCQ27921844-EB36607F-5A8D-4729-B28A-9595E3FA39E7Q28392038-0974B06D-59FD-4DCA-858F-EB4A4A748DBFQ28393648-93C13996-724B-45F4-AE12-0231072FD83EQ28394848-7FDA0AB5-EE00-4C3B-A268-DA6DDC230517Q28396967-8758CD09-6897-404E-B41E-FCB839821F55Q30234405-CCD2F5A6-D581-4D34-A8D9-EDF21DAC2539Q30426417-AA956D89-59C1-4C7A-B83F-42BBB1503C08Q30462007-4CF4B541-0AAA-4704-9EDF-2B8CE856C1BCQ35024730-984F4EFA-106F-4C3B-B7D4-EBE7D71F8582Q35925542-04939F4D-0210-4F1C-9117-0F72AF0F3A12Q36570891-2E956B65-D8E0-4B37-AC2C-95CD24317118Q37142339-C0172EA5-AA98-462C-BADF-0D912BEB8E0DQ37219791-9CF1D759-D0C4-4AA9-952A-4F277FFF2BB7Q37278918-CA66F490-B2B9-4B4B-A393-F07A78344357Q37543221-B8D1944B-FD1D-40F6-94FD-B68EC9D9634CQ38038302-71AD4BDA-4D2F-4167-85DA-5BDD8FB8A9B5Q38160820-17FAA568-21D0-4EDB-8893-64EE839ACB54Q38182013-F8B96ACF-13DF-4196-8FB4-1B64CB0152CAQ39547338-E7D680F3-213C-4F0C-9565-995DC60B34ACQ39596745-88C68359-89E4-4F1B-A686-C56E3A2C252CQ39645678-9AACFCEE-E400-4B83-AB4E-2FF9C34A0227Q40492427-C951D105-67E5-41A5-AA48-9D235A99AA34Q40533275-67AC3BA3-2022-4D09-A239-927386FB58BAQ46054824-61759FB3-FF36-4995-98C0-98A0F0A4D19AQ52585517-6D70403D-6237-4EA7-AF8D-DC3D300610D7Q57898752-FF0A8F7C-CB3E-4D93-955D-A3DE3A0A5527
P2860
Mode of action: neurotoxicity induced by thyroid hormone disruption during development--hearing loss resulting from exposure to PHAHs.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Mode of action: neurotoxicity ...... ulting from exposure to PHAHs.
@ast
Mode of action: neurotoxicity ...... ulting from exposure to PHAHs.
@en
type
label
Mode of action: neurotoxicity ...... ulting from exposure to PHAHs.
@ast
Mode of action: neurotoxicity ...... ulting from exposure to PHAHs.
@en
prefLabel
Mode of action: neurotoxicity ...... ulting from exposure to PHAHs.
@ast
Mode of action: neurotoxicity ...... ulting from exposure to PHAHs.
@en
P2860
P1476
Mode of action: neurotoxicity ...... ulting from exposure to PHAHs.
@en
P2093
R Thomas Zoeller
P2860
P304
P356
10.1080/10408440591007304
P407
P577
2005-10-01T00:00:00Z