about
Meeting report: moving upstream-evaluating adverse upstream end points for improved risk assessment and decision-makingThyroid-hormone-disrupting chemicals: evidence for dose-dependent additivity or synergismDevelopmental neurotoxicity of pyrethroid insecticides: critical review and future research needsCurrent perspectives on the use of alternative species in human health and ecological hazard assessmentsPutative adverse outcome pathways relevant to neurotoxicityShort-term exposure to triclosan decreases thyroxine in vivo via upregulation of hepatic catabolism in Young Long-Evans ratsTranscriptional response of rat frontal cortex following acute in vivo exposure to the pyrethroid insecticides permethrin and deltamethrint4 workshop report: Pathways of ToxicityCorrelation of tissue concentrations of the pyrethroid bifenthrin with neurotoxicity in the rat.Evidence for dose-additive effects of pyrethroids on motor activity in ratsSetting exposure standards: a decision process.An empirical approach to sufficient similarity: combining exposure data and mixtures toxicology data.Evaluation of the ToxRTool's ability to rate the reliability of toxicological data for human health hazard assessments.A qualitative retrospective analysis of positive control data in developmental neurotoxicity studies.Using ToxCast™ Data to Reconstruct Dynamic Cell State Trajectories and Estimate Toxicological Points of Departure.Overview: Using mode of action and life stage information to evaluate the human relevance of animal toxicity data.Screening methods for thyroid hormone disruptors.Optimal design for the precise estimation of an interaction threshold: the impact of exposure to a mixture of 18 polyhalogenated aromatic hydrocarbonsThe effects of triclosan on puberty and thyroid hormones in male Wistar rats.Developmental triclosan exposure decreases maternal and neonatal thyroxine in rats.The human toxome projectIn utero and lactational exposure to bisphenol A, in contrast to ethinyl estradiol, does not alter sexually dimorphic behavior, puberty, fertility, and anatomy of female LE rats.International STakeholder NETwork (ISTNET): creating a developmental neurotoxicity (DNT) testing road map for regulatory purposesAdditivity of pyrethroid actions on sodium influx in cerebrocortical neurons in primary culture.Integrated Model of Chemical Perturbations of a Biological Pathway Using 18 In Vitro High-Throughput Screening Assays for the Estrogen Receptor.Workgroup report: incorporating in vitro alternative methods for developmental neurotoxicity into international hazard and risk assessment strategiesDevelopmental triclosan exposure decreases maternal, fetal, and early neonatal thyroxine: a dynamic and kinetic evaluation of a putative mode-of-actionNTP-CERHR Expert Panel Report on the reproductive and developmental toxicity of methylphenidate.NTP-CERHR Expert Panel Report on the reproductive and developmental toxicity of amphetamine and methamphetamine.Mode of action: neurotoxicity induced by thyroid hormone disruption during development--hearing loss resulting from exposure to PHAHs.Mode of action: developmental thyroid hormone insufficiency--neurological abnormalities resulting from exposure to propylthiouracil.Using in vitro high throughput screening assays to identify potential endocrine-disrupting chemicals.Implications of the use of neonatal birth weight, growth, viability, and survival data for predicting developmental neurotoxicity: a survey of the literature.Undertaking positive control studies as part of developmental neurotoxicity testing: a report from the ILSI Research Foundation/Risk Science Institute expert working group on neurodevelopmental endpoints.Identification and interpretation of developmental neurotoxicity effects: a report from the ILSI Research Foundation/Risk Science Institute expert working group on neurodevelopmental endpoints.Thyroid disrupting chemicals: mechanisms and mixtures.A retrospective performance assessment of the developmental neurotoxicity study in support of OECD test guideline 426Thyroid-disrupting chemicals: interpreting upstream biomarkers of adverse outcomes.Critical analysis of literature on low-dose synergy for use in screening chemical mixtures for risk assessment.Reflex modification and the detection of toxicant-induced auditory dysfunction.
P50
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P50
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Kevin M Crofton
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Kevin M Crofton
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P1053
J-4798-2015
P106
P2038
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P496
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