Pathways of matrix metalloproteinase induction in heart failure: bioactive molecules and transcriptional regulation.
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Friends or Foes: Matrix Metalloproteinases and Their Multifaceted Roles in Neurodegenerative DiseasesInflammasome activation in airway epithelial cells after multi-walled carbon nanotube exposure mediates a profibrotic response in lung fibroblastsPathophysiology of Heart Failure.Pathway and network approaches for identification of cancer signature markers from omics data.Differential effect of wall tension on matrix metalloproteinase promoter activation in the thoracic aorta.Short-term disruption in regional left ventricular electrical conduction patterns increases interstitial matrix metalloproteinase activityCardiac remodeling in obesity.Effects of spironolactone on atrial structural remodelling in a canine model of atrial fibrillation produced by prolonged atrial pacing.Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-α-induced cardiomyocyte deathDiscordant activation of gene promoters for matrix metalloproteinases and tissue inhibitors of the metalloproteinases following myocardial infarction.Determinants of extracellular matrix remodelling are differentially expressed in paediatric and adult dilated cardiomyopathyCurcumin suppresses gelatinase B mediated norepinephrine induced stress in H9c2 cardiomyocytes.Involvement of Depressive Catecholamines as Thrombosis Risk/Inflammatory Markers in Non-Smoker, Non-Obese Congestive Heart Failure, Linked to Increased Epidermal Growth Factor-Receptor (EGF-R) Production.Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature.Prenatal nicotine increases matrix metalloproteinase 2 (MMP-2) expression in fetal guinea pig heartsAcute actions and novel targets of matrix metalloproteinases in the heart and vasculatureAllopurinol reduces antigen-specific and polyclonal activation of human T cells.Differential Expression of Matrix Metalloproteinases 2, 9 and Cytokines by Neutrophils and Monocytes in the Clinical Forms of Chagas Disease.Cardiomyopathy in the dystrophin/utrophin-deficient mouse model of severe muscular dystrophy is characterized by dysregulation of matrix metalloproteinases.Role of xanthine oxidoreductase in cardiac nitroso-redox imbalanceFrom cardiac repair to cardiac regeneration--ready to translate?Reversing chronic remodeling in heart failure.Towards comprehensive cardiac repair and regeneration after myocardial infarction: Aspects to consider and proteins to deliver.Differential effects of epsilon-aminocaproic acid and aprotinin on matrix metalloproteinase release in patients following cardiopulmonary bypass.Nampt secreted from cardiomyocytes promotes development of cardiac hypertrophy and adverse ventricular remodeling.Anti-Proliferative Effect of an Aqueous Extract of Prunella vulgaris in Vascular Smooth Muscle Cells.Relationships between plasma levels of matrix metalloproteinases and neurohormonal profile in patients with heart failure.Matrix metalloproteinases 2 and 9 are differentially expressed in patients with indeterminate and cardiac clinical forms of Chagas diseaseSubcellular remodelling may induce cardiac dysfunction in congestive heart failure.Biomarkers of pathophysiology in hypertrophic cardiomyopathy: implications for clinical management and prognosis.Deep sequence analysis of gene expression identifies osteopontin as a downstream effector of integrin-linked kinase (ILK) in cardiac-specific ILK knockout miceTemporal and spatial expression of matrix metalloproteinases and tissue inhibitors of metalloproteinases following myocardial infarction.Cardiac fibroblasts, fibrosis and extracellular matrix remodeling in heart disease.Targeting osteopontin, the silent partner of Na+/H+ exchanger isoform 1 in cardiac remodeling.Characterization of the paracrine effects of human skeletal myoblasts transplanted in infarcted myocardium.Intracellular signaling of cardiac fibroblasts.Cardiac Physiology of Aging: Extracellular Considerations.A Review of the Molecular Mechanisms Underlying the Development and Progression of Cardiac Remodeling.Extracellular Ubiquitin: Role in Myocyte Apoptosis and Myocardial Remodeling.Synergic and antagonistic relationship between MMP-2 and MMP-9 with fibrosis and inflammation in Chagas' cardiomyopathy.
P2860
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P2860
Pathways of matrix metalloproteinase induction in heart failure: bioactive molecules and transcriptional regulation.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Pathways of matrix metalloprot ...... nd transcriptional regulation.
@ast
Pathways of matrix metalloprot ...... nd transcriptional regulation.
@en
type
label
Pathways of matrix metalloprot ...... nd transcriptional regulation.
@ast
Pathways of matrix metalloprot ...... nd transcriptional regulation.
@en
prefLabel
Pathways of matrix metalloprot ...... nd transcriptional regulation.
@ast
Pathways of matrix metalloprot ...... nd transcriptional regulation.
@en
P1476
Pathways of matrix metalloprot ...... nd transcriptional regulation.
@en
P2093
Francis G Spinale
P304
P356
10.1016/J.CARDIORES.2005.10.004
P577
2006-01-19T00:00:00Z