Basement membrane and repair of injury to peripheral nerve: defining a potential role for macrophages, matrix metalloproteinases, and tissue inhibitor of metalloproteinases-1.
about
Quantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environmentFriends or Foes: Matrix Metalloproteinases and Their Multifaceted Roles in Neurodegenerative DiseasesWallerian degeneration: the innate-immune response to traumatic nerve injuryDamage-induced neuronal endopeptidase (DINE) is a unique metallopeptidase expressed in response to neuronal damage and activates superoxide scavengersSpiral ganglion cells and macrophages initiate neuro-inflammation and scarring following cochlear implantation.Endoneurial remodeling by TNFalph- and TNFalpha-releasing proteases. A spatial and temporal co-localization study in painful neuropathy.Regeneration of soft tissues is promoted by MMP1 treatment after digit amputation in mice.Matrix metalloproteinase expression and inhibition after sciatic nerve axotomy.MMP-9 sheds the beta2 integrin subunit (CD18) from macrophages.Uridine 5'-triphosphate promotes in vitro Schwannoma cell migration through matrix metalloproteinase-2 activationEffects of 660 and 780 nm low-level laser therapy on neuromuscular recovery after crush injury in rat sciatic nerve.MMPs initiate Schwann cell-mediated MBP degradation and mechanical nociception after nerve damage.Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve.Matrix metalloproteinase inhibition enhances the rate of nerve regeneration in vivo by promoting dedifferentiation and mitosis of supporting schwann cellsNerve repair: a neurobiologist's view.The MMP-9/TIMP-1 axis controls the status of differentiation and function of myelin-forming Schwann cells in nerve regeneration.Antiinflammatory activity of melatonin in central nervous systemEffect of low-level laser therapy (LLLT) on acute neural recovery and inflammation-related gene expression after crush injury in rat sciatic nerve.CCAAT/enhancer binding protein β expression is increased in the brain during HIV-1-infection and contributes to regulation of astrocyte tissue inhibitor of metalloproteinase-1.Wallerian degeneration: gaining perspective on inflammatory events after peripheral nerve injury.The unfolded protein response is a major mechanism by which LRP1 regulates Schwann cell survival after injury.Differential acute and chronic responses of tumor necrosis factor-deficient mice to experimental brain injuryTNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.Biomechanical regulation of human monocyte/macrophage molecular functionDelayed progression of edema formation around a hematoma expressing high levels of VEGF and mmp-9 in a patient with traumatic brain injury: case report.Tissue plasminogen activator-mediated fibrinolysis protects against axonal degeneration and demyelination after sciatic nerve injury.Mitigation of diabetes-related complications in implanted collagen and elastin scaffolds using matrix-binding polyphenol.Targeting invading macrophage-derived PGE2, IL-6 and calcitonin gene-related peptide in injured nerve to treat neuropathic pain.Regulation of cytokine expression by Schwann cells in response to α2-macroglobulin binding to LRP1.MMP-9 controls Schwann cell proliferation and phenotypic remodeling via IGF-1 and ErbB receptor-mediated activation of MEK/ERK pathway.Schwann cell LRP1 regulates remak bundle ultrastructure and axonal interactions to prevent neuropathic pain.Growth factor and stem cell enhanced conduits in peripheral nerve regeneration and repair.Pre-degenerated peripheral nerves co-cultured with bone marrow-derived cells: a new technique for harvesting high-purity Schwann cells.Evidence of innervation following extracellular matrix scaffold-mediated remodelling of muscular tissuesMacrophages contribute to the maintenance of stable regenerating neurites following peripheral nerve injury.Therapeutic potential of matrix metalloprotease inhibitors in neuropathic pain.The neuroimmunology of degeneration and regeneration in the peripheral nervous system.MMP2-9 cleavage of dystroglycan alters the size and molecular composition of Schwann cell domains.Modulation of tight junction proteins in the perineurium for regional pain control.Schwann cells reposition a peripheral nerve to isolate it from postembryonic remodeling of its targets.
P2860
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P2860
Basement membrane and repair of injury to peripheral nerve: defining a potential role for macrophages, matrix metalloproteinases, and tissue inhibitor of metalloproteinases-1.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Basement membrane and repair o ...... bitor of metalloproteinases-1.
@ast
Basement membrane and repair o ...... bitor of metalloproteinases-1.
@en
type
label
Basement membrane and repair o ...... bitor of metalloproteinases-1.
@ast
Basement membrane and repair o ...... bitor of metalloproteinases-1.
@en
prefLabel
Basement membrane and repair o ...... bitor of metalloproteinases-1.
@ast
Basement membrane and repair o ...... bitor of metalloproteinases-1.
@en
P2093
P2860
P356
P1476
Basement membrane and repair o ...... bitor of metalloproteinases-1.
@en
P2093
D A Rappolee
J L Underwood
M La Fleur
P2860
P304
P356
10.1084/JEM.184.6.2311
P407
P577
1996-12-01T00:00:00Z