Extrathymic T cell deletion and allogeneic stem cell engraftment induced with costimulatory blockade is followed by central T cell tolerance.
about
Deletional and regulatory mechanisms coalesce to drive transplantation tolerance through mixed chimerismImmuno-intervention for the induction of transplantation tolerance through mixed chimerismDonor and recipient leukocytes in organ allografts of recipients with variable donor-specific tolerance: with particular reference to chronic rejectionMinimal conditioning required in a murine model of T cell depletion, thymic irradiation and high-dose bone marrow transplantation for the induction of mixed chimerism and tolerance.Co-receptor and co-stimulation blockade for mixed chimerism and tolerance without myelosuppressive conditioning.Allospecific rejection of MHC class I-deficient bone marrow by CD8 T cells.Transplantation tolerance: the concept and its applicability.The problem of anti-pig antibodies in pig-to-primate xenografting: current and novel methods of depletion and/or suppression of production of anti-pig antibodies.Transplantation tolerance-where do we stand?Treg-therapy allows mixed chimerism and transplantation tolerance without cytoreductive conditioningFinally, CTLA4Ig graduates to the clinicThe changed balance of regulatory and naive T cells promotes tolerance after TLI and anti-T-cell antibody conditioning.Requirements for induction and maintenance of peripheral tolerance in stringent allograft modelsThe tolerant recipient: looking great in someone else's genes.LAG-3, TGF-β, and cell-intrinsic PD-1 inhibitory pathways contribute to CD8 but not CD4 T-cell tolerance induced by allogeneic BMT with anti-CD40L.Human islet cell transplantation--future prospects.Costimulatory pathways in transplantation.Increased apoptosis of immunoreactive host cells and augmented donor leukocyte chimerism, not sustained inhibition of B7 molecule expression are associated with prolonged cardiac allograft survival in mice preconditioned with immature donor dendritiThe role of natural killer T cells in costimulation blockade-based mixed chimerismCTLA4Ig promotes the induction of hematopoietic chimerism and tolerance independently of Indoleamine-2,3-dioxygenaseMurine mobilized peripheral blood stem cells have a lower capacity than bone marrow to induce mixed chimerism and tolerance.Recipient dendritic cells, but not B cells, are required antigen-presenting cells for peripheral alloreactive CD8+ T-cell tolerance.Peripheral deletional tolerance of alloreactive CD8 but not CD4 T cells is dependent on the PD-1/PD-L1 pathwayDistinct requirements for achievement of allotolerance versus reversal of autoimmunity via nonmyeloablative mixed chimerism induction in NOD mice.PD-1-dependent mechanisms maintain peripheral tolerance of donor-reactive CD8+ T cells to transplanted tissue.The role of TNF receptor and TNF superfamily molecules in organ transplantation.Graft-versus-host disease can be separated from graft-versus-lymphoma effects by control of lymphocyte trafficking with FTY720.Induction of transplantation tolerance in non-human primate preclinical modelsThe balance of deletion and regulation in allograft tolerance.T-cell costimulatory pathways in allograft rejection and tolerance.Heterologous immunity: an overlooked barrier to tolerance.Hematopoietic chimerism and central tolerance created by peripheral-tolerance induction without myeloablative conditioning.Dipeptidyl peptidase IV (DPPIV/CD26) inhibition does not improve engraftment of unfractionated syngeneic or allogeneic bone marrow after nonmyeloablative conditioningSimultaneous bone marrow and intestine transplantation promotes marrow-derived hematopoietic stem cell engraftment and chimerism.Mixed chimerism and split tolerance: mechanisms and clinical correlations.Hematopoietic chimerism and transplantation tolerance: a role for regulatory T cells.Polyclonal Recipient nTregs Are Superior to Donor or Third-Party Tregs in the Induction of Transplantation ToleranceTolerance-inducing strategies in islet transplantationPrenatal Allogeneic Tolerance in Mice Remains Stable Despite Potent Viral Immune ActivationTreatment of severe autoimmune disease by stem-cell transplantation.
P2860
Q26801029-8703663F-83FA-4020-8AEB-D81EBE942F2FQ27001243-EB9AE60C-1C2E-4C8A-BFE6-6332A858D67FQ30964993-7F146880-0095-40A1-B4C4-31EEAF616124Q31056396-29ECEFCD-9373-4529-B93E-49B66549BFE5Q33241067-E99A5DE4-41A2-424B-908B-08B9A640B954Q33709427-BA268D58-0743-4305-875C-44B9D0573357Q33732945-7499795E-B948-4E6C-BAB2-824B909B570DQ33742186-7C0D551A-241C-4172-ADFE-5EAF8BEDA27CQ33764678-361F7E27-F9E4-448F-867E-E1F209D9E4DAQ33796796-2415DB77-0F39-4F4F-A069-3FC5879FBD2DQ33852345-0E2A5EC0-AF5A-433B-A5B4-0D6C6946D853Q33914718-15C0579B-4EF5-40B8-9238-97995EAAA1BDQ33947967-F6EA76B9-A57F-4707-AFB5-EFB97DFD5EF8Q34116397-4B36F895-72E9-4BF4-97F8-A19D41829EB7Q34172252-1EBDFE6D-8618-439D-9FEF-EF7FD4CBAC0DQ34184785-5D463014-DECE-4F89-9607-75F31EC0B8DEQ34187594-7876CD0A-F919-4A7A-9C91-760E0EF5E54BQ34300891-FBDE318B-A717-47E4-B113-658571CBB3B0Q34356212-3AB454E9-9D80-4A79-8C58-F16FCEBF4594Q34356219-57E7B973-7A5C-4708-8B1E-852121457E5CQ34356227-F5EECA8B-8375-4398-9282-313419C58988Q34433840-C3E86E15-9026-498D-8416-4F427D59089AQ34593527-27B616B6-405F-4330-91DB-1D89DA7621EBQ34595629-8076BCC0-3E98-48A2-97F5-19A62876FCAFQ34597955-ADF397E0-4A80-4377-A703-7C7ADA488DA4Q34719084-1B56C980-9B7E-4277-926E-52A7CFE03158Q34834459-CFCD0F78-7484-4848-BA5F-3F89E2E9497EQ35038079-0EBB78D5-74B7-4DCB-B71F-3174D7E124EEQ35583506-5379F7D7-ECD3-4915-8DAA-5CC1FD640223Q35583511-5C5F5BC1-B7DE-436F-8558-CC9AC18090BFQ35583521-04D1290A-9068-46C0-8A5D-552A19C667DEQ35679173-5BD5F798-6C4A-4CBF-A0B5-EE56A3AD34CCQ35693453-AB9481A6-0D94-4D43-A247-482CEE07126CQ35849953-94AC1C38-F635-4F99-AFB5-A0CB191B8C2FQ35877027-B6BE31C8-D7A7-4746-B3FD-C4E77E459584Q35928393-A99EA330-9092-44FE-88D8-70F03EBA4D7FQ35932423-5B31D89A-26EC-4D5E-875D-56520BEF87BEQ36004707-0FA5E0B8-9975-4DF6-B01A-DB6A211EB8DFQ36119428-C25F808F-9B6A-4E9B-A374-60AA5B844634Q36148421-CE7AD8D3-B4A1-4F0B-9087-93656DD2C14F
P2860
Extrathymic T cell deletion and allogeneic stem cell engraftment induced with costimulatory blockade is followed by central T cell tolerance.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
Extrathymic T cell deletion an ...... d by central T cell tolerance.
@ast
Extrathymic T cell deletion an ...... d by central T cell tolerance.
@en
type
label
Extrathymic T cell deletion an ...... d by central T cell tolerance.
@ast
Extrathymic T cell deletion an ...... d by central T cell tolerance.
@en
prefLabel
Extrathymic T cell deletion an ...... d by central T cell tolerance.
@ast
Extrathymic T cell deletion an ...... d by central T cell tolerance.
@en
P2093
P2860
P356
P1476
Extrathymic T cell deletion an ...... d by central T cell tolerance.
@en
P2093
A Chandraker
K G Swenson
M H Sayegh
P2860
P304
P356
10.1084/JEM.187.12.2037
P407
P577
1998-06-01T00:00:00Z