MyD88 but not TRIF is essential for osteoclastogenesis induced by lipopolysaccharide, diacyl lipopeptide, and IL-1alpha - Wikidata - awgldk - TriplyDB

MyD88 but not TRIF is essential for osteoclastogenesis induced by lipopolysaccharide, diacyl lipopeptide, and IL-1alpha

MyD88 but not TRIF is essential for osteoclastogenesis induced by lipopolysaccharide, diacyl lipopeptide, and IL-1alpha

http://www.wikidata.org/entity/Q36402446

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Osteoimmunology: interactions of the bone and immune system Inflammatory modulation of HSCs: viewing the HSC as a foundation for the immune response TRIF Differentially Regulates Hepatic Steatosis and Inflammation/Fibrosis in Mice MyD88-dependent silencing of transgene expression during the innate and adaptive immune response to helper-dependent adenovirus.Infection of RANKL-primed RAW-D macrophages with Porphyromonas gingivalis promotes osteoclastogenesis in a TNF-α-independent manner.Role of periodontal pathogenic bacteria in RANKL-mediated bone destruction in periodontal disease.TLR2-dependent modulation of osteoclastogenesis by Porphyromonas gingivalis through differential induction of NFATc1 and NF-kappaB.Toll-like receptors on hematopoietic progenitor cells stimulate innate immune system replenishment PIN1 inhibition suppresses osteoclast differentiation and inflammatory responses.TLR signaling that induces weak inflammatory response and SHIP1 enhances osteogenic functions.Cell proliferation and survival induced by Toll-like receptors is antagonized by type I IFNs Osteoprotegerin Regulates Pancreatic β-Cell Homeostasis upon Microbial Invasion Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activation of Toll-like Receptor 2 in Osteoblasts.Oral inflammatory diseases and systemic inflammation: role of the macrophage.The role of SH3BP2 in the pathophysiology of cherubism.Signal transduction of lipopolysaccharide-induced osteoclast differentiation.Osteoclasts - the innate immune cells of the bone.Intercellular Communication between Keratinocytes and Fibroblasts Induces Local Osteoclast Differentiation: a Mechanism Underlying Cholesteatoma-Induced Bone Destruction Lipopolysaccharide-induced osteoclastogenesis from mononuclear precursors: a mechanism for osteolysis in chronic otitis Protein kinase D1 is essential for MyD88-dependent TLR signaling pathway Osteoimmunology: cytokines and the skeletal system.γ-Glutamyltranspeptidase is an endogenous activator of Toll-like receptor 4-mediated osteoclastogenesis.Selective expansion of the monocytic lineage directed by bacterial infection Methionine down-regulates TLR4/MyD88/NF-κB signalling in osteoclast precursors to reduce bone loss during osteoporosis.Identification of human metapneumovirus-induced gene networks in airway epithelial cells by microarray analysis.Inhibition of RANK expression and osteoclastogenesis by TLRs and IFN-gamma in human osteoclast precursors Cytokine regulation of lung Th17 response to airway immunization using LPS adjuvant.Mitochondrial superoxide in osteocytes perturbs canalicular networks in the setting of age-related osteoporosis.Osteoimmunology: a brief introduction Inflammatory and immune pathways in the pathogenesis of periodontal disease.Roles of cathelicidins in inflammation and bone loss.Acute toll-like receptor 4 activation impairs rat renal microvascular autoregulatory behaviour.MyD88-BLT2-dependent cascade contributes to LPS-induced interleukin-6 production in mouse macrophage.Role of the Toll-like receptor pathway in the recognition of orthopedic implant wear-debris particles.Rab27a and Rab27b are involved in stimulation-dependent RANKL release from secretory lysosomes in osteoblastic cells.Signaling pathways in osteoblast proinflammatory responses to infection by Porphyromonas gingivalis.The blocking of uPAR suppresses lipopolysaccharide-induced inflammatory osteoclastogenesis and the resultant bone loss through attenuation of integrin β3/Akt pathway Porphyromonas gingivalis Stimulates TLR2-PI3K Signaling to Escape Immune Clearance and Induce Bone Resorption Independently of MyD88.Roles of cathelicidin-related antimicrobial peptide in murine osteoclastogenesis.MyD88 expression in the rat dental follicle: implications for osteoclastogenesis and tooth eruption.

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P2860

MyD88 but not TRIF is essential for osteoclastogenesis induced by lipopolysaccharide, diacyl lipopeptide, and IL-1alpha

http://www.wikidata.org/entity/Q36402446

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

MyD88 but not TRIF is essentia ...... cyl lipopeptide, and IL-1alpha

@ast

MyD88 but not TRIF is essentia ...... cyl lipopeptide, and IL-1alpha

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type

label

MyD88 but not TRIF is essentia ...... cyl lipopeptide, and IL-1alpha

@ast

MyD88 but not TRIF is essentia ...... cyl lipopeptide, and IL-1alpha

@en

prefLabel

MyD88 but not TRIF is essentia ...... cyl lipopeptide, and IL-1alpha

@ast

MyD88 but not TRIF is essentia ...... cyl lipopeptide, and IL-1alpha

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P1433

Journal of Experimental Medicine

P1476

MyD88 but not TRIF is essentia ...... cyl lipopeptide, and IL-1alpha

@en

P2093

Haruhiko Takada

http://www.w3.org/2001/XMLSchema#string

Kenichiro Shibata

http://www.w3.org/2001/XMLSchema#string

Kiyoshi Takeda

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Koji Suda

http://www.w3.org/2001/XMLSchema#string

Mariko Yamaki

http://www.w3.org/2001/XMLSchema#string

Masahiro Yamamoto

http://www.w3.org/2001/XMLSchema#string

Midori Nakamura

http://www.w3.org/2001/XMLSchema#string

Naoyuki Takahashi

http://www.w3.org/2001/XMLSchema#string

Nobuaki Sato

http://www.w3.org/2001/XMLSchema#string

Nobuyuki Udagawa

http://www.w3.org/2001/XMLSchema#string

P2860

Stimulation by toll-like receptors inhibits osteoclast differentiation

osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

LPS-TLR4 signaling to IRF-3/7 and NF-kappaB involves the toll adapters TRAM and TRIF

Modulation of osteoclast differentiation and function by the new members of the tumor necrosis factor receptor and ligand families

Bone versus immune system

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

NF-kappaB1/p105 regulates lipopolysaccharide-stimulated MAP kinase signaling by governing the stability and function of the Tpl2 kinase

Targeted disruption of the biglycan gene leads to an osteoporosis-like phenotype in mice

TRAM is specifically involved in the Toll-like receptor 4-mediated MyD88-independent signaling pathway

Identification of Lps2 as a key transducer of MyD88-independent TIR signalling

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601-611

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10.1084/JEM.20040689

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5

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200

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2004-09-01T00:00:00Z

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8359496

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15353553

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lipopolysaccharide

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2212746

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