CD8(+) T cells can block herpes simplex virus type 1 (HSV-1) reactivation from latency in sensory neurons.
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Herpes simplex virus and varicella zoster virus, the house guests who never leaveSelective retention of herpes simplex virus-specific T cells in latently infected human trigeminal gangliaPersistent expression of chemokine and chemokine receptor RNAs at primary and latent sites of herpes simplex virus 1 infectionEvidence that spontaneous reactivation of herpes virus does not occur in miceRe-evaluating the role of natural killer cells in innate resistance to herpes simplex virus type 1Potential function of miRNAs in herpetic stromal keratitisBovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting ApoptosisT cell memory in the context of persistent herpes viral infectionsImmunological aspects of acute and recurrent herpes simplex keratitisIlluminating viral infections in the nervous systemKeeping it in check: chronic viral infection and antiviral immunity in the brainGenital Herpes: Insights into Sexually Transmitted Infectious Disease17-beta estradiol promotion of herpes simplex virus type 1 reactivation is estrogen receptor dependentSustained CD8+ T cell memory inflation after infection with a single-cycle cytomegalovirus.Characterization of the IFN-gamma T-cell responses to immediate early antigens in humans with genital herpesOcular and neural distribution of feline herpesvirus-1 during active and latent experimental infection in cats.Frequent release of low amounts of herpes simplex virus from neurons: results of a mathematical model.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleInterferon-beta suppresses herpes simplex virus type 1 replication in trigeminal ganglion cells through an RNase L-dependent pathwayThe locus encompassing the latency-associated transcript of herpes simplex virus type 1 interferes with and delays interferon expression in productively infected neuroblastoma cells and trigeminal Ganglia of acutely infected mice.Public TCR use by herpes simplex virus-2-specific human CD8 CTLs.Herpes simplex virus and the chemokines that mediate the inflammation.Herpes simplex virus-1 infects the olfactory bulb shortly following ocular infection and exhibits a long-term inflammatory profile in the form of effector and HSV-1-specific T cells.Protective T-cell-based immunity induced in neonatal mice by a single replicative cycle of herpes simplex virusGamma interferon can prevent herpes simplex virus type 1 reactivation from latency in sensory neurons.Herpes simplex virus-specific memory CD8+ T cells are selectively activated and retained in latently infected sensory gangliaReversible nerve damage and corneal pathology in murine herpes simplex stromal keratitis.Role of CD8+ T cells and lymphoid dendritic cells in protection from ocular herpes simplex virus 1 challenge in immunized mice.Gamma interferon can block herpes simplex virus type 1 reactivation from latency, even in the presence of late gene expressionRole of inflammatory cytokine-induced cyclooxygenase 2 in the ocular immunopathologic disease herpetic stromal keratitis.Long-term presence of virus-specific plasma cells in sensory ganglia and spinal cord following intravaginal inoculation of herpes simplex virus type 2.Recent progress in herpes simplex virus immunobiology and vaccine researchExpression of herpes simplex virus 1-encoded microRNAs in human trigeminal ganglia and their relation to local T-cell infiltrates.A herpes simplex virus type 1 mutant expressing a baculovirus inhibitor of apoptosis gene in place of latency-associated transcript has a wild-type reactivation phenotype in the mouse.B7 costimulation molecules encoded by replication-defective, vhs-deficient HSV-1 improve vaccine-induced protection against corneal diseaseA novel p40-independent function of IL-12p35 is required for progression and maintenance of herpes stromal keratitisHeterogeneity and evolution of thymidine kinase and DNA polymerase mutants of herpes simplex virus type 1: implications for antiviral therapy.CD4 T-cell help programs a change in CD8 T-cell function enabling effective long-term control of murine gammaherpesvirus 68: role of PD-1-PD-L1 interactions.Delaying the expression of herpes simplex virus type 1 glycoprotein B (gB) to a true late gene alters neurovirulence and inhibits the gB-CD8+ T-cell response in the trigeminal ganglionHoming in on the cellular immune response to HSV-2 in humans.
P2860
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P2860
CD8(+) T cells can block herpes simplex virus type 1 (HSV-1) reactivation from latency in sensory neurons.
description
2000 nî lūn-bûn
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name
CD8(+) T cells can block herpe ...... om latency in sensory neurons.
@ast
CD8(+) T cells can block herpe ...... om latency in sensory neurons.
@en
type
label
CD8(+) T cells can block herpe ...... om latency in sensory neurons.
@ast
CD8(+) T cells can block herpe ...... om latency in sensory neurons.
@en
prefLabel
CD8(+) T cells can block herpe ...... om latency in sensory neurons.
@ast
CD8(+) T cells can block herpe ...... om latency in sensory neurons.
@en
P2093
P2860
P356
P1476
CD8(+) T cells can block herpe ...... om latency in sensory neurons.
@en
P2093
P2860
P304
P356
10.1084/JEM.191.9.1459
P407
P577
2000-05-01T00:00:00Z