Modification of cardiac sodium channels by carboxyl reagents. Trimethyloxonium and water-soluble carbodiimide.
about
The extracellular K+ concentration dependence of outward currents through Kir2.1 channels is regulated by extracellular Na+ and Ca2+.Extrapore residues of the S5-S6 loop of domain 2 of the voltage-gated skeletal muscle sodium channel (rSkM1) contribute to the mu-conotoxin GIIIA binding site.Ion permeation, divalent ion block, and chemical modification of single sodium channels. Description by single- and double-occupancy rate-theory models.Surface charge potentiates conduction through the cardiac ryanodine receptor channel.Conduction through the inward rectifier potassium channel, Kir2.1, is increased by negatively charged extracellular residues.Unitary conductance of Na+ channel isoforms in cardiac and NB2a neuroblastoma cells.
P2860
Modification of cardiac sodium channels by carboxyl reagents. Trimethyloxonium and water-soluble carbodiimide.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Modification of cardiac sodium ...... nd water-soluble carbodiimide.
@ast
Modification of cardiac sodium ...... nd water-soluble carbodiimide.
@en
type
label
Modification of cardiac sodium ...... nd water-soluble carbodiimide.
@ast
Modification of cardiac sodium ...... nd water-soluble carbodiimide.
@en
prefLabel
Modification of cardiac sodium ...... nd water-soluble carbodiimide.
@ast
Modification of cardiac sodium ...... nd water-soluble carbodiimide.
@en
P2860
P356
P1476
Modification of cardiac sodium ...... nd water-soluble carbodiimide.
@en
P2093
C M Baumgarten
S C Dudley
P2860
P304
P356
10.1085/JGP.101.5.651
P577
1993-05-01T00:00:00Z