The Aryl Hydrocarbon Receptor is a Critical Regulator of Tissue Factor Stability and an Antithrombotic Target in Uremia.
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Indoxyl Sulfate-Review of Toxicity and Therapeutic StrategiesThe systemic nature of CKD.Nutrients Turned into Toxins: Microbiota Modulation of Nutrient Properties in Chronic Kidney Disease.Indoxyl sulfate (IS)-mediated immune dysfunction provokes endothelial damage in patients with end-stage renal disease (ESRD).Indoxyl sulfate - the uremic toxin linking hemostatic system disturbances with the prevalence of cardiovascular disease in patients with chronic kidney disease.An Aryl Hydrocarbon Receptor-Mediated Amplification Loop That Enforces Cell Migration in ER-/PR-/Her2- Human Breast Cancer CellsImpact of Indoxyl Sulfate on Progenitor Cell-Related Neovascularization of Peripheral Arterial Disease and Post-Angioplasty Thrombosis of Dialysis Vascular Access.The Uremic Toxin Indoxyl Sulfate Accelerates Thrombotic Response after Vascular Injury in Animal Models.The gut-kidney axis.Vasculopathy in the setting of cardiorenal syndrome: roles of protein-bound uremic toxins.Dioxins and related environmental contaminants increase TDP-43 levels.Dialysis Vascular Access Intervention and the Search for Biomarkers.Modulation of a Circulating Uremic Solute via Rational Genetic Manipulation of the Gut Microbiota.Microbiome perturbation by oral vancomycin reduces plasma concentration of two gut-derived uremic solutes, indoxyl sulfate and p-cresyl sulfate, in end-stage renal disease.Uremic Advanced Glycation End Products and Protein-Bound Solutes Induce Endothelial Dysfunction Through Suppression of Krüppel-Like Factor 2.Targeting STUB1-tissue factor axis normalizes hyperthrombotic uremic phenotype without increasing bleeding risk.Uremic Solute-Aryl Hydrocarbon Receptor-Tissue Factor Axis Associates with Thrombosis after Vascular Injury in Humans.Indoxyl Sulfate Promotes Macrophage IL-1β Production by Activating Aryl Hydrocarbon Receptor/NF-κ/MAPK Cascades, but the NLRP3 inflammasome Was Not Activated.Detection of aryl hydrocarbon receptor agonists in human samples.A mass spectrometric method for quantification of tryptophan-derived uremic solutes in human serum.Uremic Toxin Clearance and Cardiovascular Toxicities.Deleting Death and Dialysis: Conservative Care of Cardio-Vascular Risk and Kidney Function Loss in Chronic Kidney Disease (CKD).Indoleamine 2,3-dioxygenase 1 in coronary atherosclerotic plaque enhances tissue factor expression in activated macrophagesImpacts of Indoxyl Sulfate and p-Cresol Sulfate on Chronic Kidney Disease and Mitigating Effects of AST-120Implication of gut microbiota metabolites in cardiovascular and metabolic diseases
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P2860
The Aryl Hydrocarbon Receptor is a Critical Regulator of Tissue Factor Stability and an Antithrombotic Target in Uremia.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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name
The Aryl Hydrocarbon Receptor ...... tithrombotic Target in Uremia.
@ast
The Aryl Hydrocarbon Receptor ...... tithrombotic Target in Uremia.
@en
type
label
The Aryl Hydrocarbon Receptor ...... tithrombotic Target in Uremia.
@ast
The Aryl Hydrocarbon Receptor ...... tithrombotic Target in Uremia.
@en
prefLabel
The Aryl Hydrocarbon Receptor ...... tithrombotic Target in Uremia.
@ast
The Aryl Hydrocarbon Receptor ...... tithrombotic Target in Uremia.
@en
P2093
P2860
P356
P1476
The Aryl Hydrocarbon Receptor ...... tithrombotic Target in Uremia.
@en
P2093
Anqi Zhang
David H Sherr
Elazer R Edelman
Janice Weinberg
Jean Francis
Kumaran Kolandaivelu
Laith Al-Rabadi
Mercedes Balcells
Michael P Pollastri
Moshe Shashar
P2860
P304
P356
10.1681/ASN.2014121241
P577
2015-05-27T00:00:00Z