Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity. - Wikidata - awgldk - TriplyDB

Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

http://www.wikidata.org/entity/Q36420529

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Excitatory amino acid transporter 2 associates with phosphorylated tau and is localized in neurofibrillary tangles of tauopathic brains NAIP interacts with hippocalcin and protects neurons against calcium-induced cell death through caspase-3-dependent and -independent pathways Mechanism of ceftriaxone induction of excitatory amino acid transporter-2 expression and glutamate uptake in primary human astrocytes Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors Chronic Glutamate Toxicity in Neurodegenerative Diseases-What is the Evidence?Glutamate transporter 1: target for the treatment of alcohol dependence Anti-Yo antibody uptake and interaction with its intracellular target antigen causes Purkinje cell death in rat cerebellar slice cultures: a possible mechanism for paraneoplastic cerebellar degeneration in humans with gynecological or breast cancers Flip and flop splice variants of AMPA receptor subunits in the spinal cord of amyotrophic lateral sclerosis Excitotoxicity and ALS: what is unique about the AMPA receptors expressed on spinal motor neurons?Age-related changes in rat cerebellar basket cells: a quantitative study using unbiased stereological methods Amyotrophic lateral sclerosis 2-deficiency leads to neuronal degeneration in amyotrophic lateral sclerosis through altered AMPA receptor trafficking Novel Neuroprotective Multicomponent Therapy for Amyotrophic Lateral Sclerosis Designed by Networked Systems Modulation of the neural glutamate transporter EAAC1 by the addicsin-interacting protein ARL6IP1 Advances in the treatment of anxiety: targeting glutamate.Neuronal uptake of anti-Hu antibody, but not anti-Ri antibody, leads to cell death in brain slice cultures.Hereditary non-spherocytic haemolytic anaemia due to red blood cell glutathione synthetase deficiency in four unrelated patients from Spain: clinical and molecular studies.N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx.Glial transporters for glutamate, glycine and GABA I. Glutamate transporters.Expression of AMPA and NMDA receptor subunits in the cervical spinal cord of wobbler mice Nordihydroguaiaretic acid increases glutamate uptake in vitro and in vivo: therapeutic implications for amyotrophic lateral sclerosis A beta-lactam antibiotic dampens excitotoxic inflammatory CNS damage in a mouse model of multiple sclerosis.Altered expression of glutamate signaling, growth factor, and glia genes in the locus coeruleus of patients with major depression.Neurotoxic mechanisms of degeneration in motor neuron diseases.Glutamate transporters and the excitotoxic path to motor neuron degeneration in amyotrophic lateral sclerosis.Progressive spinal muscular atrophies.A two-compartment organotypic model of mammalian peripheral nerve repair.Glutamate as a hippocampal neuron survival factor: an inherited defect in the trisomy 16 mouse.IL-6 induces regionally selective spinal cord injury in patients with the neuroinflammatory disorder transverse myelitis.BMAA--an unusual cyanobacterial neurotoxin.Oligodendroglia metabolically support axons and contribute to neurodegeneration.SKF83959 suppresses excitatory synaptic transmission in rat hippocampus via a dopamine receptor-independent mechanism.Glutamate transporter EAAT2: a new target for the treatment of neurodegenerative diseases.Selected statins produce rapid spinal motor neuron loss in vitro.Human T cell leukemia virus type I and neurologic disease: events in bone marrow, peripheral blood, and central nervous system during normal immune surveillance and neuroinflammation.Familial amyotrophic lateral sclerosis.Therapeutic developments in the treatment of amyotrophic lateral sclerosis.Chronic inhibition of superoxide dismutase produces apoptotic death of spinal neurons.The role of astroglia in neuroprotection Pleiotrophin is a neurotrophic factor for spinal motor neurons.Functional genomic analysis unravels a metabolic-inflammatory interplay in adrenoleukodystrophy

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P2860

Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

http://www.wikidata.org/entity/Q36420529

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1993 nî lūn-bûn

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1993年の論文

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1993年学术文章

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1993年学术文章

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1993年学术文章

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1993年学术文章

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1993年学术文章

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1993年學術文章

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1993年學術文章

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1993年學術文章

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name

Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

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Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

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type

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Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

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Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

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prefLabel

Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

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Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

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Proceedings of the National Academy of Sciences of the United States of America

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Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity.

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J D Rothstein

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L Jin

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M Dykes-Hoberg

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R W Kuncl

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P2860

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

Abnormal excitatory amino acid metabolism in amyotrophic lateral sclerosis

A simple method for organotypic cultures of nervous tissue

N-methyl-D-aspartate receptors are transiently expressed in the developing spinal cord ventral horn.

Excitatory amino acid neurotoxicity and neurodegenerative disease.

Functional interactions between neurons and astrocytes I. Turnover and metabolism of putative amino acid transmitters.

AMPA glutamate receptor subunits are differentially distributed in rat brain.

Decreased glutamate transport by the brain and spinal cord in amyotrophic lateral sclerosis.

Importance of glutamine for gamma-aminobutyric acid synthesis in rat neostriatum in vivo.

GYKI 52466 antagonizes glutamate responses but not NMDA and kainate responses in rat abducens motoneurones.

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