Hemodynamic profile, responsiveness to anandamide, and baroreflex sensitivity of mice lacking fatty acid amide hydrolase. - Wikidata - awgldk - TriplyDB

Hemodynamic profile, responsiveness to anandamide, and baroreflex sensitivity of mice lacking fatty acid amide hydrolase.

Hemodynamic profile, responsiveness to anandamide, and baroreflex sensitivity of mice lacking fatty acid amide hydrolase.

http://www.wikidata.org/entity/Q36426831

about

Fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury The endocannabinoid system as an emerging target of pharmacotherapy Dynamic correction for parallel conductance, GP, and gain factor, alpha, in invasive murine left ventricular volume measurements Endothelium-dependent metabolism by endocannabinoid hydrolases and cyclooxygenases limits vasorelaxation to anandamide and 2-arachidonoylglycerol A bio-telemetric device for measurement of left ventricular pressure-volume loops using the admittance technique in conscious, ambulatory rats.Inhibitor of fatty acid amide hydrolase normalizes cardiovascular function in hypertension without adverse metabolic effects Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood flow regulation.Myofilament calcium sensitization delays decompensated hypertrophy differently between the sexes following myocardial infarction Cannabinoid-2 receptor agonist HU-308 protects against hepatic ischemia/reperfusion injury by attenuating oxidative stress, inflammatory response, and apoptosis 'Entourage' effects of N-palmitoylethanolamide and N-oleoylethanolamide on vasorelaxation to anandamide occur through TRPV1 receptors.Serum contents of endocannabinoids are correlated with blood pressure in depressed women.Cannabinoids as therapeutic agents in cardiovascular disease: a tale of passions and illusions Local epicardial inotropic drug delivery allows targeted pharmacologic intervention with preservation of myocardial loading conditions.Cannabinoid receptors in acute and chronic complications of atherosclerosis.Endocannabinoids and cannabinoid receptors in ischaemia-reperfusion injury and preconditioning Decreased age-related cardiac dysfunction, myocardial nitrative stress, inflammatory gene expression, and apoptosis in mice lacking fatty acid amide hydrolase.Cannabinoid-2 receptor mediates protection against hepatic ischemia/reperfusion injury The cannabinoid system and its pharmacological manipulation--a review, with emphasis upon the uptake and hydrolysis of anandamide.Sex-related changes in cardiac function following myocardial infarction in mice.Endocannabinoids in cerebrovascular regulation.The role of the endocannabinoid system in atherosclerosis.Endocannabinoid anandamide mediates hypoxic pulmonary vasoconstriction.The case for the development of novel analgesic agents targeting both fatty acid amide hydrolase and either cyclooxygenase or TRPV1.Endocannabinoids and cardiac contractile function: pathophysiological implications.Endocannabinoids and the heart.The emerging role of the endocannabinoid system in cardiovascular disease.Endocannabinoids and the cardiovascular response to stress.Recent advances in the discovery and evaluation of fatty acid amide hydrolase inhibitors.Endocannabinoid system as a potential mechanism for n-3 long-chain polyunsaturated fatty acid mediated cardiovascular protection.Antibacterial cathelicidin peptide CAP11 suppresses the anandamide production from lipopolysaccharide-stimulated mononuclear phagocytes.Acute hypertension reveals depressor and vasodilator effects of cannabinoids in conscious rats Cardiovascular effects of cannabinoids in conscious spontaneously hypertensive rats Is there a role for anandamide in cardiovascular regulation? Insights from studies of endocannabinoid metabolism.Rapid microcomputed tomography suggests cardiac enlargement occurs during conductance catheter measurements in mice.Chronic inhibition of fatty acid amide hydrolase by URB597 produces differential effects on cardiac performance in normotensive and hypertensive rats.

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Hemodynamic profile, responsiveness to anandamide, and baroreflex sensitivity of mice lacking fatty acid amide hydrolase.

http://www.wikidata.org/entity/Q36426831

description

2005 nî lūn-bûn

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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AJPHEART.00107.2005

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American Journal of Physiology - Heart and Circulatory Physiology

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Hemodynamic profile, responsiv ...... ng fatty acid amide hydrolase.

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P2093

Attila Brassai

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George Kunos

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Jie Liu

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Judy Harvey-White

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László Offertáler

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Sándor Bátkai

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Zoltán Járai

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P2860

Endocannabinoid activation at hepatic CB1 receptors stimulates fatty acid synthesis and contributes to diet-induced obesity

Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase

Cannabinoid-induced mesenteric vasodilation through an endothelial site distinct from CB1 or CB2 receptors

Structure of a cannabinoid receptor and functional expression of the cloned cDNA

Enzymatic synthesis and degradation of anandamide, a cannabinoid receptor agonist

Platelet- and macrophage-derived endogenous cannabinoids are involved in endotoxin-induced hypotension

The endogenous cannabinoid system and its role in nociceptive behavior

Cyclohexylcarbamic acid 3'- or 4'-substituted biphenyl-3-yl esters as fatty acid amide hydrolase inhibitors: synthesis, quantitative structure-activity relationships, and molecular modeling studies

Molecular characterization of an enzyme that degrades neuromodulatory fatty-acid amides

Molecular characterization of a peripheral receptor for cannabinoids

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2

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Benjamin Cravatt III

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