Generation of varicella-zoster virus (VZV) and viral mutants from cosmid DNAs: VZV thymidylate synthetase is not essential for replication in vitro
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Varicella-zoster virusGenome-wide mutagenesis reveals that ORF7 is a novel VZV skin-tropic factorStructure of the Varicella Zoster Virus Thymidylate Synthase Establishes Functional and Structural Similarities as the Human Enzyme and Potentiates Itself as a Target of BrivudineA genetic system for rhesus monkey rhadinovirus: use of recombinant virus to quantitate antibody-mediated neutralization.Varicella-zoster virus human ganglionic latency: a current summary.Role of the varicella-zoster virus gene product encoded by open reading frame 35 in viral replication in vitro and in differentiated human skin and T cells in vivo.Regions of the varicella-zoster virus open reading frame 63 latency-associated protein important for replication in vitro are also critical for efficient establishment of latency.Varicella-zoster virus (VZV) ORF32 encodes a phosphoprotein that is posttranslationally modified by the VZV ORF47 protein kinase.Molecular mechanisms of varicella zoster virus pathogenesis.Infection of human T lymphocytes with varicella-zoster virus: an analysis with viral mutants and clinical isolatesConstruction and characterization of murine cytomegaloviruses that contain transposon insertions at open reading frames m09 and M83.In vitro and in vivo characterization of a murine cytomegalovirus with a transposon insertional mutation at open reading frame M43.Murine cytomegalovirus containing a mutation at open reading frame M37 is severely attenuated in growth and virulence in vivo.Open reading frame S/L of varicella-zoster virus encodes a cytoplasmic protein expressed in infected cellsVaricella-zoster virus T cell tropism and the pathogenesis of skin infectionVaricella-Zoster virus pathogenesis and immunobiology: new concepts emerging from investigations with the SCIDhu mouse model.Murine cytomegalovirus open reading frame M27 plays an important role in growth and virulence in mice.Mutational analysis of the repeated open reading frames, ORFs 63 and 70 and ORFs 64 and 69, of varicella-zoster virus.Rescue of a pathogenic Marek's disease virus with overlapping cosmid DNAs: use of a pp38 mutant to validate the technology for the study of gene function.Herpesvirus BACs: past, present, and future.Varicella-zoster virus open reading frame 2 encodes a membrane phosphoprotein that is dispensable for viral replication and for establishment of latency.Varicella-zoster virus (VZV) ORF17 protein induces RNA cleavage and is critical for replication of VZV at 37 degrees C but not 33 degrees C.Phosphorylation by the varicella-zoster virus ORF47 protein serine kinase determines whether endocytosed viral gE traffics to the trans-Golgi network or recycles to the cell membrane.Varicella in Chimpanzees.Murine cytomegalovirus with a transposon insertional mutation at open reading frame m155 is deficient in growth and virulence in mice.Identification of small molecule compounds that selectively inhibit varicella-zoster virus replicationVaricella-zoster virus open reading frame 10 is a virulence determinant in skin cells but not in T cells in vivoVaricella-zoster virus infection triggers formation of an interleukin-1β (IL-1β)-processing inflammasome complexRecent advances in cloning herpesviral genomes as infectious bacterial artificial chromosomesVaricella zoster virus latency.Essential functions of the unique N-terminal region of the varicella-zoster virus glycoprotein E ectodomain in viral replication and in the pathogenesis of skin infectionThe immediate-early 63 protein of Varicella-Zoster virus: analysis of functional domains required for replication in vitro and for T-cell and skin tropism in the SCIDhu model in vivo.Simian varicella virus: molecular virology.Varicella-Zoster virus ORF12 protein triggers phosphorylation of ERK1/2 and inhibits apoptosis.The varicella-zoster virus (VZV) open reading frame 47 (ORF47) protein kinase is dispensable for viral replication and is not required for phosphorylation of ORF63 protein, the VZV homolog of herpes simplex virus ICP22.Immunization with recombinant varicella-zoster virus expressing herpes simplex virus type 2 glycoprotein D reduces the severity of genital herpes in guinea pigs.Helper virus-free transfer of herpes simplex virus type 1 plasmid vectors into neural cells.Varicella-zoster virus glycoprotein I is essential for growth of virus in Vero cells.Rodent models of varicella-zoster virus neurotropism.The varicella-zoster virus genome
P2860
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P2860
Generation of varicella-zoster virus (VZV) and viral mutants from cosmid DNAs: VZV thymidylate synthetase is not essential for replication in vitro
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Generation of varicella-zoster ...... ntial for replication in vitro
@ast
Generation of varicella-zoster ...... ntial for replication in vitro
@en
type
label
Generation of varicella-zoster ...... ntial for replication in vitro
@ast
Generation of varicella-zoster ...... ntial for replication in vitro
@en
prefLabel
Generation of varicella-zoster ...... ntial for replication in vitro
@ast
Generation of varicella-zoster ...... ntial for replication in vitro
@en
P2860
P356
P1476
Generation of varicella-zoster ...... ntial for replication in vitro
@en
P2093
P2860
P304
P356
10.1073/PNAS.90.15.7376
P407
P577
1993-08-01T00:00:00Z