Rod microglia: elongation, alignment, and coupling to form trains across the somatosensory cortex after experimental diffuse brain injury. - Wikidata - awgldk - TriplyDB

Rod microglia: elongation, alignment, and coupling to form trains across the somatosensory cortex after experimental diffuse brain injury.

Rod microglia: elongation, alignment, and coupling to form trains across the somatosensory cortex after experimental diffuse brain injury.

http://www.wikidata.org/entity/Q36478500

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The ischemic environment drives microglia and macrophage function Bipolar/rod-shaped microglia are proliferating microglia with distinct M1/M2 phenotypes.Rod microglia: a morphological definition Considerations for Experimental Animal Models of Concussion, Traumatic Brain Injury, and Chronic Traumatic Encephalopathy-These Matters Matter.Acute administration of catalase targeted to ICAM-1 attenuates neuropathology in experimental traumatic brain injury.Microglial Activation in Traumatic Brain Injury.Rod-like microglia are restricted to eyes with laser-induced ocular hypertension but absent from the microglial changes in the contralateral untreated eye.Cortical neurons are a prominent source of the proinflammatory cytokine osteopontin in HIV-associated neurocognitive disorders Resolvins AT-D1 and E1 differentially impact functional outcome, post-traumatic sleep, and microglial activation following diffuse brain injury in the mouse.Disease-related microglia heterogeneity in the hippocampus of Alzheimer's disease, dementia with Lewy bodies, and hippocampal sclerosis of aging.Priming the inflammatory pump of the CNS after traumatic brain injury.Galectin-3 released in response to traumatic brain injury acts as an alarmin orchestrating brain immune response and promoting neurodegeneration.Recent Advances in the Study of Bipolar/Rod-Shaped Microglia and their Roles in Neurodegeneration.Microglia in the TBI brain: The good, the bad, and the dysregulated.Diffusion MRI and the detection of alterations following traumatic brain injury.Administration of DHA Reduces Endoplasmic Reticulum Stress-Associated Inflammation and Alters Microglial or Macrophage Activation in Traumatic Brain Injury.Increased seizure susceptibility in mice 30 days after fluid percussion injury Cognitive deficits develop 1month after diffuse brain injury and are exaggerated by microglia-associated reactivity to peripheral immune challenge.Peripheral viral infection induced microglial sensome genes and enhanced microglial cell activity in the hippocampus of neonatal piglets.Experimental diffuse brain injury results in regional alteration of gross vascular morphology independent of neuropathology.The p38α MAPK regulates microglial responsiveness to diffuse traumatic brain injury.Increased tauopathy drives microglia-mediated clearance of beta-amyloid.Diffuse traumatic brain injury affects chronic corticosterone function in the rat Neuroinflammation as Fuel for Axonal Regeneration in the Injured Vertebrate Central Nervous System.A revisit of rod microglia in preclinical studies Proteome of brain glia: the molecular basis of diverse glial phenotypes.Microglia: dismantling and rebuilding circuits after acute neurological injury.Image processing methods to elucidate spatial characteristics of retinal microglia after optic nerve transection.Rod-shaped microglia morphology is associated with aging in 2 human autopsy series.Does time heal all wounds? Experimental diffuse traumatic brain injury results in persisting histopathology in the thalamus.Neuroinflammation: the devil is in the details.Distinct tau prion strains propagate in cells and mice and define different tauopathies.Acute maternal oxidant exposure causes susceptibility of the fetal brain to inflammation and oxidative stress.Quantitative microglia analyses reveal diverse morphologic responses in the rat cortex after diffuse brain injury Diffuse traumatic brain injury induces prolonged immune dysregulation and potentiates hyperalgesia following a peripheral immune challenge.The association between laminin and microglial morphology in vitro.Physical interactions between activated microglia and injured axons: do all contacts lead to phagocytosis?La Deletion from Mouse Brain Alters Pre-tRNA Metabolism and Accumulation of Pre-5.8S rRNA, with Neuron Death and Reactive Astrocytosis.Local proliferation is the main source of rod microglia after optic nerve transection.Nogo presence is inversely associated with shifts in cortical microglial morphology following experimental diffuse brain injury.

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P2860

Rod microglia: elongation, alignment, and coupling to form trains across the somatosensory cortex after experimental diffuse brain injury.

http://www.wikidata.org/entity/Q36478500

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Rod microglia: elongation, ali ...... rimental diffuse brain injury.

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Rod microglia: elongation, ali ...... rimental diffuse brain injury.

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Rod microglia: elongation, ali ...... rimental diffuse brain injury.

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Rod microglia: elongation, ali ...... rimental diffuse brain injury.

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Rod microglia: elongation, ali ...... rimental diffuse brain injury.

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Rod microglia: elongation, ali ...... erimental diffuse brain injury

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Jonathan Lifshitz

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Jordan L Harrison

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Samuel E Taylor

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Tuoxin Cao

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P2860

Role of CCL2 (MCP-1) in traumatic brain injury (TBI): evidence from severe TBI patients and CCL2-/- mice

Reelin signaling directly affects radial glia morphology and biochemical maturation

Physiology of microglia

Resting microglia directly monitor the functional state of synapses in vivo and determine the fate of ischemic terminals

Microglia: a sensor for pathological events in the CNS

Neurodegeneration in the somatosensory cortex after experimental diffuse brain injury

Continued in situ DNA fragmentation of microglia/macrophages in white matter weeks and months after traumatic brain injury.

The whisker nuisance task identifies a late-onset, persistent sensory sensitivity in diffuse brain-injured rats

Isolation of radial glial cells by fluorescent-activated cell sorting reveals a neuronal lineage.

Diffuse traumatic brain injury initially attenuates and later expands activation of the rat somatosensory whisker circuit concomitant with neuroplastic responses.

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